نتایج جستجو برای: mybpc3

تعداد نتایج: 307  

Journal: :Circulation. Cardiovascular genetics 2011
Christiane Gruner Melanie Care Katherine Siminovitch Gil Moravsky E Douglas Wigle Anna Woo Harry Rakowski

BACKGROUND Apical hypertrophic cardiomyopathy (HCM) is a unique form of HCM with left ventricular hypertrophy confined to the cardiac apex. The purpose of our study was to report genetic findings in a large series of unrelated patients with apical HCM and compare them with a nonapical HCM cohort. METHODS AND RESULTS Overall, 429 patients with HCM underwent genetic testing. The panel included ...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2017
Kaoru Ito Parth N Patel Joshua M Gorham Barbara McDonough Steven R DePalma Emily E Adler Lien Lam Calum A MacRae Syed M Mohiuddin Diane Fatkin Christine E Seidman J G Seidman

Genetic variants that cause haploinsufficiency account for many autosomal dominant (AD) disorders. Gene-based diagnosis classifies variants that alter canonical splice signals as pathogenic, but due to imperfect understanding of RNA splice signals other variants that may create or eliminate splice sites are often clinically classified as variants of unknown significance (VUS). To improve recogn...

Journal: :Journal of Muscle Research and Cell Motility 2011

2013
Yilu Wang Zhimin Wang Qi Yang Yubao Zou Hongju Zhang Chaowu Yan Xinxing Feng Yi Chen Yin Zhang Jizheng Wang Xianliang Zhou Ferhaan Ahmad Rutai Hui Lei Song

BACKGROUND Hypertrophic cardiomyopathy (HCM) due to mutations in genes encoding sarcomere proteins is most commonly inherited as an autosomal dominant trait. Since nearly 50% of HCM cases occur in the absence of a family history, a recessive inheritance pattern may be involved. METHODS A pedigree was identified with suspected autosomal recessive transmission of HCM. Twenty-six HCM-related gen...

2014
Cheryl Dambrot Stefan R Braam Leon G J Tertoolen Matthew Birket Douwe E Atsma Christine L Mummery

It has been known for over 20 years that foetal calf serum can induce hypertrophy in cultured cardiomyocytes but this is rarely considered when examining cardiomyocytes derived from pluripotent stem cells (PSC). Here, we determined how serum affected cardiomyocytes from human embryonic- (hESC) and induced pluripotent stem cells (hiPSC) and hiPSC from patients with hypertrophic cardiomyopathy li...

2011
Maximilian G. Posch Stephan Waldmuller Melanie Müller Thomas Scheffold David Fournier Miguel A. Andrade-Navarro Bernard De Geeter Sophie Guillaumont Claire Dauphin Dany Yousseff Katharina R. Schmitt Andreas Perrot Felix Berger Roland Hetzer Patrice Bouvagnet Cemil Özcelik

Secundum-type atrial septal defects (ASDII) account for approximately 10% of all congenital heart defects (CHD) and are associated with a familial risk. Mutations in transcription factors represent a genetic source for ASDII. Yet, little is known about the role of mutations in sarcomeric genes in ASDII etiology. To assess the role of sarcomeric genes in patients with inherited ASDII, we analyze...

2017
Rita Mendes de Almeida Joana Tavares Sandra Martins Teresa Carvalho Francisco J Enguita Dulce Brito Maria Carmo-Fonseca Luís Rocha Lopes

BACKGROUND High throughput sequencing technologies have revolutionized the identification of mutations responsible for genetic diseases such as hypertrophic cardiomyopathy (HCM). However, approximately 50% of individuals with a clinical diagnosis of HCM have no causal mutation identified. This may be due to the presence of pathogenic mutations located deep within the introns, which are not dete...

Journal: :Clinical genetics 2014
S Fokstuen P Makrythanasis S Nikolaev F Santoni D Robyr A Munoz J Bevillard L Farinelli C Iseli S E Antonarakis J-L Blouin

Mendelian cardiomyopathies and arrhythmias are characterized by an important genetic heterogeneity, rendering Sanger sequencing very laborious and expensive. As a proof of concept, we explored multiplex targeted high-throughput sequencing (HTS) as a fast and cost-efficient diagnostic method for individuals suffering from Mendelian cardiac disorders. We designed a DNA capture assay including all...

Journal: :Human molecular genetics 2010
Christina A Gurnett David M Desruisseau Kevin McCall Ryan Choi Zachary I Meyer Michael Talerico Sara E Miller Jeong-Sun Ju Alan Pestronk Anne M Connolly Todd E Druley Conrad C Weihl Mathew B Dobbs

Distal arthrogryposis type I (DA1) is a disorder characterized by congenital contractures of the hands and feet for which few genes have been identified. Here we describe a five-generation family with DA1 segregating as an autosomal dominant disorder with complete penetrance. Genome-wide linkage analysis using Affymetrix GeneChip Mapping 10K data from 12 affected members of this family revealed...

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