نتایج جستجو برای: calr

تعداد نتایج: 723  

Journal: :Autophagy 2013
Abhishek D Garg Aleksandra M Dudek Gabriela B Ferreira Tom Verfaillie Peter Vandenabeele Dmitri V Krysko Chantal Mathieu Patrizia Agostinis

Calreticulin surface exposure (ecto-CALR), ATP secretion, maturation of dendritic cells (DCs) and stimulation of T cells are prerequisites for anticancer therapy-induced immunogenic cell death (ICD). Recent evidence suggests that chemotherapy-induced autophagy may positively regulate ICD by favoring ATP secretion. We have recently shown that reactive oxygen species (ROS)-based endoplasmic retic...

Journal: :Blood 2014
Pontus Lundberg Axel Karow Ronny Nienhold Renate Looser Hui Hao-Shen Ina Nissen Sabine Girsberger Thomas Lehmann Jakob Passweg Martin Stern Christian Beisel Robert Kralovics Radek C Skoda

Myeloproliferative neoplasms (MPNs) are a group of clonal disorders characterized by aberrant hematopoietic proliferation and an increased tendency toward leukemic transformation. We used targeted next-generation sequencing (NGS) of 104 genes to detect somatic mutations in a cohort of 197 MPN patients and followed clonal evolution and the impact on clinical outcome. Mutations in calreticulin (C...

Journal: :Blood 2015
Keyur P Patel Kate J Newberry Rajyalakshmi Luthra Elias Jabbour Sherry Pierce Jorge Cortes Rajesh Singh Meenakshi Mehrotra Mark J Routbort Madan Luthra Taghi Manshouri Fabio P Santos Hagop Kantarjian Srdan Verstovsek

Although most patients with myelofibrosis (MF) derive benefit from ruxolitinib, some are refractory, have a suboptimal response, or quickly lose their response. To identify genes that may predict response to ruxolitinib, we performed targeted next-generation sequencing (NGS) of a panel of 28 genes recurrently mutated in hematologic malignancies in a cohort of patients with MF who were treated w...

Journal: :Blood 2016
William M Nauseef

In this issue of Blood, Theocharides et al report one of likely many functionally important untoward consequences of mutant calreticulin that may arise in the clinical context of myelofibrosis. Even when viewed through the narrow prism of neutrophil biology, mutant calreticulin will adversely affect myeloid cells throughout their lifespan, from the production of critical glycoproteins in bone m...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید