نتایج جستجو برای: foxp3 protein

تعداد نتایج: 1240954  

2016
Qingqing Quan Fan Wang Xucai Wang

Objective: To explore the expression of CD4+CD25+ regulatory T cell (Treg) and Foxp3 in peripheral blood of patients with chronic obstructive pulmonary disease (COPD) and its clinical significance by performing a casecontrol study. Methods: Eighty-two COPD patients treated in our hospital from Jan 2013 to Jun 2014 were enrolled as case group and 76 healthy individuals as control group. All the ...

ژورنال: :مجله دانشکده پزشکی اصفهان 0
مهتاب تاپاک کارشناس ارشد، گروه ایمنی شناسی، دانشکده ی پزشکی، دانشگاه علوم پزشکی اصفهان، اصفهان، ایران علیرضا عندلیب دانشیار، گروه ایمنی شناسی، دانشکده ی پزشکی، دانشگاه علوم پزشکی اصفهان، اصفهان، ایران پیمان متقی دانشیار، گروه داخلی، دانشکده ی پزشکی، دانشگاه علوم پزشکی اصفهان، اصفهان، ایران شادی بابازاده متخصص رادیوتراپی و انکولوژی، بیمارستان سیدالشهدا (ع)، دانشگاه علوم پزشکی اصفهان، اصفهان، ایران عباس رضایی استاد، گروه ایمنی شناسی، دانشکده ی پزشکی، دانشگاه علوم پزشکی اصفهان، اصفهان، ایران منصور ثالثی استادیار، گروه داخلی، دانشکده ی پزشکی، دانشگاه علوم پزشکی اصفهان، اصفهان، ایران

مقدمه: آرتریت روماتویید به عنوان بیماری th1 غالب تعریف می شود. سلول های tregs گروه جدیدی از سلول های t هستند که سایر سلول های اجرایی سیستم ایمنی از جمله th1 و th2 را تنظیم می کنند. foxp3 عامل توسعه و همچنین حفظ فعالیت مهاری سلول های treg است. در این مطالعه فراوانی سلول های cd4 + foxp3 + treg و cd8 + foxp3 + treg را در مبتلایان به آرتریت روماتویید بررسی کردیم. روش ها: نمونه ی خون محیطی از 31 بیم...

Journal: :Journal of immunology 2012
Qiuxia Li Arvind Shakya Xiaohua Guo Hongbo Zhang Dean Tantin Peter E Jensen Xinjian Chen

Foxp3 plays an essential role in conferring suppressive functionality to CD4(+)/Foxp3(+) regulatory T cells (Tregs). Although studies showed that Foxp3 has to form cooperative complexes with NFAT to bind to target genes, it remains unclear whether NFAT is available in the nucleus of primary Tregs for Foxp3 access. It is generally believed that NFAT in resting cells resides in the cytoplasm, and...

Journal: :Journal of immunology 2009
Katharina Lahl Christian T Mayer Tobias Bopp Jochen Huehn Christoph Loddenkemper Gérard Eberl Gerald Wirnsberger Klaus Dornmair Robert Geffers Edgar Schmitt Jan Buer Tim Sparwasser

Foxp3(+) regulatory T cells (Tregs) are crucial for preventing autoimmunity. We have demonstrated that depletion of Foxp3(+) Tregs results in the development of a scurfy-like disease, indicating that Foxp3(-) effector T cells are sufficient to induce autoimmunity. It has been postulated that nonfunctional Tregs carrying potentially self-reactive T cell receptors may contribute to scurfy (sf) pa...

2009
Reiner K. W. Mailer Kirsten Falk Olaf Rötzschke

BACKGROUND Phenotype and function of regulatory T cells (Treg) largely depend on the presence of the transcription factor FOXP3. In contrast to mice, human Treg cells express isoforms of this protein. Besides the full length version (FOXP3fl), an isoform lacking the exon 2 (FOXP3Delta2) is co-expressed in comparable amounts. Recently, a third splice variant has been described that in addition t...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2014
Ling Lu Qin Lan Zhiyuan Li Xiaohui Zhou Jian Gu Qiang Li Julie Wang Maogen Chen Ya Liu Yi Shen David D Brand Bernhard Ryffel David A Horwitz Francisco P Quismorio Zhongmin Liu Bin Li Nancy J Olsen Song Guo Zheng

Recent studies have demonstrated that thymus-derived naturally occurring CD4(+)Foxp3(+) regulatory T cells (Tregs) in human and mouse may be unstable and dysfunctional in the presence of proinflammatory cytokines. All-trans RA (atRA), the active derivative of vitamin A, has been shown to regulate Treg and T effector cell differentiation. We hypothesize atRA stabilizes human natural Tregs (nTreg...

Journal: :The Journal of clinical investigation 2013
Lauren E Mays Susanne Ammon-Treiber Benedikt Mothes Mohammed Alkhaled Jennifer Rottenberger Eva Sophie Müller-Hermelink Melanie Grimm Markus Mezger Sandra Beer-Hammer Esther von Stebut Nikolaus Rieber Bernd Nürnberg Matthias Schwab Rupert Handgretinger Marco Idzko Dominik Hartl Michael S D Kormann

Chemically modified mRNA is capable of inducing therapeutic levels of protein expression while circumventing the threat of genomic integration often associated with viral vectors. We utilized this novel therapeutic tool to express the regulatory T cell transcription factor, FOXP3, in a time- and site-specific fashion in murine lung, in order to prevent allergic asthma in vivo. We show that modi...

Journal: :Journal of immunology 2005
Felicita Baratelli Ying Lin Li Zhu Seok-Chul Yang Nathalie Heuzé-Vourc'h Gang Zeng Karen Reckamp Mariam Dohadwala Sherven Sharma Steven M Dubinett

Naturally occurring CD4+CD25+ regulatory T cells (T reg) are pivotal in suppressing immune responses and maintaining tolerance. The identification of molecules controlling T reg differentiation and function is important in understanding host immune responses in malignancy and autoimmunity. In this study we show that PGE2 enhances the in vitro inhibitory function of human purified CD4+CD25+ T re...

Abdolrahim Nikzamir, Mohammad Barzegar, Mohammad Reza Zali, Mohammad Rostami-Nejad, Mohammadreza Razzaghi, Mostafa Rezaei-Tavirani, Nastaran Asri,

Celiac disease (CD) is a systemic immune-mediated disorder caused by the dietary gluten in individuals who are genetically susceptible to the disease. In fact, CD is a T cell-mediated immune disease in which gluten-derived peptides activate the lamina propria CD4+ Teff cells, and these T-cell subsets can cause the intestinal tissue damages. Also, there are additional subsets of CD4+ T cells wit...

2015
M Tahara Y Kondo M Yokosawa H Tsuboi S Takahashi S Shibayama I Matsumoto T Sumida

Programmed cell death-1 (PD-1) plays an important role in peripheral T cell tolerance, but whether or not it affects the differentiation of helper T cell subsets remains elusive. Here we describe the importance of PD-1 in the control of T helper type 1 (Th1) cell activation and development of forkhead box protein 3 (FoxP3(+)) regulatory T cells (Tr(egs)). PD-1-deficient T cell-specific T-bet tr...

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