نتایج جستجو برای: pd l2

تعداد نتایج: 79105  

2016
Hiroyuki Inoue Jae-Hyun Park Kazuma Kiyotani Makda Zewde Azusa Miyashita Masatoshi Jinnin Yukiko Kiniwa Ryuhei Okuyama Ryota Tanaka Yasuhiro Fujisawa Hiroshi Kato Akimichi Morita Jun Asai Norito Katoh Kenji Yokota Masashi Akiyama Hironobu Ihn Satoshi Fukushima Yusuke Nakamura

Immune checkpoint inhibitors blocking the interaction between programmed death-1 (PD-1) and PD-1 ligand-1 (PD-L1) are revolutionizing the cancer immunotherapies with durable clinical responses. Although high expression of PD-L1 in tumor tissues has been implicated to correlate with the better response to the anti-PD-1 therapies, this association has been controversial. In this study, to charact...

2014
LIJUAN HAN FEIFEI LIU RUPING LI ZHAOMING LI XINFENG CHEN ZHIYUAN ZHOU XUDONG ZHANG TENGPENG HU YI ZHANG KEN YOUNG SUKE SUN JIANGUO WEN MINGZHI ZHANG

Extranodal natural killer/T-cell lymphoma (ENKL) is marked by a profound cellular immune deficiency that may influence the capacity of T cells to extract an efficient antitumor immune response. It has been confirmed that the B7-CD28 pathway may promote tumor immune evasion by providing a negative regulatory signal. The current study analyzed the expression of programmed death 1 (PD-1)/programme...

2015
Yiping Yang

The PD pathway The cDNA of programmed cell death 1 (PD-1) was isolated in 1992 from a murine T cell hybridoma and a hematopoietic progenitor cell line undergoing apoptosis (1). Genetic ablation studies showed that deficiencies in PD-1 resulted in different autoimmune phenotypes in various mouse strains (2, 3). PD-1–deficient allogeneic T cells with transgenic T cell receptors exhibited augmente...

Journal: :The Journal of Experimental Medicine 2003
Alan D. Salama Tanuja Chitnis Jaime Imitola Mohammed Javeed I. Ansari Hisaya Akiba Fumihiko Tushima Miyuki Azuma Hideo Yagita Mohamed H. Sayegh Samia J. Khoury

Experimental autoimmune encephalomyelitis (EAE) is mediated by autoantigen-specific T cells dependent on critical costimulatory signals for their full activation and regulation. We report that the programmed death-1 (PD-1) costimulatory pathway plays a critical role in regulating peripheral tolerance in murine EAE and appears to be a major contributor to the resistance of disease induction in C...

Journal: :Dalton transactions 2017
Alpesh K Sharma Hemant Joshi Renu Bhaskar Satyendra Kumar Ajai K Singh

Schiff base ligands (L1: sulfated and L2: selenated) having a ferrocene core synthesized by reacting ferrocene-carboxaldehyde with 2-(phenylthio/seleno)ethylamine on treatment with Na2PdCl4 in the presence of NaOAc give cyclopalladated complexes [Pd(L1/L2-H)Cl] (1/2). Complex 1 of a sulfated Schiff base L1, on reacting with one equivalent of triphenylphosphine gives complex [Pd(L1-H)PPh3Cl] (3)...

2016
Emmanuel Stephen-Victor Varun Kumar Sharma Mrinmoy Das Anupama Karnam Chaitrali Saha Maxime Lecerf Caroline Galeotti Srinivas V. Kaveri Jagadeesh Bayry

The programed death-1 (PD-1)-programed death ligand-1 (PD-L1) and PD-L2 co-inhibitory pathway has been implicated in the evasion strategies of Mycobacterium tuberculosis. Specifically, M. tuberculosis-induced PD-L1 orchestrates expansion of regulatory T cells and suppression of Th1 response. However, the role of PD pathway in regulating Th17 response to M. tuberculosis has not been investigated...

Journal: :Arteriosclerosis, thrombosis, and vascular biology 2011
De-xiu Bu Margarite Tarrio Elena Maganto-Garcia George Stavrakis Goro Tajima James Lederer Petr Jarolim Gordon J Freeman Arlene H Sharpe Andrew H Lichtman

OBJECTIVE Programmed cell death-1 (PD-1) is a member of the CD28 superfamily that delivers negative signals on interaction with its 2 ligands, PD-L1 and PD-L2. We studied the contribution of the PD-1 pathway to regulation of T cells that promote atherosclerotic lesion formation and inflammation. METHODS AND RESULTS We show that compared with Ldlr-/- control mice, Pd1-/-Ldlr-/- mice developed ...

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