نتایج جستجو برای: etomoxir

تعداد نتایج: 143  

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2002
Jagan N Thupari Leslie E Landree Gabriele V Ronnett Francis P Kuhajda

C75, a known inhibitor of fatty acid synthase is postulated to cause significant weight loss through decreased hypothalamic neuropeptide Y (NPY) production. Peripherally, C75, an alpha-methylene-gamma-butyrolactone, reduces adipose tissue and fatty liver, despite high levels of malonyl-CoA. To investigate this paradox, we studied the effect of C75 on fatty acid oxidation and energy production i...

2016
Ming-Da Wang Han Wu Shuai Huang Hui-Lu Zhang Chen-Jie Qin Ling-Hao Zhao Gong-Bo Fu Xu Zhou Xian-Ming Wang Liang Tang Wen Wen Wen Yang Shan-Hua Tang Dan Cao Lin-Na Guo Min Zeng Meng-Chao Wu He-Xin Yan Hong-Yang Wang

Due to a high rate of nutrient consumption and inadequate vascularization, hepatocellular carcinoma (HCC) cells constantly undergo metabolic stress during tumor development. Hepatitis B virus (HBV) X protein (HBx) has been implicated in the pathogenesis of HBV-induced HCC. In this study, we investigated the functional roles of HBx in HCC adaptation to metabolic stress. Up-regulation of HBx incr...

Journal: :Journal of vascular research 2012
Craig C Beeson Gyda C Beeson Haley Buff Juanita Eldridge Aiguo Zhang Arun Seth Marina Demcheva John N Vournakis Robin C Muise-Helmericks

BACKGROUND Poly-N-acetyl glucosamine nanofibers derived from a marine diatom have been used to increase cutaneous wound healing. These nanofibers exert their activity by specifically activating integrins, which makes them a useful tool for dissecting integrin-mediated pathways. We have shown that short-fiber poly-N-acetyl glucosamine nanofiber (sNAG) treatment of endothelial cells results in in...

Journal: :The Journal of Biological Chemistry 2009
Seiji Nakamura Toshinari Takamura Naoto Matsuzawa-Nagata Hiroaki Takayama Hirofumi Misu Hiroyo Noda Satoko Nabemoto Seiichiro Kurita Tsuguhito Ota Hitoshi Ando Ken-ichi Miyamoto Shuichi Kaneko

Visceral adiposity in obesity causes excessive free fatty acid (FFA) flux into the liver via the portal vein and may cause fatty liver disease and hepatic insulin resistance. However, because animal models of insulin resistance induced by lipid infusion or a high fat diet are complex and may be accompanied by alterations not restricted to the liver, it is difficult to determine the contribution...

Journal: :American journal of physiology. Endocrinology and metabolism 2013
Francisco Visiedo Fernando Bugatto Viviana Sánchez Irene Cózar-Castellano Jose L Bartha Germán Perdomo

Placentas of women with gestational diabetes mellitus (GDM) exhibit an altered lipid metabolism. The mechanism by which GDM is linked to alterations in placental lipid metabolism remains obscure. We hypothesized that high glucose levels reduce mitochondrial fatty acid oxidation (FAO) and increase triglyceride accumulation in human placenta. To test this hypothesis, we measured FAO, fatty acid e...

Journal: :The British journal of nutrition 2009
Blandine Gatta Christine Zuberbuehler Myrtha Arnold Roberte Aubert Wolfgang Langhans Didier Chapelot

The role of NEFA in eating behaviour is still poorly known. Our objective was to examine whether etomoxir (ETO), an inhibitor of NEFA oxidation, or ( - )-hydroxycitrate (HCA), an inhibitor of lipogenesis which may indirectly stimulate NEFA oxidation, alters satiety. Post-lunch satiety was measured in eight normal-weight male subjects who were deprived of time cues and received on three occasion...

2012
Shan Jiang Wei Wang Jess Miner Michael Fromm

Trans-10, cis-12 conjugated linoleic acid (t10c12 CLA) reduces triglyceride (TG) levels in adipocytes through multiple pathways, with AMP-activated protein kinase (AMPK) generally facilitating, and peroxisome proliferator-activated receptor γ (PPARγ) generally opposing these reductions. Sirtuin 1 (SIRT1), a histone/protein deacetylase that affects energy homeostasis, often functions coordinatel...

2009
David Sebastián Maria Guitart Celia García-Martínez Caroline Mauvezin Josep M. Orellana-Gavaldà Dolors Serra Anna M. Gómez-Foix Fausto G. Hegardt Guillermina Asins

Carnitine palmitoyltransferase 1 (CPT1) catalyzes the first step in long-chain fatty acid import into mitochondria, and it is believed to be rate limiting for beta-oxidation of fatty acids. However, in muscle, other proteins may collaborate with CPT1. Fatty acid translocase/CD36 (FAT/CD36) may interact with CPT1 and contribute to fatty acid import into mitochondria in muscle. Here, we demonstra...

2016
Yong Zhang Zhiyun Zhao Bilun Ke Lin Wan Hui Wang Jianping Ye

It is generally accepted that ATP regulates mitochondrial function through the AMPK signaling pathway. However, the AMPK-independent pathway remains largely unknown. In this study, we investigated ATP surplus in the negative regulation of mitochondrial function with a focus on pyruvate dehydrogenase (PDH) phosphorylation and protein acetylation. PDH phosphorylation was induced by a high fat die...

Journal: :The American journal of physiology 1998
Rhonda O Deems Robert C Anderson James E Foley

Increased fatty acid oxidation contributes to hyperglycemia in patients with non-insulin-dependent diabetes mellitus. To improve glucose homeostasis in these patients, we have designed a novel, reversible inhibitor of carnitine palmitoyltransferase I (CPT I) that potently inhibits fatty acid oxidation. SDZ-CPI-975 significantly lowered glucose levels in normal 18-h-fasted nonhuman primates and ...

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