نتایج جستجو برای: lymphocyteassociated antigen 4 ctla

تعداد نتایج: 1469769  

Journal: :The Journal of Experimental Medicine 1992
P S Linsley J L Greene P Tan J Bradshaw J A Ledbetter C Anasetti N K Damle

T cell costimulation by molecules on the antigen presenting cell (APC) is required for optimal T cell proliferation. The B7 molecule on APC binds the T lymphocyte receptor CD28, triggering increased interleukin 2 (IL-2) production and subsequent T cell proliferation. CTLA-4 is a predicted T cell membrane receptor homologous to CD28, which also binds the B7 counter receptor, but whose distributi...

2014
Zineb Belcaid Jillian A. Phallen Jing Zeng Alfred P. See Dimitrios Mathios Chelsea Gottschalk Sarah Nicholas Meghan Kellett Jacob Ruzevick Christopher Jackson Emilia Albesiano Nicholas M. Durham Xiaobu Ye Phuoc T. Tran Betty Tyler John W. Wong Henry Brem Drew M. Pardoll Charles G. Drake Michael Lim

BACKGROUND Glioblastoma (GBM) is the most common malignant brain tumor in adults and is associated with a poor prognosis. Cytotoxic T lymphocyte antigen -4 (CTLA-4) blocking antibodies have demonstrated an ability to generate robust antitumor immune responses against a variety of solid tumors. 4-1BB (CD137) is expressed by activated T lymphocytes and served as a co-stimulatory signal, which pro...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2015
Chun Jing Wang Frank Heuts Vitalijs Ovcinnikovs Lukasz Wardzinski Chantelle Bowers Emily M Schmidt Alexandros Kogimtzis Rupert Kenefeck David M Sansom Lucy S K Walker

Cytotoxic T-lymphocyte-associated antigen-4 (CTLA-4) is an essential regulator of T-cell responses, and its absence precipitates lethal T-cell hyperactivity. However, whether CTLA-4 acts simply to veto the activation of certain clones or plays a more nuanced role in shaping the quality of T-cell responses is not clear. Here we report that T cells in CTLA-4-deficient mice show spontaneous T-foll...

Journal: :Journal of Autoimmune Diseases 2005
Sharad Purohit Robert Podolsky Christin Collins Weipeng Zheng Desmond Schatz Andy Muir Diane Hopkins Yi-Hua Huang Jin-Xiong She

BACKGROUND Cytotoxic T lymphocyte-associated antigen-4 (CTLA-4) plays a critical role in downregulation of antigen-activated immune response and polymorphisms at the CTLA-4 gene have been shown to be associated with several autoimmune diseases including type-1 diabetes (T1D). The etiological mutation was mapped to the CT60-A/G single nucleotide polymorphism (SNP) that is believed to control the...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2008
Jianda Yuan Sacha Gnjatic Hao Li Sarah Powel Humilidad F Gallardo Erika Ritter Geoffrey Y Ku Achim A Jungbluth Neil H Segal Teresa S Rasalan Gregor Manukian Yinyan Xu Ruth-Ann Roman Stephanie L Terzulli Melanie Heywood Evelina Pogoriler Gerd Ritter Lloyd J Old James P Allison Jedd D Wolchok

Blockade of inhibitory signals mediated by cytotoxic T lymphocyte-associated antigen 4 (CTLA-4) has been shown to enhance T cell responses and induce durable clinical responses in patients with metastatic melanoma. The functional impact of anti-CTLA-4 therapy on human immune responses is still unclear. To explore this, we analyzed immune-related adverse events and immune responses in metastatic...

2014
Michael Postow Manuarii Manuel Phillip Wong Jianda Yuan Marlene Noel Anais Courtier Nicolas Pasqual Jedd D Wolchok

Background Ipilimumab blocks the immunologic checkpoint cytotoxic T lymphocyte antigen-4 (CTLA-4) and improves overall survival in patients with metastatic melanoma. Since only a subset of patients achieves benefit from ipilimumab, biomarkers that can predict patient outcome are needed. CTLA-4 blockade has been shown to affect the peripheral T cell receptor (TCR) pool, but whether diversity of ...

Journal: :International immunology 2005
Hiroshi Kataoka Shigekazu Takahashi Kan Takase Sho Yamasaki Tadashi Yokosuka Takao Koike Takashi Saito

Cytotoxic T lymphocyte antigen-4 (CTLA-4) is constitutively expressed on CD25(+)CD4(+) regulatory T cells (Treg) and is suggested to play a role in Treg-mediated suppression. However, the results of analysis with anti-CTLA-4 have been controversial. We addressed this issue by analyzing mice over-expressing or deficient in CTLA-4. For over-expression, CTLA-4 transgenic mice expressing a full-len...

Cancer immunotherapy has gained a lot of interest over the past few years due to the success of immune checkpoint inhibitors in treating cancer (1, 2). Immune checkpoint inhibitors, such as monoclonal antibodies against cytotoxic T-lymphocyte–associated antigen 4 (CTLA-4) and programmed death 1 (PD-1), have been shown to increase survival of patients with advanced cancers (1, 2). These in...

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