نتایج جستجو برای: drug accumulation

تعداد نتایج: 726518  

Journal: :The Journal of Cell Biology 2000
Yu Chen Sanford M. Simon

While P-glycoprotein (Pgp) is the most studied protein involved in resistance to anti-cancer drugs, its mechanism of action is still under debate. Studies of Pgp have used cell lines selected with chemotherapeutics which may have developed many mechanisms of resistance. To eliminate the confounding effects of drug selection on understanding the action of Pgp, we studied cells transiently transf...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1994
S Simon D Roy M Schindler

Many anticancer drugs are classified as either weak bases or molecules whose binding to cellular structures is pH dependent. Accumulation of these drugs within tumor cells should be affected by transmembrane pH gradients. Indeed, development of multidrug resistance (MDR) in tumor cells has been correlated with an alkaline shift of cytosolic pH. To examine the role of pH in drug partitioning, th...

2007

The human ovarian cancer cell lines A2780 and A2780/CP70 were studied to investigate the cellular basis for their relative sensitivities to tetrachloro(DL-trans)-l,2-diamminecyclohexaneplatinum(IV) (ormaplatin). Cells were exposed to ormaplatin for 1 h in all experiments. As assessed by colony formation assays, the A2780/CP70 cell line [50% inhibitory dose (ICso) = 3.6 pM] was 9.5-fold more res...

2002
Brian K. Gehlbach John P. Kress

Although the administration of sedatives is a commonplace activity in the ICU, few guidelines are available to aid the clinician in this practice. The first principle of sedative administration is to define the specific problem requiring sedation and to rationally choose the drug and depth of sedation appropriate for the indication. Next, the clinician must recognize the diverse and often unpre...

2017
Zhenyou Zou Ruyi Zou Dan Zong Yonghong Shi Jinyao Chen Jie Huang Jiahui Zhu Liguan Chen Xiaoyan Bao Yuan Liu Weihao Liu Wenhui Huang Jingsang Hu Zhi Chen Xiaojie Lao Chaoqun Chen Xiaoli Huang Yao Lu Xueyin Ni Daoquan Fang Dengqiang Wu Shuangshuang Lu Mingzhu Jiang Chenyang Qiu Yuya Wu Qisha Qiu Yanyuan Dong Yangyang Su Chenmin Zhao Zhihe Zhong Jing Cai Yong Liang

MDR1 is highly expressed in MDR A2780DX5 ovarian cancer cells, MDR SGC7901R gastric cancer cells and recurrent tumours. It pumps cytoplasmic agents out of cells, leading to decreased drug accumulation in cells and making cancer cells susceptible to multidrug resistance. Here, we identified that miR-495 was predicted to target ABCB1, which encodes protein MDR1. To reduce the drug efflux and reve...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1994
G J Zaman M J Flens M R van Leusden M de Haas H S Mülder J Lankelma H M Pinedo R J Scheper F Baas H J Broxterman

The multidrug-resistance associated protein MRP is a 180- to 195-kDa membrane protein associated with resistance of human tumor cells to cytotoxic drugs. We have investigated how MRP confers drug resistance in SW-1573 human lung carcinoma cells by generating a subline stably transfected with an expression vector containing MRP cDNA. MRP-overexpressing SW-1573 cells are resistant to doxorubicin,...

2012
Janni Papakrivos Juliana M. Sá Thomas E. Wellems

BACKGROUND Chloroquine (CQ)-resistant Plasmodium falciparum malaria has been a global health catastrophe, yet much about the CQ resistance (CQR) mechanism remains unclear. Hallmarks of the CQR phenotype include reduced accumulation of protonated CQ as a weak base in the digestive vacuole of the erythrocyte-stage parasite, and chemosensitization of CQ-resistant (but not CQ-sensitive) P. falcipar...

Journal: :The Journal of biological chemistry 1994
S Ruetz P Gros

We expressed P-glycoproteins (P-gps) encoded by the three mouse mdr genes in the membranes of secretory vesicles (SV) accumulating in the yeast mutant strain sec 6-4. Expression of the Mdr1 and Mdr3 isoforms in SV membranes caused a significant increased accumulation of the drug vinblastine (VBL) over background levels measured in control SV. The Mdr1/Mdr3-mediated increased drug accumulation c...

Journal: :Drug metabolism and disposition: the biological fate of chemicals 2016
Ryota Takeuchi Kohki Shinozaki Takeo Nakanishi Ikumi Tamai

Clinical reports indicate that cardiotoxicity due to donepezil can occur after coadministration with cilostazol. We speculated that the concentration of donepezil in heart tissue might be increased as a result of interaction with cilostazol at efflux transporters such as P-glycoprotein (P-gp, ABCB1) and breast cancer resistance protein (BCRP, ABCG2), which are expressed in many tissues includin...

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