نتایج جستجو برای: glutaric aciduria

تعداد نتایج: 1568  

2017
Nikolas Boy Jana Heringer Renate Brackmann Olaf Bodamer Angelika Seitz Stefan Kölker Inga Harting

BACKGROUND Without neonatal initiation of treatment, 80-90% of patients with glutaric aciduria type 1 (GA1) develop striatal injury during the first six years of life resulting in a complex, predominantly dystonic movement disorder. Onset of motor symptoms may be acute following encephalopathic crisis or insidious without apparent crisis. Additionally, so-called late-onset GA1 has been describe...

2009
Yuanquan Song Mary A. Selak Corey T. Watson Christopher Coutts Paul C. Scherer Jessica A. Panzer Sarah Gibbs Marion O. Scott Gregory Willer Ronald G. Gregg Declan W. Ali Michael J. Bennett Rita J. Balice-Gordon

In humans, mutations in electron transfer flavoprotein (ETF) or electron transfer flavoprotein dehydrogenase (ETFDH) lead to MADD/glutaric aciduria type II, an autosomal recessively inherited disorder characterized by a broad spectrum of devastating neurological, systemic and metabolic symptoms. We show that a zebrafish mutant in ETFDH, xavier, and fibroblast cells from MADD patients demonstrat...

2010
June Dong Park ByungChan Lim Ki Joong Kim Yong Seung Hwang Seung Ki Kim Seong-Ho Kang Sung Im Cho Sung Sup Park Joon Soo Lee Jong Hee Chae

Glutaric aciduria type I (GA I) is an autosomal recessive disorder caused by a deficiency of glutaryl-CoA dehydrogenase. Although over 400 patients confirmed as GA I have been reported, reports from the Asian population had contributed to the minor proportion. We recently diagnosed two cases of GA I confirmed with mutational analysis. Here, we present their rather atypical clinical presentation...

2017
Huihui Wang Wei Chen Xueqin Zhang Chuanfu Liu Runcang Sun

The esterification of bagasse with glutaric anhydride could increase surface adhesion compatibility and the surface of derived polymers has the potential of immobilizing peptides or proteins for biomedical application. Due to its complicated components, the esterification mechanism of bagasse esterified with glutaric anhydride in ionic liquids has not been studied. In this paper, the homogenous...

Journal: :Human molecular genetics 2008
Britta Keyser Chris Mühlhausen Achim Dickmanns Ernst Christensen Nicole Muschol Kurt Ullrich Thomas Braulke

Glutaric aciduria type 1 (GA1) is an autosomal recessive neurometabolic disorder caused by mutations in the glutaryl-CoA dehydrogenase gene (GCDH), leading to an accumulation and high excretion of glutaric acid and 3-hydroxyglutaric acid. Considerable variation in severity of the clinical phenotype is observed with no correlation to the genotype. We report here for the first time on expression ...

Journal: :Archives of Iranian medicine 2014
Masumeh Baradaran Hamid Galehdari Majid Aminzadeh Reza Azizi Malmiri Raheleh Tangestani Zahra Karimi

BACKGROUND Glutaric Aciduria type 1 (GA1) is a metabolic inborn error and is characterized by increasing excursion of glutaric acid and its derivates, presented in microcephaly and dystonia. The disease is resulted from mutational inactivation in the GCDH gene encoding the glutaryl-CoA dehydrogenase. The defective enzyme causes the accumulation of an excessive level of intermediate breakdown pr...

Journal: :Zhongguo dang dai er ke za zhi = Chinese journal of contemporary pediatrics 2016
Li-Fang Feng Xiao-Hong Chen Dong-Xiao Li Yuan Ding Ying Jin Jin-Qing Song Yan-Ling Yang

A one-year-old girl visited the hospital due to limb torsion and developmental regression for one month after hand, foot and mouth disease. At the age of 11 months, she visited a local hospital due to fever for 5 days and skin rash with frequent convulsions for 2 days and was diagnosed with severe hand, foot and mouth disease, viral encephalitis, and status epilepticus. Brain MRI revealed symme...

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