نتایج جستجو برای: e gingivalis

تعداد نتایج: 1020831  

2015
Joo Youn Lee In Ah Choi Jin-Hee Kim Kyoung-Hwa Kim Eun Young Lee Eun Bong Lee Yong-Moo Lee Yeong Wook Song

BACKGROUND Periodontitis (PD) has been reported to be associated with rheumatoid arthritis (RA). Porphyromonas gingivalis (P. gingivalis) is a gram-negative anaerobic bacterium that is recognized as one of the major pathogenic organisms in PD and is the only bacterium known to express peptidylarginine deiminase (PAD). Antibody against human α-enolase (ENO1) is one of the autoantibodies in RA. E...

2012
Shuta Sugiyama Shun-suke Takahashi Fumi-aki Tokutomi Ayaka Yoshida Kyo Kobayashi Fumihiko Yoshino Satoko Wada-Takahashi Toshizo Toyama Kiyoko Watanabe Nobushiro Hamada Kazuo Todoki Masaichi-Chang-il Lee

The association of vascular reactivity between diabetes and periodontal disease has not been clarified. Gingival blood flow was measured by laser Doppler flowmetry for 31 weeks in Wistar rats, Wistar rats orally challenged with Porphyromonas gingivalis (Wistar rats + Porphyromonas gingivalis), Goto-Kakizaki rats, and Goto-Kakizaki rats orally challenged with Porphyromonas gingivalis (Goto-Kakiz...

2016
Hiroyuki Tada Takashi Matsuyama Takashi Nishioka Makoto Hagiwara Yusuke Kiyoura Hidetoshi Shimauchi Kenji Matsushita

The cytokine IL-33 is constitutively expressed in epithelial cells and it augments Th2 cytokine-mediated inflammatory responses by regulating innate immune cells. We aimed to determine the role of the periodontal pathogen, Porphyromonas gingivalis, in the enhanced expression of IL-33 in human gingival epithelial cells. We detected IL-33 in inflamed gingival epithelium from patients with chronic...

2017
Chris J. Carter James France StJohn Crean Sim K. Singhrao

Periodontal disease is of established etiology in which polymicrobial synergistic ecology has become dysbiotic under the influence of Porphyromonas gingivalis. Following breakdown of the host's protective oral tissue barriers, P. gingivalis migrates to developing inflammatory pathologies that associate with Alzheimer's disease (AD). Periodontal disease is a risk factor for cardiovascular disord...

Journal: :Infection and immunity 1997
R A Shapiro M D Cunningham K Ratcliffe C Seachord J Blake J Bajorath A Aruffo R P Darveau

CD14 is a key molecule responsible for the innate host inflammatory response to microbial infection. It is able to bind a wide variety of microbial ligands and facilitate the activation of both myeloid and nonmyeloid cells. However, its specific contribution to the innate recognition of bacteria is not known. Presently there is no information on the contribution of individual CD14 residues to E...

2012
Akiko Kukita Yuka Ichigi Ippei Takigawa Toshiyuki Watanabe Toshio Kukita Hiroshi Miyamoto

Infection of macrophages with bacteria induces the production of pro-inflammatory cytokines including TNF-α. TNF-α directly stimulates osteoclast differentiation from bone marrow macrophages in vitro as well as indirectly via osteoblasts. Recently, it was reported that bacterial components such as LPS inhibited RANKL-induced osteoclastogenesis in early stages, but promoted osteoclast differenti...

2012
Masato Yoneda Shuhei Naka Kazuhiko Nakano Koichiro Wada Hiroki Endo Hironori Mawatari Kento Imajo Ryota Nomura Kazuya Hokamura Masafumi Ono Shogo Murata Iwai Tohnai Yoshio Sumida Toshihide Shima Masae Kuboniwa Kazuo Umemura Yoshinori Kamisaki Atsuo Amano Takeshi Okanoue Takashi Ooshima Atsushi Nakajima

BACKGROUND Non-alcoholic fatty liver disease (NAFLD) is a hepatic manifestation of metabolic syndrome that is closely associated with multiple factors such as obesity, hyperlipidemia and type 2 diabetes mellitus. However, other risk factors for the development of NAFLD are unclear. With the association between periodontal disease and the development of systemic diseases receiving increasing att...

Journal: :Infection and immunity 2001
H Abaibou Z Chen G J Olango Y Liu J Edwards H M Fletcher

A 0.9-kb open reading frame encoding a unique 32-kDa protein was identified downstream of the recA gene of Porphyromonas gingivalis. Reverse transcription-PCR and Northern blot analysis showed that both the recA gene and this open reading frame are part of the same transcriptional unit. This cloned fragment was insertionally inactivated using the ermF-ermAM antibiotic resistance cassette to cre...

Journal: :Cellular microbiology 2016
Maryta N Sztukowska Akintunde Ojo Saira Ahmed Anne L Carenbauer Qian Wang Brain Shumway Howard F Jenkinson Huizhi Wang Douglas S Darling Richard J Lamont

The oral anaerobe Porphyromonas gingivalis is associated with the development of cancers including oral squamous cell carcinoma (OSCC). Here, we show that infection of gingival epithelial cells with P. gingivalis induces expression and nuclear localization of the ZEB1 transcription factor, which controls epithelial-mesenchymal transition. P. gingivalis also caused an increase in ZEB1 expression...

Journal: :Journal of periodontal & implant science 2016
Weidong Zhu Seok-Woo Lee

PURPOSE Porphyromonas gingivalis and Tannerella forsythia have been implicated as the major etiologic agents of periodontal disease. These two bacteria are frequently isolated together from the periodontal lesion, and it has been suggested that their interaction may increase each one's virulence potential. The purpose of this study was to identify proteins on the surface of these organisms that...

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