نتایج جستجو برای: chromosome 16p 133

تعداد نتایج: 133457  

Journal: :The Journal of clinical investigation 2000
A Raas-Rothschild V Cormier-Daire M Bao E Genin R Salomon K Brewer M Zeigler H Mandel S Toth B Roe A Munnich W M Canfield

Mucolipidosis IIIC, or variant pseudo-Hurler polydystrophy, is an autosomal recessive disease of lysosomal hydrolase trafficking. Unlike the related diseases, mucolipidosis II and IIIA, the enzyme affected in mucolipidosis IIIC (N-Acetylglucosamine-1-phosphotransferase [GlcNAc-phosphotransferase]) retains full transferase activity on synthetic substrates but lacks activity on lysosomal hydrolas...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2007
Lorenzo Melchor Emiliano Honrado Jia Huang Sara Alvarez Tara L Naylor María J García Ana Osorio David Blesa Michael R Stratton Barbara L Weber Juan C Cigudosa Nazneen Rahman Katherine L Nathanson Javier Benítez

PURPOSE Familial breast cancer represents 5% to 10% of all breast tumors. Mutations in the two known major breast cancer susceptibility genes, BRCA1 and BRCA2, account for a minority of familial breast cancer, whereas families without mutations in these genes (BRCAX group) account for 70% of familial breast cancer cases. EXPERIMENTAL DESIGN To better characterize and define the genomic differ...

Journal: :Blood 1995
P Marlton D F Claxton P Liu E H Estey M Beran M LeBeau J R Testa F S Collins J D Rowley M J Siciliano

The inversion of chromosome 16 [inv(16)] in acute myeloid leukemia (AML) is associated with a p-arm deletion in a subset of patients. The inversion results in two fusion genes: 5'-CBFB/MYH11-3' on 16p and 5'-MYH11/CBFB-3' on 16q. We have studied cells from 42 patients with inv(16) (38 patients) or t(16;16) (four patients) to define the frequency and characteristics of the deletion further. Usin...

Journal: :Journal of the American Academy of Child and Adolescent Psychiatry 2010
Judith S Nijmeijer Alejandro Arias-Vásquez Nanda N J Rommelse Marieke E Altink Richard J L Anney Philip Asherson Tobias Banaschewski Cathelijne J M Buschgens Ellen A Fliers Michael Gill Ruud B Minderaa Luise Poustka Joseph A Sergeant Jan K Buitelaar Barbara Franke Richard P Ebstein Ana Miranda Fernando Mulas Robert D Oades Herbert Roeyers Aribert Rothenberger Edmund J S Sonuga-Barke Hans-Christoph Steinhausen Stephen V Faraone Catharina A Hartman Pieter J Hoekstra

OBJECTIVE The genetic basis for autism spectrum disorder (ASD) symptoms in children with attention-deficit/hyperactivity disorder (ADHD) was addressed using a genome-wide linkage approach. METHOD Participants of the International Multi-Center ADHD Genetics study comprising 1,143 probands with ADHD and 1,453 siblings were analyzed. The total and subscale scores of the Social Communication Ques...

Journal: :Human molecular genetics 1996
S S Strautnieks R J Thompson A Hanukoglu M J Dillon I Hanukoglu U Kuhnle J Seckl R M Gardiner E Chung

Pseudohypoaldosteronism type 1 (PHA1, OMIM 264350) is a rare Mendelian disorder characterised by end-organ unresponsiveness to mineralocorticoids. Most steroid hormone insensitivity syndromes arise from mutations in the corresponding receptor, but available genetic evidence is against involvement of the mineralocorticoid receptor gene, MLR, in PHA1. A complete genome scan for PHA1 genes was und...

Journal: :American journal of human genetics 2005
Lauren A Weiss Mark Abney Edwin H Cook Carole Ober

Recently, a quantitative-trait locus (QTL) for whole blood serotonin level was identified in a genomewide linkage and association study in a founder population. Because serotonin level is a sexually dimorphic trait, in the present study, we evaluated the sex-specific genetic architecture of whole blood serotonin level in the same population. Here, we use an extended homozygosity-by-descent link...

2013
Ragnar Thomsen Henrik B. Rasmussen Kristian Linnet

Carboxylesterase 1 (CES1) is the major hydrolase in human liver. The enzyme is involved in the metabolism of several important therapeutic agents, drugs of abuse, and endogenous compounds. However, no studies have described the role of human CES1 in the activation of two commonly prescribed angiotensinconverting enzyme inhibitors: enalapril and ramipril. Here, we studied recombinant human CES1a...

Journal: :Nucleic Acids Research 2005
Thomas S. Price Regina Regan Richard Mott Åsa Hedman Ben Honey Rachael J. Daniels Lee Smith Andy Greenfield Ana Tiganescu Veronica Buckle Nicki Ventress Helena Ayyub Anita Salhan Susana Pedraza-Diaz John Broxholme Jiannis Ragoussis Douglas R. Higgs Jonathan Flint Samantha J. L. Knight

Comparative genome hybridization (CGH) to DNA microarrays (array CGH) is a technique capable of detecting deletions and duplications in genomes at high resolution. However, array CGH studies of the human genome noting false negative and false positive results using large insert clones as probes have raised important concerns regarding the suitability of this approach for clinical diagnostic app...

Journal: :Blood 2010
Christian Steidl Adele Telenius Sohrab P Shah Pedro Farinha Lorena Barclay Merrill Boyle Joseph M Connors Douglas E Horsman Randy D Gascoyne

In classical Hodgkin lymphoma (cHL) the mechanisms underlying primary refractory disease and relapse remain unknown. To gain further insight into cHL pathogenesis and genomic changes linked to treatment response, we studied 53 cHL patients by array comparative genomic hybridization, including 23 patients whose primary treatment failed, using DNA from microdissected HRS cells. Copy number altera...

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