نتایج جستجو برای: sideroblastic anemia

تعداد نتایج: 56367  

2009
David M. Mintzer Shira N. Billet Lauren Chmielewski

Objective. Drugs can induce almost the entire spectrum of hematologic disorders, affecting white cells, red cells, platelets, and the coagulation system. This paper aims to emphasize the broad range of drug-induced hematological syndromes and to highlight some of the newer drugs and syndromes. Methods. Medline literature on drug-induced hematologic syndromes was reviewed. Most reports and revie...

Journal: :Sao Paulo medical journal = Revista paulista de medicina 2005
Marilena Oshiro Kimiyo Nonoyama Raimundo Antônio Gomes Oliveira Orlando Cesar de Oliveira Barretto

CONTEXT AND OBJECTIVE The coenzyme of aspartate aminotransferase is pyridoxal phosphate, generated from fresh vegetables containing pyridoxine. Vitamin B6-responsive sideroblastic anemia, myelofibrosis and Peyronies syndrome respond to high pyridoxine doses. The objective was to investigate the oral pyridoxine oral dose that would lead to maximized pyridoxal phosphate saturation of red cell asp...

Journal: :The hematology journal : the official journal of the European Haematology Association 2004
Anath Gafter-Gvili Miron Prokocimer William Breuer Ioav Zeev Cabantchik Chaim Hershko

INTRODUCTION The abnormalities in iron metabolism associated with megaloblastic anemia are rapidly reversed by B(12) therapy in pernicious anemia (PA). Although non-tranferrin-bound plasma iron (NTBI) was previously shown to be associated with severe iron overload, its origin is unknown. METHODS AND RESULTS Four patients with PA were studied before and after B(12) treatment. NTBI was measured...

2010
Mohammad Taghi Arzanian Aziz Eghbali Parvaneh Karimzade Mitra Ahmadi Massoud Houshmand Nima Rezaei

BACKGROUND Pearson syndrome (PS) is a rare multisystem mitochondrial disorder of hematopoietic system, characterized by refractory sideroblastic anemia, pancytopenia, exocrine pancreatic insufficiency, and variable neurologic, hepatic, renal, and endocrine failure. CASE PRESENTATION We describe a six-month-old female infant with Pearson marrow syndrome who presented with neurological manifest...

2016
Joshua E. Mangum Justin P. Hardee Dennis K. Fix Melissa J. Puppa Johnathon Elkes Diego Altomare Yelena Bykhovskaya Dean R. Campagna Paul J. Schmidt Anoop K. Sendamarai Hart G. W. Lidov Shayne C. Barlow Nathan Fischel-Ghodsian Mark D. Fleming James A. Carson Jeffrey R. Patton

Mitochondrial myopathy with lactic acidosis and sideroblastic anemia (MLASA) is an oxidative phosphorylation disorder, with primary clinical manifestations of myopathic exercise intolerance and a macrocytic sideroblastic anemia. One cause of MLASA is recessive mutations in PUS1, which encodes pseudouridine (Ψ) synthase 1 (Pus1p). Here we describe a mouse model of MLASA due to mutations in PUS1....

2011
Florent M. Martin Xiuling Xu Katharina von Löhneysen Timothy J. Gilmartin Jeffrey S. Friedman

BACKGROUND Mice irradiated and reconstituted with hematopoietic cells lacking manganese superoxide dismutase (SOD2) show a persistent hemolytic anemia similar to human sideroblastic anemia (SA), including characteristic intra-mitochondrial iron deposition. SA is primarily an acquired, clonal marrow disorder occurring in individuals over 60 years of age with uncertain etiology. METHODOLOGY/PRI...

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