نتایج جستجو برای: periodic paralysis

تعداد نتایج: 101463  

2010
Stanislav Sokolov Todd Scheuer William A. Catterall

Hypokalemic periodic paralysis and normokalemic periodic paralysis are caused by mutations of the gating charge-carrying arginine residues in skeletal muscle Na(V)1.4 channels, which induce gating pore current through the mutant voltage sensor domains. Inward sodium currents through the gating pore of mutant R666G are only approximately 1% of central pore current, but substitution of guanidine ...

Journal: :Neurology 2004
T M Miller M R Dias da Silva H A Miller H Kwiecinski J R Mendell R Tawil P McManis R C Griggs C Angelini S Servidei J Petajan M C Dalakas L P W Ranum Y H Fu L J Ptácek

BACKGROUND Periodic paralyses and paramyotonia congenita are rare disorders causing disabling weakness and myotonia. Mutations in sodium, calcium, and potassium channels have been recognized as causing disease. OBJECTIVE To analyze the clinical phenotype of patients with and without discernible genotype and to identify other mutations in ion channel genes associated with disease. METHODS Th...

Journal: :The Journal of clinical investigation 2012
Fenfen Wu Wentao Mi Erick O Hernández-Ochoa Dennis K Burns Yu Fu Hillery F Gray Arie F Struyk Martin F Schneider Stephen C Cannon

Hypokalemic periodic paralysis (HypoPP) is a familial skeletal muscle disorder that presents with recurrent episodes of severe weakness lasting hours to days associated with reduced serum potassium (K+). HypoPP is genetically heterogeneous, with missense mutations of a calcium channel (Ca(V)1.1) or a sodium channel (Na(V)1.4) accounting for 60% and 20% of cases, respectively. The mechanistic li...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2000
A F Struyk K A Scoggan D E Bulman S C Cannon

Missense mutations of the human skeletal muscle voltage-gated Na channel (hSkM1) underlie a variety of diseases, including hyperkalemic periodic paralysis (HyperPP), paramyotonia congenita, and potassium-aggravated myotonia. Another disorder of sarcolemmal excitability, hypokalemic periodic paralysis (HypoPP), which is usually caused by missense mutations of the S4 voltage sensors of the L-type...

Journal: :Journal of the American Society of Nephrology 2012

Journal: :Neurology 2012
Christoph Lossin Tai-Seung Nam Shahab Shahangian Michael A Rogawski Seok-Yong Choi Myeong-Kyu Kim Il-Nam Sunwoo

OBJECTIVE To electrophysiologically characterize the Na(v)1.4 mutant N440K found in a Korean family with a syndrome combining symptoms of paramyotonia congenita, hyperkalemic periodic paralysis, and potassium-aggravated myotonia. METHODS We characterized transiently expressed wild-type and mutant Na(v)1.4 using whole-cell voltage-clamp analysis. RESULTS N440K produced a significant depolari...

2014
Abhishek Vijayakumar Giridhar Ashwath Durganna Thimmappa

Thyrotoxic periodic paralysis (TPP), a disorder most commonly seen in Asian men, is characterized by abrupt onset of hypokalemia and paralysis. The condition primarily affects the lower extremities and is secondary to thyrotoxicosis. The underlying hyperthyroidism is often subtle causing difficulty in early diagnosis. Factors like high-carbohydrate meal exercise, steroid, and stress can precipi...

Journal: :Critical care medicine 2006
Shih-Hua Lin Pauling Chu Chih-Jen Cheng Shi-Jye Chu Yi-Jen Hung Yuh-Feng Lin

OBJECTIVES To identify a clinically reliable index of thyrotoxic periodic paralysis (TPP), a life-threatening emergency with unique and effective therapies. DESIGN Diagnostic study. SETTING University teaching hospital. PATIENTS Fifty-three consecutive patients with hypokalemic paralysis during a 3-yr period and 30 thyrotoxic patients without paralysis as the thyrotoxic control group. I...

Journal: :Journal of neurology, neurosurgery, and psychiatry 1992
I M Kantola L T Tarssanen

Seven families with familial hypokalaemic periodic paralysis were found in Finland. Nine of the 103 asymptomatic family members studied had abnormal results on a potassium exercise test. The overall prevalence of familial hypokalaemic periodic paralysis in Finland was 0.4/100,000. Carbohydrate intake and hard exercise were the most important triggers of paralytic attacks. Half of the patients r...

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