نتایج جستجو برای: ptch1

تعداد نتایج: 516  

Journal: :Mechanisms of Development 2009
Cristina Sanchez-Camacho Juan Alberto Ortega Soledad Alcantara Paola Bovolenta

Gli repressor formation via Protein kinase A (PKA) activation. In the current study, we generated a chondrocyte-specific knockout mouse strain of Suppressor of Fused (Sufu) and demonstrated that loss of Sufu leads to elevated expression of Hh target genes, Gli1 and Patched 1 (Ptch1), in the growth plate. Histological analysis illustrated an increase of proliferating cells and a reduction of the...

Journal: :Mechanisms of Development 2009
Cynthia L. Andoniadou Carles Gaston-Massuet Juan Pedro Martinez-Barbera

Gli repressor formation via Protein kinase A (PKA) activation. In the current study, we generated a chondrocyte-specific knockout mouse strain of Suppressor of Fused (Sufu) and demonstrated that loss of Sufu leads to elevated expression of Hh target genes, Gli1 and Patched 1 (Ptch1), in the growth plate. Histological analysis illustrated an increase of proliferating cells and a reduction of the...

2015
Marc Zuckermann Volker Hovestadt Christiane B. Knobbe-Thomsen Marc Zapatka Paul A. Northcott Kathrin Schramm Jelena Belic David T. W. Jones Barbara Tschida Branden Moriarity David Largaespada Martine F. Roussel Andrey Korshunov Guido Reifenberger Stefan M. Pfister Peter Lichter Daisuke Kawauchi Jan Gronych

In vivo functional investigation of oncogenes using somatic gene transfer has been successfully exploited to validate their role in tumorigenesis. For tumour suppressor genes this has proven more challenging due to technical aspects. To provide a flexible and effective method for investigating somatic loss-of-function alterations and their influence on tumorigenesis, we have established CRISPR/...

Journal: :Current Issues in Molecular Biology 2023

Basal cell nevus syndrome (BCNS, OMIM 109400) is a familial cancer characterized by the development of numerous basal cancers and various other developmental abnormalities, including epidermal cysts skin, calcified dural folds, keratocysts jaw, palmar plantar pits, ovarian fibromas, medulloblastomas, lymphomesenteric cysts, fetal rhabdomyomas. BCNS shows autosomal dominant inheritance caused mu...

2010
Jo Harris Naoko Takebe

acrjourna understanding of the anatomy and development of fetal and adult hair follicles and the molecular of the major embryonic pathways that regulate the hair follicle have led to exciting discoveries rning the development of basal cell carcinoma (BCC). These studies have shed light on the major f Sonic hedgehog (Shh) signaling and its interactions with the insulin-like growth factor (IGF) a...

2014
Winnie W. Lo Dushanthi Pinnaduwage Nalan Gokgoz Jay S. Wunder Irene L. Andrulis

Despite the importance of Hedgehog signaling in bone development, the relationship between Hedgehog pathway expression and osteosarcoma clinical characteristics and outcome has not been investigated. In this study of 43 high-grade human osteosarcoma samples, we detected high expression levels of the Hedgehog ligand gene, IHH, and target genes, PTCH1 and GLI1, in most samples. Further analysis i...

2015
Vesna Škodrić-Trifunović Mihailo Stjepanović Živorad Savić Miroslav Ilić Ivana Kavečan Jadranka Jovanović Privrodski Vesna Spasovski Maja Stojiljković Sonja Pavlović

Nevoid basal cell carcinoma syndrome (Gorlin syndrome) is a rare autosomal dominant disorder characterized by numerous basal cell carcinomas, keratocystic odontogenic tumors of the jaws, and diverse developmental defects. This disorder is associated with mutations in tumor suppressor gene Patched 1 (PTCH1). We present two patients with Gorlin syndrome, one sporadic and one familial. Clinical ex...

Journal: :Cell stem cell 2015
Shelby C Peterson Markus Eberl Alicia N Vagnozzi Abdelmadjid Belkadi Natalia A Veniaminova Monique E Verhaegen Christopher K Bichakjian Nicole L Ward Andrzej A Dlugosz Sunny Y Wong

Basal cell carcinoma (BCC) is characterized by frequent loss of PTCH1, leading to constitutive activation of the Hedgehog pathway. Although the requirement for Hedgehog in BCC is well established, the identity of disease-initiating cells and the compartments in which they reside remain controversial. By using several inducible Cre drivers to delete Ptch1 in different cell compartments in mice, ...

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