نتایج جستجو برای: vascular permeability

تعداد نتایج: 264349  

Journal: :American journal of respiratory and critical care medicine 2004
Hiroshi Kanazawa Saeko Nomura Junichi Yoshikawa

Asthma and eosinophilic bronchitis are characterized by a similar type of eosinophilic inflammation. However, eosinophilic bronchitis differs from asthma in that there is no variable airflow obstruction or airway hyperresponsiveness. We evaluated the roles of vascular endothelial growth factor (VEGF) and microvascular permeability in causing these differences between the two diseases. Inflammat...

Journal: :British journal of pharmacology 2008
A Zarbock M Allegretti K Ley

BACKGROUND AND PURPOSE Acute lung injury (ALI) remains a major challenge in critical care medicine. Both neutrophils and chemokines have been proposed as key components in the development of ALI. The main chemokine receptor on neutrophils is CXCR2, which regulates neutrophil recruitment and vascular permeability, but no small molecule CXCR2 inhibitor has been demonstrated to be effective in ALI...

Journal: :Journal of cell science 2013
Annarita Di Lorenzo Michelle I Lin Takahisa Murata Shira Landskroner-Eiger Michael Schleicher Milankumar Kothiya Yasuko Iwakiri Jun Yu Paul L Huang William C Sessa

Transient disruption of endothelial adherens junctions and cytoskeletal remodeling are responsible for increases in vascular permeability induced by inflammatory stimuli and vascular endothelial growth factor (VEGF). Nitric oxide (NO) produced by endothelial NO synthase (eNOS) is crucial for VEGF-induced changes in permeability in vivo; however, the molecular mechanism by which endogenous NO mo...

Journal: :Infection and immunity 1980
J E Rehg

The cecal contents of guinea pigs with clindamycin-associated colitis contained a heat-labile toxin. This toxin was lethal for guinea pigs and mice, produced vascular permeability in the skin of rabbits, and was cytotoxic in tissue culture. The lethality in mice, vascular permeability in rabbit skin, and cytotoxicity in tissue culture monolayers were neutralized by Clostridium sordellii antitoxin.

Journal: :Stroke 2007
Jin-Moo Lee Guihua Zhai Qingwei Liu Ernesto R Gonzales Kejie Yin Ping Yan Chung Y Hsu Katie D Vo Weili Lin

BACKGROUND AND PURPOSE Stroke-prone spontaneous hypertensive rats (SHRsp) fed a high-salt diet develop malignant hypertension, blood-brain barrier breakdown, and spontaneous intracerebral hemorrhage (ICH). The precise spatial and temporal relationship between these events has not been well-delineated. METHODS Ten SHRsp male rats, fed a high-salt diet, were imaged weekly using MRI, starting at...

Journal: :Investigative ophthalmology & visual science 2011
Jennifer L Kielczewski Sergio Li Calzi Lynn C Shaw Jun Cai Xiaoping Qi Qing Ruan Lin Wu Li Liu Ping Hu Tailoi Chan-Ling Robert N Mames Sue Firth Robert C Baxter Patric Turowski Julia V Busik Michael E Boulton Maria B Grant

PURPOSE To examine the effect of free insulin-like growth factor (IGF) binding protein-3 (IGFBP-3), independent of the effect of insulin-like growth factors, in modulating retinal vascular permeability. METHODS We assessed the ability of a form of IGFBP-3 that does not bind IGF-1 (IGFBP-3NB), to regulate the blood retinal barrier (BRB) using two distinct experimental mouse models, laser-induc...

2006
Donald R. Senger Carole A. Perruzzi Joseph Feder Harold F. Dvorak

We have previously reported that rodent tumor cell lines secrete a potent vascular permeability factor with a molecular weight of 34,00042,000 (Senger et al. Tumor cells secrete a vascular permeability factor that promotes accumulation of ascites fluid. Science (Wash. DC), 219: 983-985,1983). This tumor-secreted vascular permeability factor (VPF) causes a rapid and completely reversible increas...

Journal: :Blood 2005
Dongxu Liu Dong Zhang Jennifer Scafidi Xiao Wu Cort C Cramer Alvin E Davis

Gram-negative bacterial endotoxemia may lead to the pathological increase of vascular permeability with systemic vascular collapse, a vascular leak syndrome, multiple organ failure (MOF), and/or shock. Previous studies demonstrated that C1 inhibitor (C1INH) protects mice from lipopolysaccharide (LPS)-induced lethal septic shock via a direct interaction with LPS. Here, we report that C1INH block...

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