نتایج جستجو برای: cd95l

تعداد نتایج: 278  

Journal: :FEMS immunology and medical microbiology 2000
K Yamada K Yoshino K Sekikawa H Madarame H Yagita A Nakane

Hydroxy acid-based matrix metalloproteinase (MMP) inhibitors have been shown to inhibit tumor infiltration and growth, endotoxin shock, and acute graft-versus-host disease. Blockade of the release of soluble tumor necrosis factor-alpha (TNF-alpha) and CD95 ligand (CD95L; FasL) from cell-associated forms is reportedly involved in the mechanism of the drug effect. We investigated the effect of a ...

2005
Joachim Roesler Jose-Maria Izquierdo Martin Ryser Angela Rösen-Wolff Manfred Gahr Juan Valcarcel Michael J. Lenardo Lixin Zheng Carl Gustav Carus

Autoimmune lymphoproliferative syndrome type Ia (ALPS Ia) is caused by mutations in the CD95/Apo-1/Fas (TNFRSF6) gene, which lead to a defective CD95 ligand (CD95L)induced apoptosis. Soluble CD95 (sCD95) has been suggested to play an important role in the pathogenesis of diverse autoimmune and malignant diseases by antagonizing CD95L. Here we evaluate a family with four in five of its members h...

2015
Caroline E. Broos Menno van Nimwegen Alex Kleinjan Bregje ten Berge Femke Muskens Johannes C.C.M. in ’t Veen Jouke T. Annema Bart N. Lambrecht Henk C. Hoogsteden Rudi W. Hendriks Mirjam Kool Bernt van den Blink

BACKGROUND Impaired regulatory T cell (Treg) function is thought to contribute to ongoing inflammatory responses in sarcoidosis, but underlying mechanisms remain unclear. Moreover, it is not known if increased apoptotic susceptibility of Tregs may contribute to an impaired immunosuppressive function in sarcoidosis. Therefore, the aim of this study is to analyze proportions, phenotype, survival,...

Journal: :Journal of leukocyte biology 2003
Rachid Marhaba Mehdi Bourouba Margot Zöller

Blockade of CD44v7 was described to cure trinitrobenzene sulfonic acid-induced colitis, a disease not developed by mice with targeted deletion of the CD44v7 exon. There was evidence for a reduction in activation-induced cell death on lamina propria lymphocytes of control as compared with CD44v7-deficient mice. To elucidate the mechanism underlying the relative apoptosis resistance of CD44v7-com...

Journal: :Journal of immunology 2005
Karsten Gülow Marcin Kaminski Katalin Darvas Dorothee Süss Min Li-Weber Peter H Krammer

Termination of an immune response requires elimination of activated T lymphocytes by activation-induced cell death (AICD). In AICD, CD95 (Apo-1/Fas) ligand (L) triggers apoptosis of CD95-positive activated T lymphocytes. In AIDS patients, AICD is strongly enhanced and accelerated. We and others have previously shown that HIV-1 trans-activator of transcription (HIV-1 Tat) sensitizes T cells towa...

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