نتایج جستجو برای: oxidative lung damage

تعداد نتایج: 627412  

Journal: :iranian journal of toxicology 0
معصومه معصومی کریمی masoomeh masoomi karimi department of immunology, torbateheydariyeh university of medical sciences, torbateheydariyeh, iran. مسلم جعفری ثانی moslem jafari sani department of clinical biochemistry, torbateheydariyeh, university of medical sciences, torbateheydariyeh, iran. علی زارعی محمودآبادی ali zaree mahmudabadi department of molecular biology center, baqiyatallah, university of medical sciences, tehran, iran. اسما جعفری ثانی asma jafari sani ms, biology research center, torbateheydariyeh, university of medical sciences, torbateheydariyeh, iran. سید رضا خطیبی seyed reza khatibi md & chief student of mph, faculty of torbatheydariyeh, university of medical sciences, torbatheydariyeh, iran.

background: cadmium is one of the most toxic heavy metals in our environment having a very strong ability to accumulate in body organs, especially in kidney. the present study was done to determine the genotoxicity and cytotoxicity in kidneys of rats exposed to cadmium. methods: male rats (n=30), kept in standard conditions were used in this study. the animals were randomly divided into 2 group...

Journal: :American journal of physiology. Lung cellular and molecular physiology 2013
Masahiro Hashizume Marc Mouner Joshua M Chouteau Olena M Gorodnya Mykhaylo V Ruchko Barry J Potter Glenn L Wilson Mark N Gillespie James C Parker

This study tested the hypothesis that oxidative mitochondrial-targeted DNA (mtDNA) damage triggered ventilator-induced lung injury (VILI). Control mice and mice infused with a fusion protein targeting the DNA repair enzyme, 8-oxoguanine-DNA glycosylase 1 (OGG1) to mitochondria were mechanically ventilated with a range of peak inflation pressures (PIP) for specified durations. In minimal VILI (1...

2014
Larissa Constantino Renata Casagrande Gonçalves Vinícius Renê Giombelli Cristiane Damiani Tomasi Francieli Vuolo Luiza Wilges Kist Giovanna Medeiros Tavares de Oliveira Matheus Augusto de Bittencourt Pasquali Maurício Reis Bogo Thais Mauad Adolfo Horn Karen V Melo Christiane Fernandes José Cláudio Fonseca Moreira Cristiane Ritter Felipe Dal-Pizzol

BACKGROUND The purpose of this research is to study the relationship between superoxide dismutase (SOD) and lung redox state in an animal model of sepsis. METHODS Sepsis was induced in rats by the cecal ligation and perforation model (CLP). After 3, 6, and 12 h, CLP protein content and expression of SOD1, SOD2, and SOD3 were evaluated, and SOD activity was assessed. Oxidative damage was deter...

2006
PNR Dekhuijzen WJC van Beurden

Oxidative stress has been implicated in the pathogenesis and progression of COPD. Both reactive oxidant species from inhaled cigarette smoke and those endogenously formed by inflammatory cells constitute an increased intrapulmonary oxidant burden. Structural changes to essential components of the lung are caused by oxidative stress, contributing to irreversible damage of both parenchyma and air...

Journal: :International journal of nanomedicine 2016
Abderrahim Nemmar Priya Yuvaraju Sumaya Beegam Javed Yasin Elsadig E Kazzam Badreldin H Ali

The use of amorphous silica (SiO2) in biopharmaceutical and industrial fields can lead to human exposure by injection, skin penetration, ingestion, or inhalation. However, the in vivo acute toxicity of amorphous SiO2 nanoparticles (SiNPs) on multiple organs and the mechanisms underlying these effects are not well understood. Presently, we investigated the acute (24 hours) effects of intraperito...

Introduction: Gaseous air pollutants can cause oxidative stress, which can lead to lung damage by inducing inflammation. Polymorphisms in the glutathione S-transferase (GST) gene are involved in the pathogenesis of many diseases, including lung disease. Two glutathione S-transferase Mu 1 (GSTM1) and glutathione S-transferase theta 1 (GSTT1) genes belong to this family, in which deletions occur ...

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