Parasitism, ranging from facultative to obligate and fromgeneral to very host specific, can be a great lifestyle, if you have the right adaptations. In the plant parasite family Orobanchaceae, which includes the agronomically destructive weed Striga, one key adaptation is the ability to sense the proximity of a host root. Host roots can be sensed either by direct contact or by perception of dif...

The antimycin-sensitive ubisemiquinone radical (QC) of the ubiquinol-cytochrome c oxidoreductase of submitochondrial particles and chromatophores of Rhodopseudomonas sphaeroides Ga has been studied by a combination of redox potentiometry and EPR spectroscopy. This g = 2.005 radical signal appears at physiological pH values and increases in intensity with increasing pH up to pH 7.6 in submitocho...

p-Quinone dioxime, a rubber vulcanization accelerator, was selected for bioassay by the National Cancer Institute because of a lack of adequate carcinogenicity data. A bioassay for the possible carcinogenicity of p-quinone dioxime was conducted using Fischer 344 rats and B6C3F1 mice. p-Quinone dioxime was administered in the feed, at either of two concentrations, to groups of 50 male and 50 fem...

NAD(P)H: quinone oxidoreductase isozyme 1 (NQO1) is a flavoenzyme and involved in protection against oxidative stress the regulation of metabolic functions, which strongly implicated neurodegenerative diseases carcinogenic processes. Furthermore, NQO1 also modes action redox-active drugs (e.g., antimalarials). Determining activity localization living organisms great importance for early disease...

Under the irradiation of red light (690 nm), quinones were converted to hydroquinones by thiols in the presence of metallophthalocyanines. The reaction proceeded via the charge separation between the triplet state of phthalocyanine and the quinone. The product determining step was protonation of the quinone anion radical, as indicated by the fact that the reaction was accelerated by the use of ...

The multiplicity reactivation of T4D phage irradiated with UV was inhibited by treatment of infected cells with 0.5 mM hydroquinone (HQ). HQ also inhibited genetic recombination of T4B rII mutants. The recombination frequency was reduced by treatment of infected cells with HQ in a buffer when compared with an untreated control. In addition to HQ, the effects of quinone, an oxide form of HQ, mer...