نتایج جستجو برای: fh

تعداد نتایج: 3804  

2013
Suzanne CM van Esch Wieke H Heideman Wilmy Cleijne Martina C Cornel Frank J Snoek

BACKGROUND Family history (FH) is considered an important factor to detect individuals at increased risk developing type 2 diabetes (T2D). Moreover, FH information could be used to personalise risk messages, which are assumed to increase risk-reducing behaviours. In this study, we aimed to explore Dutch health care professionals' attitudes regarding current or future uptake of a more extensive ...

Journal: :Journal of immunology 2008
Teresia Hallström Peter F Zipfel Anna M Blom Nadine Lauer Arne Forsgren Kristian Riesbeck

Pathogenic microbes acquire human complement inhibitors to circumvent the innate immune system. In this study, we identify two novel host-pathogen interactions, factor H (FH) and factor H-like protein 1 (FHL-1), the inhibitors of the alternative pathway that binds to Hib. A collection of clinical Haemophilus influenzae isolates was tested and the majority of encapsulated and unencapsulated boun...

2017
Hiroshi Mabuchi

Familial hypercholesterolemia (FH) is a disease characterized by a triad: elevated low-density lipoprotein (LDL) cholesterol, tendon xanthomas, and premature coronary heart disease. Thus, it can be considered as a model disease for hypercholesterolemia and atherosclerotic cardiovascular disease (ASCVD). For the diagnosis of hetero-FH, the detection of Achilles tendon xanthomas by palpation or o...

2012
Marloes Dekker Nitert Tasnim Dayeh Peter Volkov Targ Elgzyri Elin Hall Emma Nilsson Beatrice T. Yang Stefan Lang Hemang Parikh Ylva Wessman Holger Weishaupt Joanne Attema Mia Abels Nils Wierup Peter Almgren Per-Anders Jansson Tina Rönn Ola Hansson Karl-Fredrik Eriksson Leif Groop Charlotte Ling

To identify epigenetic patterns, which may predispose to type 2 diabetes (T2D) due to a family history (FH) of the disease, we analyzed DNA methylation genome-wide in skeletal muscle from individuals with (FH(+)) or without (FH(-)) an FH of T2D. We found differential DNA methylation of genes in biological pathways including mitogen-activated protein kinase (MAPK), insulin, and calcium signaling...

Journal: :Infection and immunity 2007
Lisa R Quin Chinwendu Onwubiko Quincy C Moore Megumi Fujioka Mills Larry S McDaniel Stephanie Carmicle

Pneumococcal surface protein C (PspC) binds to both human secretory immunoglobulin A (sIgA) and complement factor H (FH). FH, a regulator of the alternative pathway of complement, can also mediate adherence of different host cells. Since PspC contributes to adherence and invasion of host cells, we hypothesized that the interaction of PspC with FH may also mediate adherence of pneumococci to hum...

Journal: :American journal of epidemiology 2004
Carolyn M Hutter Melissa A Austin Steve E Humphries

Heterozygous familial hypercholesterolemia (FH) is an autosomal dominant disorder known to be associated with elevated cholesterol levels and increased risk of premature coronary heart disease. Since increased cholesterol levels lead to atherosclerosis, FH has also been proposed as a risk factor for peripheral vascular and ischemic cerebrovascular disease. Currently, the association between cli...

ژورنال: :مجله فیزیولوژی و فارماکولوژی ایران 0
محبوبه قنبری mahboubeh ghanbari 1. endocrine physiology research center, research institute for endocrine sciences, shahid beheshti university of medical sciences, tehran, iran 2. endocrine research center, research institute for endocrine sciences, shahid beheshti university of medical sciences, tehran, iran1- مرکز تحقیقات فیزیولوژی غدد درون ریز و متابولیسم، پژوهشکده علوم غدد درون ریز و متابولیسم، دانشگاه علوم پزشکی شهید بهشتی، تهران. 2- مرکز تحقیقات غدد درون ریز، پژوهشکده علوم غدد درون ریز، پژوهشکده علوم غدد درون ریز و متابولیسم دانشگاه علوم پزشکی شهید بهشتی ، تهران فاطمه باقری پور fatemeh bagheripuor 1. endocrine physiology research center, research institute for endocrine sciences, shahid beheshti university of medical sciences, tehran, iran 2. endocrine research center, research institute for endocrine sciences, shahid beheshti university of medical sciences, tehran, iran1- مرکز تحقیقات فیزیولوژی غدد درون ریز و متابولیسم، پژوهشکده علوم غدد درون ریز و متابولیسم، دانشگاه علوم پزشکی شهید بهشتی، تهران. 2- مرکز تحقیقات غدد درون ریز، پژوهشکده علوم غدد درون ریز، پژوهشکده علوم غدد درون ریز و متابولیسم دانشگاه علوم پزشکی شهید بهشتی ، تهران مرجان افغان marjan afghan 1. endocrine physiology research center, research institute for endocrine sciences, shahid beheshti university of medical sciences, tehran, iran 2. endocrine research center, research institute for endocrine sciences, shahid beheshti university of medical sciences, tehran, iran1- مرکز تحقیقات فیزیولوژی غدد درون ریز و متابولیسم، پژوهشکده علوم غدد درون ریز و متابولیسم، دانشگاه علوم پزشکی شهید بهشتی، تهران. 2- مرکز تحقیقات غدد درون ریز، پژوهشکده علوم غدد درون ریز، پژوهشکده علوم غدد درون ریز و متابولیسم دانشگاه علوم پزشکی شهید بهشتی ، تهران سجاد جدی sajad jeddi 1. endocrine physiology research center, research institute for endocrine sciences, shahid beheshti university of medical sciences, tehran, iran 2. endocrine research center, research institute for endocrine sciences, shahid beheshti university of medical sciences, tehran, iran1- مرکز تحقیقات فیزیولوژی غدد درون ریز و متابولیسم، پژوهشکده علوم غدد درون ریز و متابولیسم، دانشگاه علوم پزشکی شهید بهشتی، تهران. 2- مرکز تحقیقات غدد درون ریز، پژوهشکده علوم غدد درون ریز، پژوهشکده علوم غدد درون ریز و متابولیسم دانشگاه علوم پزشکی شهید بهشتی ، تهران اصغر قاسمی asghar ghasemi 1. endocrine physiology research center, research institute for endocrine sciences, shahid beheshti university of medical sciences, tehran, iran 2. endocrine research center, research institute for endocrine sciences, shahid beheshti university of medical sciences, tehran, iran1- مرکز تحقیقات فیزیولوژی غدد درون ریز و متابولیسم، پژوهشکده علوم غدد درون ریز و متابولیسم، دانشگاه علوم پزشکی شهید بهشتی، تهران. 2- مرکز تحقیقات غدد درون ریز، پژوهشکده علوم غدد درون ریز، پژوهشکده علوم غدد درون ریز و متابولیسم دانشگاه علوم پزشکی شهید بهشتی ، تهران

مقدمه: تست حداکثر ظرفیت ورزشی می تواند به عنوان یک تست تشخیصی در بیماران با مشکلات قلبی و عروقی بدون علائم کلینیکی در نظر گرفته شود. کمبود هورمون های تیروئیدی در دوره جنینی می تواند منجر به کاهش عملکرد سیستم قلبی عروقی شود. هدف از این مطالعه، ارزیابی میزان حداکثر ظرفیت ورزشی در زاده­های بالغ مبتلا به هیپوتیروئیدی جنینی  (fh) در موش صحرائی می­باشد. روش ها: fh  بوسیله اضافه کردن داروی پروپیل تیوا...

2012
Radim PUST Karel BURDA

The aim of this article is to quantify and analyze mutual interference of Frequency Hopping with Collision Avoidance (FH/CA) systems. The FH/CA system is a frequency hopping system where stations select the least jammed channel from several possible before the next jump. The article describes a mathematical model that allows determining the upper limit of the probability of collision of multipl...

Journal: :Alimentary pharmacology & therapeutics 2012
P Mansueto G Iacono A Seidita A D'Alcamo D Sprini A Carroccio

BACKGROUND Lymphoid aggregates are normally found throughout the small and large intestine. Known as lymphoid nodular hyperplasia (LNH), these aggregates are observed especially in young children and are not associated with clinical symptoms being considered 'physiological'. In children presenting with gastrointestinal symptoms the number and size of the lymphoid follicles are increased. Patien...

Journal: :The Journal of clinical endocrinology and metabolism 1998
D J Torpy R D Gordon J P Lin P R Huggard S E Taymans M Stowasser G P Chrousos C A Stratakis

Familial hyperaldosteronism type II (FH-II) is characterized by autosomal dominant inheritance and hypersecretion of aldosterone due to adrenocortical hyperplasia or an aldosterone-producing adenoma; unlike FH type I (FH-I), hyperaldosteronism in FH-II is not suppressible by dexamethasone. Of a total of 17 FH-II families with 44 affected members, we studied a large kindred with 7 affected membe...

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