نتایج جستجو برای: inflammatory drugs

تعداد نتایج: 465599  

Journal: :Arthritis Research & Therapy 2006
Monika Østensen Munther Khamashta Michael Lockshin Ann Parke Antonio Brucato Howard Carp Andrea Doria Raj Rai Pierluigi Meroni Irene Cetin Ronald Derksen Ware Branch Mario Motta Caroline Gordon Guillermo Ruiz-Irastorza Arsenio Spinillo Deborah Friedman Rolando Cimaz Andrew Czeizel Jean Charles Piette Ricard Cervera Roger A Levy Maurizio Clementi Sara De Carolis Michelle Petri Yehuda Shoenfeld David Faden Guido Valesini Angela Tincani

Rheumatic diseases in women of childbearing years may necessitate drug treatment during a pregnancy, to control maternal disease activity and to ensure a successful pregnancy outcome. This survey is based on a consensus workshop of international experts discussing effects of anti-inflammatory, immunosuppressive and biological drugs during pregnancy and lactation. In addition, effects of these d...

2017
Pugazhenthan Thangaraju Sajitha Venkatesan

To the Editor: We read with great interest the article entitled, “A Case of Acute Myocardial Infarction Induced by Selective Cyclooxygenase-2 Inhibitor” by Seo et al.[1] The authors have shown a correlation of aceclofenac intake with the myocardial infarction. This is really important information to be noted as aceclofenac or any nonsteroidal anti-inflammatory drugs (NSAIDS) of this group were ...

Journal: :British journal of rheumatology 1988
T Gibson

of monoclonal human IgRF autoantibodies. J Exp Med 1984;160:893-904. 44. Fong S, Chen PP, Gilbertson JA, el al. Expression of three cross reactive idiotypes on rheumatoid factor autoantibodies from patients with autoimmune diseases and seropositive adults. J Immunol 1986;137: 122-8. 45. Chen PP, Albrandt K, Orida NK, etal. Genetic basis for the cross-reactive idiotypes on the light chains of hu...

2014
Hitoshi Ishiguro Takashi Kawahara

Prostatic diseases are characterized by increased activity of cytokines, growth factors, and cyclooxygenases- (COX-) 1 and 2. Activation of COX-1 and COX-2 results in increased levels of prostaglandins and the induction of angiogenic, antiapoptotic and inflammatory processes. Inhibition of COX enzymes by members of the widely used nonsteroidal anti-inflammatory drug (NSAID) class of drugs decre...

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