نتایج جستجو برای: antiinflammatory agents

تعداد نتایج: 362305  

Journal: :Circulation 2004
Uwe Schönbeck Peter Libby

According to traditional thinking, atherosclerosis results from passive lipid deposition in the vascular wall. Thus, therapies predominantly targeted lipid metabolism. The contemporary view of atherosclerosis, however, has broadened to include an active and complex role for inflammation, orchestrated in part by mediators of the immune system. This recognition prompted the question of whether an...

2006
G. D. GUPTA R. S. GAUD

Tenoxicam is a nonsteroidal antiinflammatory drug, used in the treatment of inflammatory and degenerative disorders of the musculoskeletal system. It is from the oxicam group of nonsteroidal antiinflammatory agents. It is widely prescribed for the treatment of osteoarthritis, rheumatoid arthritis, ankylosing spondylitis, gout, extraarticular disorders, bursitis, tendonitis, and nonarticular rhe...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1994
A Ray K E Prefontaine

Glucocorticoids, which are widely used as antiinflammatory agents, downregulate the expression of the interleukin 6 gene and of additional cytokine genes involved in inflammatory responses. Conversely, the transcription factor NF-kappa B, a member of the Rel family of transcription factors, has been implicated in the induction of multiple genes involved in the early processes of immune and infl...

2016
Michael J D Daniels Jack Rivers-Auty Tom Schilling Nicholas G Spencer William Watremez Victoria Fasolino Sophie J Booth Claire S White Alex G Baldwin Sally Freeman Raymond Wong Clare Latta Shi Yu Joshua Jackson Nicolas Fischer Violette Koziel Thierry Pillot James Bagnall Stuart M Allan Pawel Paszek James Galea Michael K Harte Claudia Eder Catherine B Lawrence David Brough

Non-steroidal anti-inflammatory drugs (NSAIDs) inhibit cyclooxygenase-1 (COX-1) and COX-2 enzymes. The NLRP3 inflammasome is a multi-protein complex responsible for the processing of the proinflammatory cytokine interleukin-1β and is implicated in many inflammatory diseases. Here we show that several clinically approved and widely used NSAIDs of the fenamate class are effective and selective in...

Journal: :Pediatrics 2013
Nitin Kapur Navid Adib Keith Grimwood

Nocardia are ubiquitous environmental saprophytes that cause pneumonia and disseminated disease in immunocompromised patients. They can also cause localized cutaneous and soft tissue infections in healthy people after direct percutaneous inoculation. Nocardia arthritis is rare in both forms of the disease. Here we present the first published case of a child with septic arthritis caused by N bra...

Journal: :The Journal of clinical investigation 2008
Kai Kessenbrock Leopold Fröhlich Michael Sixt Tim Lämmermann Heiko Pfister Andrew Bateman Azzaq Belaaouaj Johannes Ring Markus Ollert Reinhard Fässler Dieter E Jenne

Neutrophil granulocytes form the body's first line of antibacterial defense, but they also contribute to tissue injury and noninfectious, chronic inflammation. Proteinase 3 (PR3) and neutrophil elastase (NE) are 2 abundant neutrophil serine proteases implicated in antimicrobial defense with overlapping and potentially redundant substrate specificity. Here, we unraveled a cooperative role for PR...

2015
Ylenia Ingrasciotta Janet Sultana Francesco Giorgianni Andrea Fontana Antonio Santangelo Daniele Ugo Tari Domenico Santoro Vincenzo Arcoraci Margherita Perrotta Luisa Ibanez Gianluca Trifirò

BACKGROUND Non-steroidal anti-inflammatory agents (NSAIDs) are known to be associated with renal damage. No clear evidence exists regarding differential risk of chronic kidney disease (CKD), specifically, across various NSAIDs. AIM The aim of this population-based case-control study was to evaluate the association between use of individual NSAIDs and risk of CKD in a general population of Sou...

Journal: :The Biochemical journal 1999
J K Gierse C M Koboldt M C Walker K Seibert P C Isakson

Non-steroidal anti-inflammatory drugs (NSAIDs) inhibit the formation of prostaglandins by cyclo-oxygenases (COX). The discovery of a second COX isoform (COX-2) associated with inflammation led to agents that selectively inhibit COX-2, e.g. celecoxib. We evaluated the kinetics of inhibition of celecoxib and several NSAIDs. Celecoxib displays classic competitive kinetics on COX-1 (Ki=10-16 microM...

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