نتایج جستجو برای: awake
تعداد نتایج: 9653 فیلتر نتایج به سال:
The mechanism of general anesthesia remains elusive. ventrolateral periaqueductal gray (vlPAG) in the midbrain regulates sleep and awake states. However, role vlPAG its circuits is unclear. We utilized opto/chemogenetics, righting reflex, electroencephalographic recording to assess consciousness changes. employed fiber photometry measure activity neurons neurotransmitters. As a result, showed t...
Sleep bruxism has been extensively studied in recent decades, but considerable scientific controversy still persists relation to awake bruxism. The present review focused on this topic, including 45 articles published between 2011 and 2020. International consensuses propose advances new diagnostic classifications, which include tools such as questionnaires, clinical examination, electromyograph...
BACKGROUND Pharmacological MRI (phMRI) is a neuroimaging technique where drug-induced hemodynamic responses can represent a pharmacodynamic biomarker to delineate underlying biological consequences of drug actions. In most preclinical studies, animals are anesthetized during image acquisition to minimize movement. However, it has been demonstrated anesthesia could attenuate basal neuronal activ...
BACKGROUND General anesthesia with single-lung ventilation is considered mandatory for thoracoscopic pulmonary resection. We assessed in a randomized study the feasibility and results of awake thoracoscopic resection of solitary pulmonary nodules. METHODS Between March 2001 and February 2003, 60 patients were randomized into two 30-patients arms: a general anesthesia arm entailing double-lume...
The effects of timolol, epinephrine, and acetazolamide on the rate of flow of aqueous humor through the anterior chamber of awake and sleeping human subjects was studied. Timolol reduced the rate in awake subjects but not sleeping subjects. Epinephrine increased the rate in sleeping subjects to a greater extent than in awake subjects. Acetazolamide reduced the rate of flow in awake subjects or ...
BACKGROUND Recent data indicate that the neuroprotective efficacy of isoflurane is not sustained. Delayed neuronal death, mediated in part by apoptosis, contributes to the gradual increase in the size of the infarction. These data suggest that isoflurane may not be able to inhibit delayed neuronal death. The prevention of apoptosis by a caspase inhibitor might provide neuroprotection in additio...
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