نتایج جستجو برای: ap 1

تعداد نتایج: 2767098  

Journal: :The American journal of physiology 1999
Anami R Patel Jian-Ying Wang

Activator protein 1 (AP-1) is a group of dimeric transcription factors composed of protooncogene (Jun and Fos) subunits that bind to a common DNA site, the AP-1 binding site. The proteins of c-Jun, JunB, and Fos are essential for initiation of the cell cycle. Conversely, the activation of the junD gene slows cell growth in some cell types. The current study tests the hypothesis that polyamines ...

Journal: :American journal of physiology. Gastrointestinal and liver physiology 2011
Aida Habtezion Raymond Kwan Alice L Yang Maureen E Morgan Ehsaan Akhtar Stephen P Wanaski Stephen D Collins Eugene C Butcher Ahmad Kamal M Bishr Omary

Heme oxygenase-1 (HO-1) induction by hemin or Panhematin protects against experimental pancreatitis. As a preclinical first step toward determining whether HO-1 upregulation is a viable target in acute pancreatitis (AP) patients, we tested the hypothesis that HO-1 expression in peripheral blood mononuclear cell (PBMC) subsets of hospitalized patients with mild AP is upregulated then normalizes ...

2015
Xiangnan Zheng Minzhang Cheng Liang Xiang Jian Liang Liping Xie Rongqing Zhang

Activator protein-1 (AP-1) is an important bZIP transcription factor that regulates a series of physiological processes by specifically activating transcription of several genes, and one of its well-chartered functions in mammals is participating in bone mineralization. We isolated and cloned the complete cDNA of a Jun/AP-1 homolog from Pinctada fucata and called it Pf-AP-1. Pf-AP-1 had a highl...

Journal: :Cancer research 1999
M Ding Z Dong F Chen D Pack W Y Ma J Ye X Shi V Castranova V Vallyathan

Activation of activator protein (AP-1) by crocidolite asbestos was examined in vitro in a JB6 P+ cell line stably transfected with AP-1-luciferase reporter plasmid and in vivo using AP-1-luciferase reporter transgenic mice. In in vitro studies, crocidolite asbestos caused a dose- and time-dependent induction of AP-1 activation in cultured JB6 cells. The elevated AP-1 activity persisted for at l...

Journal: :Carcinogenesis 1999
M Ding J J Li S S Leonard J P Ye X Shi N H Colburn V Castranova V Vallyathan

The present study was undertaken to test the hypothesis that the toxicity and carcinogenicity of vanadium might arise from elevation of reactive oxygen species leading to activation of the transcription factor activator protein-1 (AP-1). The AP-1 transactivation response has been implicated as causal in transformation responses to phorbol esters and growth factors. To investigate the possible a...

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