نتایج جستجو برای: budding date

تعداد نتایج: 159008  

2016
Wenxiong Shi

The super-saturated surfactant monolayer collapses with the nanoparticles (NPs) at the water/trichloroethylene (TCE) interface are investigated using molecular dynamics (MD) simulations. The results show that sodium alkyl sulfate (SDS) monolayer collapse is initiated by buckling and followed primarily by budding and the bud encapsulating the NPs and oil molecules. The developed bud detaches fro...

Journal: :Journal of virology 2003
David E Ott Lori V Coren Raymond C Sowder Julian Adams Ulrich Schubert

Proteasome inhibitors reduce the budding of human immunodeficiency virus types 1 (HIV-1) and 2, simian immunodeficiency virus, and Rous sarcoma virus. To investigate this effect further, we examined the budding of other retroviruses from proteasome inhibitor-treated cells. The viruses tested differed in their Gag organization, late (L) domain usage, or assembly site from those previously examin...

Journal: :Virology 2011
Jeremy S Rossman Robert A Lamb

Influenza A virus causes seasonal epidemics, sporadic pandemics and is a significant global health burden. Influenza virus is an enveloped virus that contains a segmented negative strand RNA genome. Assembly and budding of progeny influenza virions is a complex, multi-step process that occurs in lipid raft domains on the apical membrane of infected cells. The viral proteins hemagglutinin (HA) a...

Journal: :Biochemical Society transactions 2014
Agnieszka Martyna Jeremy Rossman

Influenza A virus belongs to the Orthomyxoviridae family. It is an enveloped virus that contains a segmented and negative-sense RNA genome. Influenza A viruses cause annual epidemics and occasional major pandemics, are a major cause of morbidity and mortality worldwide, and have a significant financial impact on society. Assembly and budding of new viral particles are a complex and multi-step p...

Journal: :The Journal of Cell Biology 2002
Frantisek Supek David T. Madden Susan Hamamoto Lelio Orci Randy Schekman

SEC16 encodes a 240-kD hydrophilic protein that is required for transport vesicle budding from the ER in Saccharomyces cerevisiae. Sec16p is tightly and peripherally bound to ER membranes, hence it is not one of the cytosolic proteins required to reconstitute transport vesicle budding in a cell-free reaction. However, Sec16p is removed from the membrane by salt washes, and using such membranes ...

Journal: :The EMBO journal 1998
M J Schnell L Buonocore E Boritz H P Ghosh R Chernish J K Rose

The cytoplasmic domains of viral glycoproteins are often involved in specific interactions with internal viral components. These interactions can concentrate glycoproteins at virus budding sites and drive efficient virus budding, or can determine virion morphology. To investigate the role of the vesicular stomatitis virus (VSV) glycoprotein (G) cytoplasmic and transmembrane domains in budding, ...

2014
Janna M. Bigalke Thomas Heuser Daniela Nicastro Ekaterina E. Heldwein

The nuclear egress complex (NEC) of herpesviruses such as HSV-1 is essential for the exit of nascent capsids from the cell nucleus. The NEC drives nuclear envelope vesiculation in cells, but the precise budding mechanism and the potential involvement of cellular proteins are unclear. Here we report that HSV-1 NEC alone is sufficient for membrane budding in vitro and thus represents a complete m...

2016
Arnold Park Tatyana Yun Frederic Vigant Olivier Pernet Sohui T Won Brian E Dawes Wojciech Bartkowski Alexander N Freiberg Benhur Lee

The budding of Nipah virus, a deadly member of the Henipavirus genus within the Paramyxoviridae, has been thought to be independent of the host ESCRT pathway, which is critical for the budding of many enveloped viruses. This conclusion was based on the budding properties of the virus matrix protein in the absence of other virus components. Here, we find that the virus C protein, which was previ...

2006
Ron Alterovitz

Planning and Optimization Algorithms for Image-Guided Medical Procedures

2015
José A. Galván Melina Helbling Viktor H. Koelzer Mario P. Tschan Martin D. Berger Marion Hädrich Beat Schnüriger Eva Karamitopoulou Heather Dawson Daniel Inderbitzin Alessandro Lugli Inti Zlobec

Tumor budding in colorectal cancer is likened to an epithelial-mesenchymal transition (EMT) characterized predominantly by loss of E-cadherin and up-regulation of E-cadherin repressors like TWIST1 and TWIST2. Here we investigate a possible epigenetic link between TWIST proteins and the tumor budding phenotype. TWIST1 and TWIST2 promoter methylation and protein expression were investigated in si...

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