نتایج جستجو برای: ob gene

تعداد نتایج: 1148506  

Journal: :The Journal of biological chemistry 2003
Naoya Yahagi Hitoshi Shimano Takashi Matsuzaka Yuho Najima Motohiro Sekiya Yoshimi Nakagawa Tomohiro Ide Sachiko Tomita Hiroaki Okazaki Yoshiaki Tamura Yoko Iizuka Ken Ohashi Takanari Gotoda Ryozo Nagai Satoshi Kimura Shun Ishibashi Jun-Ichi Osuga Nobuhiro Yamada

The tumor suppressor p53 is a transcription factor that activates or represses its target genes after various genotoxic stresses. We have previously shown that sterol regulatory element-binding protein-1 (SREBP-1), a key transcriptional regulator of triglyceride synthesis, and the lipogenic enzymes under its control are markedly suppressed in adipocytes from genetically obese ob/ob mice. Here w...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2003
Gabriella Segal-Lieberman Richard L Bradley Efi Kokkotou Michael Carlson Daniel J Trombly Xiaomei Wang Sarah Bates Martin G Myers Jeffrey S Flier Eleftheria Maratos-Flier

Energy homeostasis is regulated by a complex network involving peripheral and central signals that determine food intake and energy expenditure. Melanin-concentrating hormone (MCH) plays an essential role in this process. Animals treated with MCH develop hyperphagia and obesity. Ablation of the prepro-MCH gene leads to a lean phenotype, as does ablation of the rodent MCH receptor, MCHR-1. MCH i...

Journal: :American journal of physiology. Endocrinology and metabolism 2006
Katsumi Iizuka Bonnie Miller Kosaku Uyeda

The transcription factor carbohydrate response element-binding protein (ChREBP) mediates insulin-independent, glucose-stimulated gene expression of multiple liver enzymes responsible for converting excess carbohydrate to fatty acids for long-term storage. To investigate ChREBP's role in the development of obesity and obesity-associated metabolic dysregulation, ChREBP-deficient mice were intercr...

Journal: :Molecular medicine 1996
K Giese W J Fantl C Vitt J C Stephans L Cousens M Wachowicz L T Williams

BACKGROUND Obesity, the condition of excessive accumulation of fat is a poorly understood disorder and is a risk factor for type II diabetes, hypertension, and hyperlipidaemia. Recently, a putative mouse obese gene was cloned and its product, termed ob protein, was shown to be involved in the regulation of body weight. MATERIALS AND METHODS Bacterial and insect cells were used for expression ...

Journal: :The Journal of clinical investigation 1997
N G Chen A G Swick D R Romsos

Hypersecretion of insulin from the pancreas is among the earliest detectable metabolic alterations in some genetically obese animals including the ob/ob mouse and in some obesity-prone humans. Since the primary cause of obesity in the ob/ob mouse is a lack of leptin due to a mutation in the ob gene, we tested the hypothesis that leptin targets a regulatory pathway in pancreatic islets to preven...

2010
Sara Becerril Amaia Rodríguez Victoria Catalán Neira Sáinz Beatriz Ramírez María Collantes Iván Peñuelas Javier Gómez-Ambrosi Gema Frühbeck

BACKGROUND Leptin and nitric oxide (NO) on their own participate in the control of non-shivering thermogenesis. However, the functional interplay between both factors in this process has not been explored so far. Therefore, the aim of the present study was to analyze the impact of the absence of the inducible NO synthase (iNOS) gene in the regulation of energy balance in ob/ob mice. METHODS A...

2008
Allan M. Ramirez David R. Nunley Mauricio Rojas Jesse Roman

Obliterative bronchiolitis (OB) and Bronchiolitis Obliterans Syndrome (BOS) are frequent complications in the lung transplant recipient, and are the leading cause of mortality after transplantation. The mechanisms responsible for OB remain elusive, but inflammatory and tissue remodeling responses are implicated. We hypothesized that alterations in markers of tissue remodeling in BALF of lung tr...

2003
S. A. Shore Y. M. Rivera-Sanchez I. N. Schwartzman R. A. Johnston

Shore, S. A., Y. M. Rivera-Sanchez, I. N. Schwartzman, and R. A. Johnston. Responses to ozone are increased in obese mice. J Appl Physiol 95: 938–945, 2003. First published June 6, 2003; 10.1152/japplphysiol.00336.2003.—Epidemiological data indicate an increased incidence of asthma in overweight adults and children. Ozone (O3) is a common trigger for asthma. Accordingly, the purpose of this stu...

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