نتایج جستجو برای: metachromatic leukodystrophy

تعداد نتایج: 3076  

2015
Jan-Mendelt Tillema Marloes GM Derks Petra J W Pouwels Pim de Graaf Diane F van Rappard Frederik Barkhof Marjan E Steenweg Marjo S van der Knaap Nicole I Wolf

OBJECTIVE Metachromatic leukodystrophy (MLD) is an inherited lysosomal disorder due to a deficiency in arylsulfatase A with progressive demyelination and neurological decline. This retrospective MRI study investigated the extent of cortical involvement at time of diagnosis, and clinical correlates to both conventional and regional volumetric measures of brain involvement. METHODS 3D-T1-weight...

2015
Christine í Dali Norman W Barton Mohamed H Farah Mihai Moldovan Jan-Eric Månsson Nitin Nair Morten Dunø Lotte Risom Hongmei Cao Luying Pan Marcia Sellos-Moura Andrea M Corse Christian Krarup

OBJECTIVE Metachromatic leukodystrophy (MLD) is an autosomal recessive lysosomal storage disorder due to deficient activity of arylsulfatase A (ASA) that causes accumulation of sulfatide and lysosulfatide. The disorder is associated with demyelination and axonal loss in the central and peripheral nervous systems. The late infantile form has an early-onset, rapidly progressive course with severe...

Journal: :Journal of neurology, neurosurgery, and psychiatry 1964
P M FULLERTON

2011
Shahriar Koochekpour

The human PSAP-precursor gene spans approximately 20 kb in length of the long arm of chromosome 10 and consists at least 15 exons. The size of exons range from 57 to 1200 bp and the size of the introns vary from 91 to more than 3800 bp in length. The PSAP gene can be cateogorized as a polycistronic gene. Further analysis of PSAP intronic positions has indicated that it may be evolved from an an...

Journal: :AJNR. American journal of neuroradiology 2007
Antonio C M Maia Antônio J da Rocha Carlos J da Silva Sérgio Rosemberg

Finding in Metachromatic Leukodystrophy Metachromatic leukodystrophy (MLD) is a set of several disorders caused by deficient lysosomal activity. This deficiency results in accumulation of a metachromatic lipid material, galactosylceramide sulfatide, leading to the breakdown of the myelin sheath in both central and peripheral nervous systems, initially sparing the subcortical “U” fibers. Several...

Journal: :The Canadian journal of neurological sciences. Le journal canadien des sciences neurologiques 1988
L J De Meirleir M J Taylor W J Logan

Evoked potentials were studied in 22 children with leukodystrophy [10 metachromatic leukodystrophy (MLD), 4 Pelizaeus-Merzbacher (PM), 3 Krabbes, 2 adrenoleukodystrophy (ALD), and one each of Alexander's, Canavan's and multiple sulphatase deficiency (MSD) diseases]. The ABRs were abnormal in all patients (except for the younger ALD), but varied with the type of leukodystrophy. The PM and Krabbe...

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