نتایج جستجو برای: myofibrillar myopathy

تعداد نتایج: 14255  

2010
Ji-Man Hong Seung-Min Kim Il-Nam Sunwoo Se-Hoon Kim Tai-Seung Kim Dong-Suk Shim Young-Chul Choi

PURPOSE Nemaline myopathy (NM) is a clinical heterogeneous congenital myopathy characterized by the presence of subsarcolemmal or cytoplasmic rod-like structures that call nemaline bodies in the muscle fibers. The purpose of this study was to investigate the clinical diversity and pathological features of Korean patients with NM. MATERIALS AND METHODS Eight patients underwent analyses of clin...

Journal: :Neurology 2013
Ozge Ceyhan-Birsoy Pankaj B Agrawal Carlos Hidalgo Klaus Schmitz-Abe Elizabeth T DeChene Lindsay C Swanson Rachel Soemedi Nasim Vasli Susan T Iannaccone Perry B Shieh Natasha Shur Jane M Dennison Michael W Lawlor Jocelyn Laporte Kyriacos Markianos William G Fairbrother Henk Granzier Alan H Beggs

OBJECTIVE To identify causative genes for centronuclear myopathies (CNM), a heterogeneous group of rare inherited muscle disorders that often present in infancy or early life with weakness and hypotonia, using next-generation sequencing of whole exomes and genomes. METHODS Whole-exome or -genome sequencing was performed in a cohort of 29 unrelated patients with clinicopathologic diagnoses of ...

Journal: :Brain : a journal of neurology 2010
Christoph S Clemen Karthikeyan Tangavelou Karl-Heinz Strucksberg Steffen Just Linda Gaertner Hanna Regus-Leidig Maria Stumpf Jens Reimann Roland Coras Reginald O Morgan Maria-Pilar Fernandez Andreas Hofmann Stefan Müller Benedikt Schoser Franz-Georg Hanisch Wolfgang Rottbauer Ingmar Blümcke Stephan von Hörsten Ludwig Eichinger Rolf Schröder

Mutations of the human valosin-containing protein gene cause autosomal-dominant inclusion body myopathy associated with Paget disease of bone and frontotemporal dementia. We identified strumpellin as a novel valosin-containing protein binding partner. Strumpellin mutations have been shown to cause hereditary spastic paraplegia. We demonstrate that strumpellin is a ubiquitously expressed protein...

2016
A. Maerkens M. Olivé A. Schreiner S. Feldkirchner J. Schessl J. Uszkoreit K. Barkovits A. K. Güttsches V. Theis M. Eisenacher M. Tegenthoff L. G. Goldfarb R. Schröder B. Schoser P. F. M. van der Ven D. O. Fürst M. Vorgerd K. Marcus R. A. Kley

INTRODUCTION Myofibrillar myopathies are characterized by progressive muscle weakness and impressive abnormal protein aggregation in muscle fibers. In about 10 % of patients, the disease is caused by mutations in the MYOT gene encoding myotilin. The aim of our study was to decipher the composition of protein deposits in myotilinopathy to get new information about aggregate pathology. RESULTS ...

2013
Camilo Toro Montse Olivé Marinos C Dalakas Kumaraswami Sivakumar Juan M Bilbao Felix Tyndel Noemí Vidal Eva Farrero Nyamkhishig Sambuughin Lev G Goldfarb

BACKGROUND Hereditary myopathy with early respiratory failure (HMERF) was described in several North European families and recently linked to a titin gene (TTN) mutation. We independently studied HMERF-like diseases with the purpose to identify the cause, refine diagnostic criteria, and estimate the frequency of this disease among myopathy patients of various ethnic origins. METHODS Whole exo...

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