نتایج جستجو برای: myc gene

تعداد نتایج: 1149921  

2010
Renê Gerhard Suely Nonogaki José Humberto Tavares Guerreiro Fregnani Fernando Augusto Soares Maria Aparecida Nagai

OBJECTIVES The aim of this study was to examine the expression of the N-myc downstream-regulated gene 1 protein in benign and malignant lesions of the thyroid gland by immunohistochemistry. INTRODUCTION N-myc downstream-regulated gene 1 encodes a protein whose expression is induced by various stimuli, including cell differentiation, exposure to heavy metals, hypoxia, and DNA damage. Increased...

Journal: :Molecular and cellular biology 1997
L M Facchini S Chen W W Marhin J N Lear L Z Penn

Increasing evidence supports an important biological role for Myc in the downregulation of specific gene transcription. Recent studies suggest that c-Myc may suppress promoter activity through proteins of the basal transcription machinery. We have previously reported that Myc protein, in combination with additional cellular factors, suppresses transcription initiation from the c-myc promoter. T...

Journal: :Molecular and cellular biology 2005
Francesco Faiola Xiaohui Liu Szuying Lo Songqin Pan Kangling Zhang Elena Lymar Anthony Farina Ernest Martinez

The c-Myc oncoprotein (Myc) controls cell fate by regulating gene transcription in association with a DNA-binding partner, Max. While Max lacks a transcription regulatory domain, the N terminus of Myc contains a transcription activation domain (TAD) that recruits cofactor complexes containing the histone acetyltransferases (HATs) GCN5 and Tip60. Here, we report a novel functional interaction be...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1995
W R Atchley W M Fitch

The myc gene family encodes a group of transcription factors that regulate cell proliferation and differentiation. These genes are widely studied because of their importance as proto-oncogenes. Phylogenetic analyses are described here for 45 Myc protein sequences representing c-, N-, L-, S-, and B-myc genes. A gene duplication early in vertebrate evolution produced the c-myc lineage and another...

Journal: :The Journal of general virology 1997
S Y Lee X Qu W Chen A Poloumienko N MacAfee B Morin C Lucarotti M Krause

Attempts to develop baculovirus-based insecticides by insertion of genes encoding enzyme inhibitors, neuropeptides or toxins have met with some success. However, it is often difficult to ensure correct processing or secretion of the encoded peptides. Here we tested a simpler strategy by insertion of an antisense fragment of a host gene to block translation of a protein essential for larval grow...

2011
Gretchen Poortinga Meaghan Wall Elaine Sanij Kasia Siwicki Jason Ellul Daniel Brown Timothy P. Holloway Ross D. Hannan Grant A. McArthur

Loss of c-MYC is required for downregulation of ribosomal RNA (rRNA) gene (rDNA) transcription by RNA Polymerase I (Pol I) during granulocyte differentiation. Here, we demonstrate a robust reduction of Pol I loading onto rDNA that along with a depletion of the MYC target gene upstream binding factor (UBF) and a switch from epigenetically active to silent rDNA accompanies this MYC reduction. We ...

Journal: :The Journal of Experimental Medicine 1999
Nobuo Kiuchi Koichi Nakajima Makoto Ichiba Toshiyuki Fukada Masahiro Narimatsu Katsunori Mizuno Masahiko Hibi Toshio Hirano

The signal transducers and activators of transcription (STAT) family members have been implicated in regulating the growth, differentiation, and death of normal and transformed cells in response to either extracellular stimuli, including cytokines and growth factors, or intracellular tyrosine kinases. c-myc expression is coordinately regulated by multiple signals in these diverse cellular respo...

Journal: :Cancer research 2015
Yuting Sun Jessica L Bell Daniel Carter Samuele Gherardi Rebecca C Poulos Giorgio Milazzo Jason W H Wong Rima Al-Awar Andrew E Tee Pei Y Liu Bing Liu Bernard Atmadibrata Matthew Wong Toby Trahair Quan Zhao Jason M Shohet Ygal Haupt Johannes H Schulte Peter J Brown Cheryl H Arrowsmith Masoud Vedadi Karen L MacKenzie Stefan Hüttelmaier Giovanni Perini Glenn M Marshall Antony Braithwaite Tao Liu

MYCN gene amplification in neuroblastoma drives a gene expression program that correlates strongly with aggressive disease. Mechanistically, trimethylation of histone H3 lysine 4 (H3K4) at target gene promoters is a strict prerequisite for this transcriptional program to be enacted. WDR5 is a histone H3K4 presenter that has been found to have an essential role in H3K4 trimethylation. For this r...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2012
Robert Fouret Julien Laffaire Paul Hofman Michèle Beau-Faller Julien Mazieres Pierre Validire Philippe Girard Sophie Camilleri-Bröet Fabien Vaylet François Leroy-Ladurie Jean-Charles Soria Pierre Fouret

PURPOSE To identify genetic changes that could drive cancer pathogenesis in never and ever smokers with lung adenocarcinoma. EXPERIMENTAL DESIGN We analyzed the copy number and gene expression profiles of lung adenocarcinomas in 165 patients and related the alterations to smoking status. Having found differences in the tumor profiles, we integrated copy number and gene expression data from 80...

Journal: :Oncology reports 2008
Xiaohong Lu Peter K Vogt Dale L Boger John Lunec

Inhibition of MYC/MAX dimerization by a small-molecule antagonist (IIA6B17) has been shown to interfere with MYC-induced transformation of chick embryo fibroblasts, suggesting that the functional inhibitors of the MYC family of oncoproteins have potential as therapeutic agents. In the present study, a functional MYC reporter gene assay has been developed, using a luciferase gene construct under...

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