نتایج جستجو برای: fh protein

تعداد نتایج: 1237776  

Journal: :Genetics and molecular research : GMR 2016
H X Wang Y X Zhao

The aim of this study was to identify potential markers of atherosclerosis development in familial hypercholesterolemia (FH) patients. GSE13985 microarray data, generated using blood samples from 5 FH patients and 5 matched controls, was downloaded from the Gene Expression Omnibus. Differentially expressed genes (DEGs) between FH and controls were identified and a protein-protein interaction (P...

2010
Isabel De Castro-Orós Miguel Pocoví Fernando Civeira

Familial hypercholesterolemia (FH) is a genetic disorder of lipoprotein metabolism characterized by high plasma concentrations of low-density lipoprotein cholesterol (LDLc), tendon xanthomas, and increased risk of premature coronary heart disease. FH is one of the most common inherited disorders; there are 10,000,000 people with FH worldwide, mainly heterozygotes. The most common FH cause is mu...

Journal: :Journal of immunology 2012
Elena Tonti Maya Fedeli Anna Napolitano Matteo Iannacone Ulrich H von Andrian Luca G Guidotti Sergio Abrignani Giulia Casorati Paolo Dellabona

B cells require MHC class II (MHC II)-restricted cognate help and CD40 engagement by CD4(+) T follicular helper (T(FH)) cells to form germinal centers and long-lasting Ab responses. Invariant NKT (iNKT) cells are innate-like lymphocytes that jumpstart the adaptive immune response when activated by the CD1d-restricted lipid α-galactosylceramide (αGalCer). We previously observed that immunization...

Journal: :International journal of molecular medicine 2014
Haibin Chen Liang Wang Jinfa Jiang

Familial hypercholesterolemia (FH) is a genetic disorder characterized by a high serum concentration of low-density lipoprotein (LDL) cholesterol. The high LDL cholesterol level leads to an excess deposition of cholesterol in the arterial walls and accelerated atherosclerosis, thereby increasing the risk of premature coronary heart disease. In the present study, we used a DNA microarray approac...

Journal: :Journal of immunology 2013
Hanne Amdahl Ilse Jongerius Taru Meri Tanja Pasanen Satu Hyvärinen Karita Haapasalo Jos A van Strijp Suzan H Rooijakkers T Sakari Jokiranta

Staphylococcus aureus is a major human pathogen causing more than a tenth of all septicemia cases and often superficial and deep infections in various tissues. One of the immune evasion strategies of S. aureus is to secrete proteins that bind to the central complement opsonin C3b. One of these, extracellular complement binding protein (Ecb), is known to interfere directly with functions of C3b....

Journal: :Acta biochimica Polonica 2017
Faisal A Al-Allaf Abdullah Alashwal Zainularifeen Abduljaleel Mohiuddin M Taher Abdellatif Bouazzaoui Hala Abalkhail Ahmad F Al-Allaf Mohammad Athar

Familial hypercholesterolemia (FH) is most commonly caused by mutations in the LDL receptor (LDLR), which is responsible for hepatic clearance of LDL from the blood circulation. We described a severely affected FH proband and their first-degree blood relatives; the proband was resistant to statin therapy and was managed on an LDL apheresis program. In order to find the causative genetic variant...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2012
Nadine Bollig Anne Brüstle Kerstin Kellner Waltraud Ackermann Elfadil Abass Hartmann Raifer Bärbel Camara Cornelia Brendel Gavin Giel Evita Bothur Magdalena Huber Christoph Paul Alexandra Elli Richard A Kroczek Roza Nurieva Chen Dong Ralf Jacob Tak W Mak Michael Lohoff

Follicular T-helper (T(FH)) cells cooperate with GL7(+)CD95(+) germinal center (GC) B cells to induce antibody maturation. Herein, we identify the transcription factor IRF4 as a T-cell intrinsic precondition for T(FH) cell differentiation and GC formation. After immunization with protein or infection with the protozoon Leishmania major, draining lymph nodes (LNs) of IFN-regulatory factor-4 (Irf...

1997
Josette Hillion Maryvonne Le Coniat Philippe Jonveaux Roland Berger

Fusion genes implicating the MLL gene have been recently FH family members whose genes are already known to be demonstrated in various 11q23 chromosomal abnormalities involved in chromosomal translocations of human maligin human hematopoietic malignancies. We analyzed a nancies, AFX and FKHR. Strikingly, in these translocations t(6;11)(q21;q23) translocation detected in a secondary acute the br...

2015
Simon J. Clark Paul N. Bishop

The recent revolution in age-related macular degeneration (AMD) genetics has demonstrated that genetic alterations affecting the alternative pathway of the complement cascade have a major influence on AMD risk. One of the two most important genetic loci is on chromosome 1 and contains genes encoding complement factor H (FH) and the factor H related proteins (FHR proteins). In macular tissue, es...

2012
Pilar Nozal Stefanie Strobel Meritxell Ibernon Dolores López Pilar Sánchez-Corral Santiago Rodríguez de Córdoba Mihály Józsi Margarita López-Trascasa

Complement alternative pathway dysregulation seems to be the pathophysiological basis of Dense Deposit Disease (DDD). Here, we describe a monoclonal anti-factor H (FH) autoantibody in a woman diagnosed with DDD with a monoclonal gammapathy. Enzyme-linked immunosorbent assays evidenced the presence of anti-FH antibodies in the patient's serum and showed that they were associated with the monoclo...

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