نتایج جستجو برای: vemurafenib

تعداد نتایج: 1054  

2014
Carlo Capalbo Paolo Marchetti Anna Coppa Antonella Calogero Emanuela Anastasi Amelia Buffone Francesca Belardinilli Matteo Gulino Paola Frati Carlo Catalano Enrico Cortesi Giuseppe Giannini Alberto Gulino

As the knowledge on cancer genetic alterations progresses, it fosters the need for more personalized therapeutic intervention in modern cancer management. Recently, mutations in KRAS, BRAF, and PIK3CA genes have emerged as important mechanisms of resistance to EGFR-targeted therapy in metastatic colorectal cancer (mCRC). Here we report the first case of a mCRC patient whose disease had progress...

2014
Chung-Pu Wu Suresh V. Ambudkar

Melanoma is the most serious type of skin cancer and one of the most common cancers in the world. Advanced melanoma is often resistant to conventional therapies and has high potential for metastasis and low survival rates. Vemurafenib is a small molecule inhibitor of the BRAF serine-threonine kinase recently approved by the United States Food and Drug Administration to treat patients with metas...

2016
Brian P. Danysh Erin Y. Rieger Deepankar K. Sinha Caitlin V. Evers Gilbert J. Cote Maria E. Cabanillas Marie-Claude Hofmann

The BRAF V600E mutation is commonly observed in papillary thyroid cancer (PTC) and predominantly activates the MAPK pathway. Presence of BRAF V600E predicts increasing risk of recurrence and higher mortality rate, and treatment options for such patients are limited. Vemurafenib, a BRAF V600E inhibitor, is initially effective, but cells inevitably develop alternative mechanisms of pathway activa...

Journal: :Blood 2013
Julien Haroche Fleur Cohen-Aubart Jean-François Emile Laurent Arnaud Philippe Maksud Frédéric Charlotte Philippe Cluzel Aurélie Drier Baptiste Hervier Neïla Benameur Sophie Besnard Jean Donadieu Zahir Amoura

Histiocytoses are rare disorders of unknown origin with highly heterogeneous prognosis. BRAFV600E gain-of-function mutations have been observed in 57% of cases of Langerhans cell histiocytosis (LCH) and 54% of cases of Erdheim-Chester disease (ECD), but not in other types of histiocytoses. Targeted therapy with an inhibitor of mutated BRAF (vemurafenib) improves survival of patients with melano...

2015
Monika Joshi Shawn J. Rice Xin Liu Bruce Miller Chandra P. Belani

V-Raf Murine Sarcoma Viral Oncogene Homolog B (BRAF) mutated lung cancer is relatively aggressive and is resistant to currently available therapies. In a recent phase II study for patients with BRAF-V600E non-small cell lung cancer (NSCLC), BRAF V600E inhibitor demonstrated evidence of activity, but 30% of this selected group progressed while on treatment, suggesting a need for developing alter...

Journal: :The New England journal of medicine 2015
David M Hyman Igor Puzanov Vivek Subbiah Jason E Faris Ian Chau Jean-Yves Blay Jürgen Wolf Noopur S Raje Eli L Diamond Antoine Hollebecque Radj Gervais Maria Elena Elez-Fernandez Antoine Italiano Ralf-Dieter Hofheinz Manuel Hidalgo Emily Chan Martin Schuler Susan Frances Lasserre Martina Makrutzki Florin Sirzen Maria Luisa Veronese Josep Tabernero José Baselga

BACKGROUND BRAF V600 mutations occur in various nonmelanoma cancers. We undertook a histology-independent phase 2 "basket" study of vemurafenib in BRAF V600 mutation-positive nonmelanoma cancers. METHODS We enrolled patients in six prespecified cancer cohorts; patients with all other tumor types were enrolled in a seventh cohort. A total of 122 patients with BRAF V600 mutation-positive cancer...

Journal: :Archives of dermatology 2012
Victor Huang Donna Hepper Milan Anadkat Lynn Cornelius

BACKGROUND The development of a novel BRAF inhibitor, vemurafenib, has been associated with impressive tumor regression in patients with BRAF-positive stage IV melanoma. In the phase 3 clinical trials, dermatologic toxic effects associated with vemurafenib were described, namely, the development of eruptive squamous cell carcinomas. Herein, 3 cases are presented that highlight the development o...

2013
Harina Vin Sandra S Ojeda Grace Ching Marco L Leung Vida Chitsazzadeh David W Dwyer Charles H Adelmann Monica Restrepo Kristen N Richards Larissa R Stewart Lili Du Scarlett B Ferguson Deepavali Chakravarti Karin Ehrenreiter Manuela Baccarini Rosamaria Ruggieri Jonathan L Curry Kevin B Kim Ana M Ciurea Madeleine Duvic Victor G Prieto Stephen E Ullrich Kevin N Dalby Elsa R Flores Kenneth Y Tsai

Vemurafenib and dabrafenib selectively inhibit the v-Raf murine sarcoma viral oncogene homolog B1 (BRAF) kinase, resulting in high response rates and increased survival in melanoma. Approximately 22% of individuals treated with vemurafenib develop cutaneous squamous cell carcinoma (cSCC) during therapy. The prevailing explanation for this is drug-induced paradoxical ERK activation, resulting in...

Journal: :Cancer letters 2015
Inna Zubrilov Orit Sagi-Assif Sivan Izraely Tsipi Meshel Shlomit Ben-Menahem Ravit Ginat Metsada Pasmanik-Chor Clara Nahmias Pierre-Olivier Couraud Dave S B Hoon Isaac P Witz

V600E being the most common mutation in BRAF, leads to constitutive activation of the MAPK signaling pathway. The majority of V600E BRAF positive melanoma patients treated with the BRAF inhibitor vemurafenib showed initial good clinical responses but relapsed due to acquired resistance to the drug. The aim of the present study was to identify possible biomarkers associated with the emergence of...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2013
Mei Li M Kwong Bart Neyns James C Yang

The clinical strengths of immunotherapy and small-molecule inhibitors targeting the mitogen-activated protein kinase (MAPK) pathway appear to be largely complementary for the treatment of advanced melanoma. In current practice, most patients with BRAF V600 mutant melanomas will see both modalities. Several in vitro and in vivo studies suggest that combining immunotherapy with MAPK inhibition ma...

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