نتایج جستجو برای: cd55

تعداد نتایج: 1341  

Journal: :Blood 2014
Antonio M Risitano Daniel Ricklin Yijun Huang Edimara S Reis Hui Chen Patrizia Ricci Zhuoer Lin Caterina Pascariello Maddalena Raia Michela Sica Luigi Del Vecchio Fabrizio Pane Florea Lupu Rosario Notaro Ranillo R G Resuello Robert A DeAngelis John D Lambris

Paroxysmal nocturnal hemoglobinuria (PNH) is characterized by complement-mediated intravascular hemolysis due to the lack of CD55 and CD59 on affected erythrocytes. The anti-C5 antibody eculizumab has proven clinically effective, but uncontrolled C3 activation due to CD55 absence may result in opsonization of erythrocytes, possibly leading to clinically meaningful extravascular hemolysis. We in...

2012
T. Naraoka Y. Ishibashi E. Tsuda Y. Yamamoto T. Kusumi I. Kakizaki S. Toh

OBJECTIVES This study aimed to investigate time-dependent gene expression of injured human anterior cruciate ligament (ACL), and to evaluate the histological changes of the ACL remnant in terms of cellular characterisation. METHODS Injured human ACL tissues were harvested from 105 patients undergoing primary ACL reconstruction and divided into four phases based on the period from injury to su...

2004
Elaine M. Sloand Lori Mainwaring Keyvan Keyvanfar Jichun Chen Jaroslaw Maciejewski Harvey G. Klein

In paroxysmal nocturnal hemoglobinuria (PNH), an acquired mutation of the PIGA gene results in the absence of glycosylphosphatidylinositol (GPI)–anchored cell surface membrane proteins in affected hematopoietic cells. Absence of GPI-anchored proteins on erythrocytes is responsible for their increased sensitivity to complement-mediated lysis, resulting in hemolytic anemia. Cell-to-cell transfer ...

Journal: :Blood 2004
Elaine M Sloand Lori Mainwaring Keyvan Keyvanfar Jichun Chen Jaroslaw Maciejewski Harvey G Klein Neal S Young

In paroxysmal nocturnal hemoglobinuria (PNH), an acquired mutation of the PIGA gene results in the absence of glycosylphosphatidylinositol (GPI)-anchored cell surface membrane proteins in affected hematopoietic cells. Absence of GPI-anchored proteins on erythrocytes is responsible for their increased sensitivity to complement-mediated lysis, resulting in hemolytic anemia. Cell-to-cell transfer ...

2007
Young Ok Kim Sun Woo Lim Can Li Hee Jung Kang Kyung Ohk Ahn Hyun Joo Yang Jung Yeon Ghee Su hyun Kim Jin Young Kim Bum Soon Choi Jin Kim Chul Woo Yang

PURPOSE Local activation of the complement system plays a role in target organ damage. The aim of our study was to investigate the influence of cyclosporine (CsA)- induced renal injury on the complement system in the kidney. MATERIALS AND METHODS Mice fed a low salt (0.01%) diet were treated with vehicle (VH, olive oil, 1 mL/kg/day) or CsA (30 mg/kg/day) for one or four weeks. Induction of ch...

Journal: :Journal of virology 2005
M Reza Nokhbeh Samir Hazra David A Alexander Ahmar Khan Morgan McAllister Erik J Suuronen May Griffith Kenneth Dimock

Enterovirus 70 (EV70), the causative agent of acute hemorrhagic conjunctivitis, exhibits a restricted tropism for conjunctival and corneal cells in vivo but infects a wide spectrum of mammalian cells in culture. Previously, we demonstrated that human CD55 is a receptor for EV70 on HeLa cells but that EV70 also binds to sialic acid-containing receptors on a variety of other human cell lines. Vir...

2000
John N. Waitumbi Malachi O. Opollo Richard O. Muga Ambrose O. Misore José A. Stoute

Severe anemia is one of the most lethal complications in children infected with Plasmodium falciparum. The pathogenesis of this anemia is not completely understood. Experimental data from malaria-infected humans and animal models suggest that uninfected red cells have a shortened life span. This study looked for changes in the red cell surfaces of children with severe malarial anemia that could...

Journal: :FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2006
Mark J Kwakkenbos Mourad Matmati Ole Madsen Walter Pouwels YongYi Wang Ronald E Bontrop Peter J Heidt Robert M Hoek Jörg Hamann

The epidermal growth factor (EGF)-TM7 receptors CD97, EMR1, EMR2, EMR3, and EMR4 form a group of adhesion class heptahelical molecules predominantly expressed by cells of the immune system. These receptors bind cellular ligands through EGF-like domains, localized N-terminal to a large extracellular region. Remarkably, EMR2 possesses a chimeric structure with a seven-span transmembrane (TM7) reg...

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