نتایج جستجو برای: mesangial hypercellularity

تعداد نتایج: 5493  

Journal: :American journal of physiology. Renal physiology 2009
Julia Lichtnekert Volker Vielhauer Daniel Zecher Onkar P Kulkarni Sebastian Clauss Stephan Segerer Veit Hornung Tanya N Mayadas Bruce Beutler Shizuo Akira Hans-Joachim Anders

Viral RNA or bacterial products can activate glomerular mesangial cells via a subset of Toll-like receptors (Tlr). Because Tlr2-deficient mice were recently found to have attenuated nephrotoxic serum nephritis (NSN), we hypothesized that endogenous Tlr agonists can activate glomerular mesangial cells. Primary mesangial cells from C57BL/6 mice expressed Tlr1-6 and Tlr11 mRNA at considerable leve...

Journal: :Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association 2000
I Z Pawluczyk K P Harris

BACKGROUND Cholesterol feeding has been shown to accelerate the development of glomerulosclerosis in many experimental renal diseases, possibly by promoting the infiltration of macrophages into the glomerulus. METHODS In order to assess whether hyperlipidaemia could directly modulate macrophage function to promote glomerulosclerosis, confluent quiescent mesangial cells were exposed to residen...

Journal: :The Journal of biological chemistry 2001
G Chen L Paka Y Kako P Singhal W Duan S Pillarisetti

Mesangial expansion is a key feature in the pathogenesis of numerous renal diseases involving the glomerulus. Studies indicate that mutations in apolipoprotein E (apoE) might independently contribute to kidney dysfunction. Although the role of apoE as an atheroprotective molecule is well established, its role in kidney is unclear. In this study, we sought to explore whether apoE has a protectiv...

Journal: :International Journal of Human Genetics 2022

In China, primary glomerulonephritis is the leading cause of end-stage renal disease (ESRD), and patients who require kidney transplantation often with ESRD. Glomerulonephritis primary’s most prevalent subtype Mesangial proliferative (MsPGN). Although cell apoptosis human mesangial cells (HMCs) may be harmful in MsPGN, it plays a major role elimination excessive HMCs reparative glomerular remod...

2018
Er-Min Wang Qiu-Ling Fan Yuan Yue Li Xu

BACKGROUND To investigate the protective effect of ursolic acid (UA) on high glucose (HG)-induced human glomerular mesangial cell injury and to determine whether UA inhibits cell proliferation and reactive oxygen species (ROS) production by suppressing PI3K/Akt/mTOR pathway activation. MATERIAL AND METHODS Human mesangial cells were cultured with normal glucose (NG group), high glucose (HG grou...

2016
Masanori Wakisaka Tetsuhiko Nagao Mototaka Yoshinari

PURPOSE Mesangial cells play an important role in regulating glomerular filtration by altering their cellular tone. We report the presence of a sodium glucose cotransporter (SGLT) in rat mesangial cells. This study in rat mesangial cells aimed to evaluate the expression and role of SGLT2. METHODS The SGLT2 expression in rat mesangial cells was assessed by Western blotting and reverse transcri...

2012
Laureline Berthelot Christina Papista Thiago T. Maciel Martine Biarnes-Pelicot Emilie Tissandie Pamela H.M. Wang Houda Tamouza Agnès Jamin Julie Bex-Coudrat Aurelie Gestin Ahmed Boumediene Michelle Arcos-Fajardo Patrick England Evangéline Pillebout Francine Walker Eric Daugas François Vrtosvnik Martin Flamant Marc Benhamou Michel Cogné Ivan C. Moura Renato C. Monteiro

IgA nephropathy (IgAN) is a common cause of renal failure worldwide. Treatment is limited because of a complex pathogenesis, including unknown factors favoring IgA1 deposition in the glomerular mesangium. IgA receptor abnormalities are implicated, including circulating IgA-soluble CD89 (sCD89) complexes and overexpression of the mesangial IgA1 receptor, TfR1 (transferrin receptor 1). Herein, we...

Journal: :American journal of physiology. Renal physiology 2009
Jin Wu Changlin Mei Helen Vlassara Gary E Striker Feng Zheng

Chronic inflammation and increased oxidative stress (OS) play an important role in diabetic nephropathy progression. Herein, we show that mesangial cells from streptozotocin-induced aging diabetic mice, a model of progressive diabetic nephropathy, exhibited increased OS and a proinflammatory phenotype characterized by elevated chemokines and ICAM-1 expression. This phenotypic change was consist...

Journal: :Journal of the American Society of Nephrology : JASN 2015
Peiwen Wu Yanxia Wang Mark E Davis Jonathan E Zuckerman Sarika Chaudhari Malcolm Begg Rong Ma

Accumulation of extracellular matrix derived from glomerular mesangial cells is an early feature of diabetic nephropathy. Ca(2+) signals mediated by store-operated Ca(2+) channels regulate protein production in a variety of cell types. The aim of this study was to determine the effect of store-operated Ca(2+) channels in mesangial cells on extracellular matrix protein expression. In cultured hu...

Journal: :Journal of the American Society of Nephrology : JASN 2011
Ivonne Loeffler Ulrike Hopfer Dirk Koczan Gunter Wolf

Mesangial cells in diabetic mice and human kidneys with diabetic nephropathy exhibit increased type VIII collagen, a nonfibrillar protein that exists as a heterodimer composed of α1(VIII) and α2(VIII), encoded by Col8a1 and Col8a2, respectively. Because TGF-β1 promotes the development of diabetic glomerulosclerosis, we studied whether type VIII collagen modulates the effects of TGF-β1 in mesang...

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