منابع مشابه
TABES DORSALIS WITH SPECIAL REFERENCE TO PRIMARY OPTIC ATROPHY*t
For many years, dating back to about 1930, neurosyphilis in general and primary optic atrophy of syphilitic origin in particular have been the subjects of special interest and repeated study in the Syphilis Division of the Department of Medicine and the Department of Ophthalmology of the Johns Hopkins Hospital. As to optic atrophy, Moore, Woods, and myself are responsible for the concepts and d...
متن کاملTabes dorsalis with special reference to primary optic atrophy.
For many years, dating back to about 1930, neurosyphilis in general and primary optic atrophy of syphilitic origin in particular have been the subjects of special interest and repeated study in the Syphilis Division of the Department of Medicine and the Department of Ophthalmology of the Johns Hopkins Hospital. As to optic atrophy, Moore, Woods, and myself are responsible for the concepts and d...
متن کاملTabes dorsalis: electrodiagnostic features.
Electrodiagnostic data have not been previously reported in tabes dorsalis. A patient with tabes dorsalis is described whose nerve conduction studies and median nerve somatosensory evoked responses (SEPs) were normal. H-reflexes were absent. SEPs of the tibial nerve suggested posterior column dysfunction. These electrodiagnostic findings correlate precisely with the known pathology of tabes dor...
متن کاملMuscle Deconditioning and Disuse Atrophy
Lack of physical activity can cause a muscle to atrophy. Muscle deconditioning may become apparent within days to weeks of decreased physical activity. For example, we have all known individuals who were physically fit during their high school days and then became less active over time. The cessation of regular activity causes the muscles to decrease in size, but contrary to what some people sa...
متن کاملMolecular Mechanisms of Muscle Atrophy
Skeletal muscle atrophy has extreme adverse consequences. Molecular mechanisms that mediate the process of atrophy are not well defined. Recent studies have focused on diverse molecular cascades that control the activation of ubiquitin ligases, indicating that the involvement of the ubiquitin proteasome may be common to a range of atrophic stimuli.
متن کاملذخیره در منابع من
با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید
ژورنال
عنوان ژورنال: The Journal of Nervous and Mental Disease
سال: 1905
ISSN: 0022-3018
DOI: 10.1097/00005053-190504000-00020