نتایج جستجو برای: exon deletion

تعداد نتایج: 99871  

2017
Tao Li Zhao-jing Zhang Xin Ma Xue Lv Hai Xiao Qian-nan Guo Hong-yan Liu Hong-dan Wang Dong Wu Gui-yu Lou Xin Wang Chao-yang Zhang Shi-xiu Liao

BACKGROUND Patients with Duchenne muscular dystrophy (DMD) usually have severe and fatal symptoms. At present, there is no effective treatment for DMD, thus it is very important to avoid the birth of children with DMD by effective prenatal diagnosis. We identified a de novo DMD gene mutation in a Chinese family, and make a prenatal diagnosis. METHODS First, multiplex ligation-dependent probe ...

Journal: :Journal of medical genetics 1987
B Horsthemke A Dunning S Humphries

DNA samples from 70 unrelated UK patients with heterozygous familial hypercholesterolaemia were screened by Southern blot hybridisation with a 5' fragment of the human low density lipoprotein (LDL) receptor cDNA. In the majority of cases, the restriction fragment pattern of the LDL receptor gene was indistinguishable from that observed in normal subjects. However, three patients were found to h...

2014
Yingjie Liu Ruiwu Wang Bo Sun Tao Mi Jingqun Zhang Yongxin Mu Ju Chen Michael J. Bround James D. Johnson Anne M. Gillis S. R. Wayne Chen

A large genomic deletion in human cardiac ryanodine receptor (RYR2) gene has been detected in a number of unrelated families with various clinical phenotypes, including catecholaminergic polymorphic ventricular tachycardia (CPVT). This genomic deletion results in an in-frame deletion of exon-3 (Ex3-del). To understand the underlying disease mechanism of the RyR2 Ex3-del mutation, we generated a...

Journal: :The Journal of clinical investigation 1998
J Bloomer C Bruzzone L Zhu Y Scarlett S Magness D Brenner

Protoporphyria is a genetic disorder in which a deficiency of mitochondrial ferrochelatase activity causes accumulation of protoporphyrin that produces severe liver damage in some patients. In this study, mutations of the ferrochelatase gene were examined in eight unrelated patients who had liver transplantation. RNA was prepared from liver and/ or lymphoblasts, and specific reverse transcripta...

Journal: :Clinical chemistry 2006
Roger D Klein Erik C Thorland Patrick R Gonzales Patricia A Beck Daniel J Dykas James M McGrath Allen E Bale

BACKGROUND Glycerol kinase deficiency (GKD) is an X-linked recessive disorder that presents in both isolated and complex forms. The contiguous deletion that leads to GKD also commonly affects NR0B1 (DAX1), the gene associated with adrenal hypoplasia congenita, and DMD, the Duchenne muscular dystrophy gene. Molecular testing to delineate this deletion is expensive and has only limited availabili...

2016
Dan Pu Hao Liang Fang Wei David Akin Ziding Feng QingXiang Yan Yin Li Yan Zhen Lin Xu Gaochao Dong Huajing Wan Jingsi Dong Xiaoming Qiu Changlong Qin Daxing Zhu Xi Wang Tong Sun Wenbiao Zhang Canjun Li Xiaojun Tang Youlin Qiao David T.W. Wong Qinghua Zhou

BACKGROUND This article describes a pilot study evaluating a novel liquid biopsy system for non-small cell lung cancer (NSCLC) patients. The electric field-induced release and measurement (EFIRM) method utilizes an electrochemical biosensor for detecting oncogenic mutations in biofluids. METHODS Saliva and plasma of 17 patients were collected from three cancer centers prior to and after surgi...

Journal: :iranian journal of public health 0
s kheradmand kia dd farhud s zeinali ar mowjoodi h najmabadi f pourfarzad

duchenne muscular dystrophy (dmd) and the milder allelic becker muscular dystrophy (bmd) are x-linked disorders. both dmd & bmd result from heterogenous mutation in the dystrophin gene and in about 65% of the cases one or more exons of the gene are deleted or duplicated. one third of cases arise from new mutation and the rest are familial. to analyze the prevalence of deletion in iranian patien...

2016
Lei Zhuang Simon D. Gerber Stefan Kuchen Peter M. Villiger Beat Trueb

Multiple osteochondromas (also called hereditary multiple exostoses) is an autosomal dominant disorder characterized by multiple cartilaginous tumors, which are caused by mutations in the genes for exostosin-1 (EXT1) and exostosin-2 (EXT2). The goal of this study was to elucidate the genetic alterations in a family with three affected members. Isolation of RNA from the patients' blood followed ...

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