نتایج جستجو برای: ray repair cross complementing protein 3

تعداد نتایج: 3524314  

ژورنال: طب جنوب 2016
بهادری, محمدهادی, صالحی, زیور, مرزبند, سمیرا, مشایخی, فرهاد,

زمینه: X-ray Repair Cross Complementing group 1 (XRCC1) به عنوان یک پروتئین داربستی در ترمیم برداشت باز آلی (BER) و ترمیم شکست تک رشته DNA (SSBR) عمل می‌کند. پلی‌مورفیسم‌های این ژن موجب تغییر در بازده ترمیم می‌شوند که ممکن است زمینه ابتلای افراد به بیماری‌های مختلف را فراهم سازد. هدف از این پژوهش بررسی ارتباط بین پلی‌مورفیسم XRCC1 Arg194Trp و ناباروری ایدوپاتیک مردان در استان گیلان می­باشد. مو...

2017
Grazia Lazzari Maria Iole Natalicchio Angela Terlizzi Francesco Perri Giovanni Silvano

BACKGROUND There has recently been a strong interest in the inter-individual variation in normal tissue and tumor response to radiotherapy (RT), because tissue radiosensitivity seems to be under genetic control. Evidence is accumulating on the role of polymorphic genetic variants, such as single nucleotide polymorphisms (SNPs) that could influence normal tissue response after radiation. The mos...

Journal: :Cell 1995
Abdelilah Aboussekhra Maureen Biggerstaff Mahmud K.K Shivji Juhani A Vilpo Vincent Moncollin Vladimir N Podust Miroslava Protić Ulrich Hübscher Jean-Marc Egly Richard D Wood

Nucleotide excision repair is the principal way by which human cells remove UV damage from DNA. Human cell extracts were fractionated to locate active components, including xeroderma pigmentosum (XP) and ERCC factors. The incision reaction was then reconstituted with the purified proteins RPA, XPA, TFIIH (containing XPB and XPD), XPC, UV-DDB, XPG, partially purified ERCC1/XPF complex, and a fac...

Journal: :Journal of cell science 2007
Martijn S Luijsterburg Joachim Goedhart Jill Moser Hanneke Kool Bart Geverts Adriaan B Houtsmuller Leon H F Mullenders Wim Vermeulen Roel van Driel

Damage DNA binding protein 2 (DDB2) has a high affinity for UV-damaged DNA and has been implicated in the initial steps of global genome nucleotide excision repair (NER) in mammals. DDB2 binds to CUL4A and forms an E3 ubiquitin ligase. In this study, we have analyzed the properties of DDB2 and CUL4A in vivo. The majority of DDB2 and CUL4A diffuse in the nucleus with a diffusion rate consistent ...

2014
Kaiwu Xu Zhihui Chen Changjiang Qin Xinming Song

BACKGROUND Analysis using publicly available algorithms predicts that X-ray repair complementing defective repair in Chinese hamster cells 2 (XRCC2), a key component in the homologous recombination repair pathway, is a potential target of micro-ribonucleic acid-7 (miR-7). Some studies have shown that both miR-7 and XRCC2 are associated with cancer development. For this purpose, we searched for ...

Journal: :Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology 2002
Ruey-Hong Wong Chung-Li Du Jung-Der Wang Chang-Chuan Chan Jiin-Chyuan J Luo Tsun-Jen Cheng

Mutant p53 protein and anti-p53 antibody in circulating blood can be detectedamong individuals with mutations of the p53 tumor suppressor gene. Plasma mutant p53 protein and anti-p53 antibody have also been associated with vinyl chloride monomer (VCM) exposure, although the mechanism of VCM-related carcinogenesis remains unclear. Polymorphisms of metabolic and DNA repair genes have been implica...

2015
Ping Xue Lin Gao Sha Xiao Guopei Zhang Mingyang Xiao Qianye Zhang Xiao Zheng Yuan Cai Cuihong Jin Jinghua Yang Shengwen Wu Xiaobo Lu Qing-Yi Wei

OBJECTIVES Individual variations in the capacity of DNA repair machinery to relieve benzene-induced DNA damage may be the key to developing chronic benzene poisoning (CBP), an increasingly prevalent occupational disease in China. ERCC1 (Excision repair cross complementation group 1) is located on chromosome 19q13.2-3 and participates in the crucial steps of Nucleotide Excision Repair (NER); mor...

2010
Natsuko Kondo Akihisa Takahashi Koji Ono Takeo Ohnishi

The cytotoxic effects of alkylating agents are strongly attenuated by cellular DNA repair processes, necessitating a clear understanding of the repair mechanisms. Simple methylating agents form adducts at N- and O-atoms. N-methylations are removed by base excision repair, AlkB homologues, or nucleotide excision repair (NER). O(6)-methylguanine (MeG), which can eventually become cytotoxic and mu...

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