نتایج جستجو برای: diabetic cardiomyopathy

تعداد نتایج: 147964  

Journal: :Revista da Associação Médica Brasileira 2019

Journal: :The Journal of Japan Atherosclerosis Society 1989

Journal: :Circulation research 2011
Rajesh Katare Andrea Caporali Lorena Zentilin Elisa Avolio Graciela Sala-Newby Atsuhiko Oikawa Daniela Cesselli Antonio Paolo Beltrami Mauro Giacca Costanza Emanueli Paolo Madeddu

RATIONALE Studies in transgenic mice showed the key role of (Pim-1) (proviral integration site for Moloney murine leukemia virus-1) in the control of cardiomyocyte function and viability. OBJECTIVE We investigated whether Pim-1 represents a novel mechanistic target for the cure of diabetic cardiomyopathy, a steadily increasing cause of nonischemic heart failure. METHODS AND RESULTS In strep...

2015
Javier Palazón Xavier Palomer Emma Barroso Manuel Vázquez Carrera

Diabetic cardiomyopathy is characterized by structural and functional alterations in the heart muscle of people with diabetes that finally lead to heart failure. Metabolic disturbances characterized by increased lipid oxidation, intramyocardial triglyceride accumulation and reduced glucose utilization have all been involved in the pathogenesis of diabetic cardiomyopathy. On the other hand, evid...

Journal: :Circulation. Heart failure 2014
Jun Mori Vaibhav B Patel Osama Abo Alrob Ratnadeep Basu Tariq Altamimi Jessica Desaulniers Cory S Wagg Zamaneh Kassiri Gary D Lopaschuk Gavin Y Oudit

BACKGROUND The angiotensin-converting enzyme 2 and angiotensin-(1-7) (Ang 1-7)/MasR (Mas receptor) axis are emerging as a key pathway that can modulate the development of diabetic cardiomyopathy. We studied the effects of Ang 1-7 on diabetic cardiomyopathy in db/db diabetic mice to elucidate the therapeutic effects and mechanism of action. METHODS AND RESULTS Ang 1-7 was administered to 5-mon...

2014
Guorong Lin Roger W. Brownsey Kathleen M. MacLeod

OBJECTIVES The RhoA/ROCK pathway contributes to diabetic cardiomyopathy in part by promoting the sustained activation of PKCβ2 but the details of their interaction are unclear. The purpose of this study was to investigate if over-activation of ROCK in the diabetic heart leads to direct phosphorylation and activation of PKCβ2, and to determine if their interaction affects PDK-1/Akt signaling. ...

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