نتایج جستجو برای: fingolimod fty720

تعداد نتایج: 1720  

Journal: :Drug metabolism and disposition: the biological fate of chemicals 2011
Yi Jin Markus Zollinger Hubert Borell Alfred Zimmerlin Christopher J Patten

Fingolimod (FTY720, Gilenya, 2-amino-2-[2-(4-octylphenyl)ethyl]-1,3-propanediol) is a novel drug recently approved in the United States for the oral treatment of relapsing multiple sclerosis. The compound is eliminated predominantly by ω-hydroxylation, followed by further oxidation. The ω-hydroxylation was the major metabolic pathway in human liver microsomes (HLM). The enzyme kinetics in HLM w...

Journal: :Brain, Behavior, and Immunity 2016
Meray Serdar Josephine Herz Karina Kempe Katharina Lumpe Barbara S. Reinboth Stéphane V. Sizonenko Xinlin Hou Ralf Herrmann Martin Hadamitzky Rolf Heumann Wiebke Hansen Marco Sifringer Yohan van de Looij Ursula Felderhoff-Müser Ivo Bendix

Cerebral white matter injury is a leading cause of adverse neurodevelopmental outcome in prematurely born infants involving cognitive deficits in later life. Despite increasing knowledge about the pathophysiology of perinatal brain injury, therapeutic options are limited. In the adult demyelinating disease multiple sclerosis the sphingosine-1-phosphate (S1P) receptor modulating substance fingol...

Journal: :JAMA neurology 2014
Ying Fu Junwei Hao Ningnannan Zhang Li Ren Na Sun Yu-Jing Li Yaping Yan DeRen Huang Chunshui Yu Fu-Dong Shi

IMPORTANCE Pronounced inflammatory reactions occurring shortly after intracerebral hemorrhage (ICH) contribute to the formation and progression of perihematomal edema (PHE) and secondary brain injury. We hypothesized that modulation of brain inflammation reduces edema, thus improving clinical outcomes in patients with ICH. OBJECTIVE To investigate whether oral administration of fingolimod, a ...

2016
Laurent M. Willems Nadine Zahn Nerea Ferreirós Klaus Scholich Nicola Maggio Thomas Deller Andreas Vlachos

A hallmark of several major neurological diseases is neuronal cell death. In addition to this primary pathology, secondary injury is seen in connected brain regions in which neurons not directly affected by the disease are denervated. These transneuronal effects on the network contribute considerably to the clinical symptoms. Since denervated neurons are viable, they are attractive targets for ...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2017
Veit Rothhammer Jessica E Kenison Emily Tjon Maisa C Takenaka Kalil Alves de Lima Davis M Borucki Chun-Cheih Chao Annabel Wilz Manon Blain Luke Healy Jack Antel Francisco J Quintana

Multiple sclerosis (MS) is an autoimmune inflammatory demyelinating disease of the CNS that causes disability in young adults as a result of the irreversible accumulation of neurological deficits. Although there are potent disease-modifying agents for its initial relapsing-remitting phase, these therapies show limited efficacy in secondary progressive MS (SPMS). Thus, there is an unmet clinical...

2017
Kai Cui Yajun Ruan Zhe Tang Ke Rao Tao Wang Shaogang Wang Zhong Chen Jihong Liu

INTRODUCTION Erectile dysfunction (ED) in patients with diabetes mellitus (DM) seriously affects their quality of life. However, these patients show a poor effect rate for oral phosphodiesterase type 5 inhibitors. Thus, new treatment methods are urgently needed. Fingolimod hydrochloride (FTY720) was approved in 2010 for the treatment of patients with the relapsing-remitting form of multiple scl...

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