نتایج جستجو برای: androgen antagonists

تعداد نتایج: 74039  

Journal: :Cancer research 2005
Giuseppe Pandini Rossana Mineo Francesco Frasca Charles T Roberts Marco Marcelli Riccardo Vigneri Antonino Belfiore

In this study, we show that androgens up-regulate insulin-like growth factor-I receptor (IGF-IR) expression and sensitize prostate cancer cells to the biological effects of IGF-I. Both dihydrotestosterone and the synthetic androgen R1881 induced an approximately 6-fold increase in IGF-IR expression in androgen receptor (AR)-positive prostate cancer cells LNCaP. In accordance with IGF-IR up-regu...

Journal: :Cancer research 2003
Kazuo Nishimura Huei-Ju Ting Yasunori Harada Takashi Tokizane Norio Nonomura Hong-Yo Kang Hong-Chiang Chang Shuyuan Yeh Hiroshi Miyamoto Masaru Shin Katsuyuki Aozasa Akihiko Okuyama Chawnshang Chang

The partial agonist effect of antiandrogens has been well documented, and such effect is amplified by derived mutant androgen receptors (ARs) in prostate cancer cells. Here we report the identification of gelsolin (GSN) as an AR-associated protein. Hydroxyflutamide (HF), as well as androgens, can promote the interaction between AR and GSN in a dose-dependent manner. GSN interacts with AR DNA-bi...

2012
Frances Orton Erika Rosivatz Martin Scholze Andreas Kortenkamp

BACKGROUND Many pesticides in current use have recently been revealed as in vitro androgen receptor (AR) antagonists, but information about their combined effects is lacking. OBJECTIVE We investigated the combined effects and the competitive AR antagonism of pesticide mixtures. METHODS We used the MDA-kb2 assay to test a combination of eight AR antagonists that did not also possess AR agoni...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2011
Harsh Vardhan Jain Steven K Clinton Arvinder Bhinder Avner Friedman

Prostate cancer progression depends in part on the complex interactions between testosterone, its active metabolite DHT, and androgen receptors. In a metastatic setting, the first line of treatment is the elimination of testosterone. However, such interventions are not curative because cancer cells evolve via multiple mechanisms to a castrate-resistant state, allowing progression to a lethal ou...

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