نتایج جستجو برای: frameshift mutations

تعداد نتایج: 174767  

Journal: :Journal of bacteriology 2009
Joseph F Petrosino Rodrigo S Galhardo Liza D Morales Susan M Rosenberg

In some enterobacterial pathogens, but not in Escherichia coli, loss-of-function mutations are a common route to clinically relevant beta-lactam antibiotic resistance. We previously constructed an assay system for studying enterobacterial beta-lactam resistance mutations using the well-developed genetics of E. coli by integrating enterobacterial ampRC genes into the E. coli chromosome. Like the...

Journal: :Advances in Bioscience and Clinical Medicine 2022

Background: Breast cancer is the most common malignancy and leading cause of death among women in Bangladesh. Mutations BRCA genes increase risk for breast cancer. A large number distinct mutations polymorphisms BRCA1 gene have been reported worldwide, a frameshift mutation exon 2 (185delAG) one commonly mutations. Therefore, study was planned to determine frequencies 2. Materials Methods: cros...

Journal: :International Journal of Colorectal Disease 2005

Journal: :Current Biology 1995
R. E. Lenski P. D. Sniegowski

Adaptive frameshift mutations in the lacZ gene of Escherichia coli are, unusually, nearly all short deletions, perhaps caused by slipped-strand mispairings in mononucleotide runs. But are they directed?

Journal: :Asian Journal of Pharmaceutical and Clinical Research 2023

Background: It is well-established that myeloproliferative diseases coexist with CLAR and JAK2. In Ph+ chronic myeloid leukemia (CML), only a few case reports indicate the existence of CLAR, JAK2V617F, JAK2 exon 12 mutations. Methods: This study examined CALR mutation profiles in Sudanese Chronic Myeloid Leukemia patients Philadelphia-positive patients. Blood samples were collected from 100 CML...

Journal: :The Journal of biological chemistry 2012
Eloisa Carta Seo-Kyung Chung Victoria M James Angela Robinson Jennifer L Gill Nathalie Remy Jean-François Vanbellinghen Cheney J G Drew Sophie Cagdas Duncan Cameron Frances M Cowan Mireria Del Toro Gail E Graham Adnan Y Manzur Amira Masri Serge Rivera Emmanuel Scalais Rita Shiang Kate Sinclair Catriona A Stuart Marina A J Tijssen Grahame Wise Sameer M Zuberi Kirsten Harvey Brian R Pearce Maya Topf Rhys H Thomas Stéphane Supplisson Mark I Rees Robert J Harvey

Hereditary hyperekplexia or startle disease is characterized by an exaggerated startle response, evoked by tactile or auditory stimuli, leading to hypertonia and apnea episodes. Missense, nonsense, frameshift, splice site mutations, and large deletions in the human glycine receptor α1 subunit gene (GLRA1) are the major known cause of this disorder. However, mutations are also found in the genes...

Journal: :Archives of ophthalmology 2006
Sudha Nallasamy Femida Kherani Dinah Yaeger Jennifer McCallum Maninder Kaur Marcella Devoto Laird G Jackson Ian D Krantz Terri L Young

OBJECTIVE To evaluate individuals with Cornelia de Lange syndrome previously screened for mutations in the NIPBL gene for genotype-phenotype correlations with regard to severity of ophthalmologic findings. METHODS Fifty-four patients with Cornelia de Lange syndrome (26 mutation positive and 28 mutation negative) with varying extent and severity of ophthalmologic findings participated in the s...

Journal: :Archives of surgery 2002
Maria A Kouvaraki Jeffrey E Lee Suzanne E Shapiro Robert F Gagel Steven I Sherman Rena V Sellin Gilbert J Cote Douglas B Evans

HYPOTHESIS Multiple endocrine neoplasia type 1 (MEN 1) syndrome is an autosomal dominant disorder caused by germline mutations in the MEN1 gene and characterized by multiple endocrine tumors, most notably in the parathyroid glands, pituitary, and pancreas. The syndrome demonstrates variable expressivity and considerable genetic heterogeneity. Patient data were examined for possible associations...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2008
Pia Alhopuro Denis Phichith Sari Tuupanen Heli Sammalkorpi Miranda Nybondas Juha Saharinen James P Robinson Zhaohui Yang Li-Qiong Chen Torben Orntoft Jukka-Pekka Mecklin Heikki Järvinen Charis Eng Gabriela Moeslein Darryl Shibata Richard S Houlston Anneke Lucassen Ian P M Tomlinson Virpi Launonen Ari Ristimäki Diego Arango Auli Karhu H Lee Sweeney Lauri A Aaltonen

A recent study described a recessive ATPase activating germ-line mutation in smooth-muscle myosin (smmhc/myh11) underlying the zebrafish meltdown (mlt) phenotype. The mlt zebrafish develops intestinal abnormalities reminiscent of human Peutz-Jeghers syndrome (PJS) and juvenile polyposis (JP). To examine the role of MYH11 in human intestinal neoplasia, we searched for MYH11 mutations in patients...

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