نتایج جستجو برای: tumor necrosis factor superfamily

تعداد نتایج: 1221977  

Journal: :Hypertension 2010

Journal: :The Journal of Experimental Medicine 2000
Kaimei Song Yiguang Chen Rüdiger Göke Andreas Wilmen Cheryl Seidel Alexandra Göke Brendan Hilliard Youhai Chen

The tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) induces apoptosis of tumor cells but not normal cells; its role in normal nontransformed tissues is unknown. We report here that chronic blockade of TRAIL in mice exacerbated autoimmune arthritis, and that intraarticular TRAIL gene transfer ameliorated the disease. In vivo, TRAIL blockade led to profound hyperproliferation of s...

Journal: :The Journal of rheumatology. Supplement 2010
Edward C Keystone Carl F Ware

Tumor necrosis factor (TNF) plays a crucial role in the pathogenesis of immune-mediated inflammatory diseases (IMID). As a result, the inhibition of TNF is an important therapeutic avenue in the treatment of these pathophysiologically diverse disease states. This section discusses TNF, its receptors, and its role in immunoregulation and inflammation, as well as the currently available anti-TNF-...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1999
S M McWhirter S S Pullen J M Holton J J Crute M R Kehry T Alber

Tumor necrosis factor receptor superfamily members convey signals that promote diverse cellular responses. Receptor trimerization by extracellular ligands initiates signaling by recruiting members of the tumor necrosis factor receptor-associated factor (TRAF) family of adapter proteins to the receptor cytoplasmic domains. We report the 2.4-A crystal structure of a 22-kDa, receptor-binding fragm...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1999
H Nakano S Sakon H Koseki T Takemori K Tada M Matsumoto E Munechika T Sakai T Shirasawa H Akiba T Kobata S M Santee C F Ware P D Rennert M Taniguchi H Yagita K Okumura

TRAF5 [tumor necrosis factor (TNF) receptor-associated factor 5] is implicated in NF-kappaB and c-Jun NH(2)-terminal kinase/stress-activated protein kinase activation by members of the TNF receptor superfamily, including CD27, CD30, CD40, and lymphotoxin-beta receptor. To investigate the functional role of TRAF5 in vivo, we generated TRAF5-deficient mice by gene targeting. Activation of either ...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2003
Masahide Tone Yukiko Tone Elizabeth Adams Stephen F Yates Mark R Frewin Stephen P Cobbold Herman Waldmann

Recently, agonist antibodies to glucocorticoid-induced tumor necrosis factor receptor (GITR) (tumor necrosis factor receptor superfamily 18) have been shown to neutralize the suppressive activity of CD4+CD25+ regulatory T cells. It was anticipated that this would be the role of the physiological ligand. We have identified and expressed the gene for mouse GITR ligand and have confirmed that its ...

Journal: :Journal of cell science 2002
Jee Y Chung Young Chul Park Hong Ye Hao Wu

The tumor necrosis factor (TNF) receptor associated factors (TRAFs) have emerged as the major signal transducers for the TNF receptor superfamily and the interleukin-1 receptor/Toll-like receptor (IL-1R/TLR) superfamily. TRAFs collectively play important functions in both adaptive and innate immunity. Recent functional and structural studies have revealed the individuality of each of the mammal...

Journal: :مجله علوم اعصاب شفای خاتم 0
mahtab irani a. islamic azad university, mashhad branch, mashhad, iran b. member of mashhad neuroscience research group of islamic azad university, mashhad branch, mashhad, iran fatemeh yardehnavi a. islamic azad university, mashhad branch, mashhad, iran b. member of mashhad neuroscience research group of islamic azad university, mashhad branch, mashhad, iran zohreh j memari islamic azad university, mashhad branch, mashhad, iran

one of the most important problems in neuroscience researches is the understanding what is the communication between the immune system and central nervous system. proinflammatory factors play an important role in this communication. the dysregulation of proinflammatory factors such as cytokines and chemokines is a central feature in the development of neuroinflammation.one of the important  cyt...

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