نتایج جستجو برای: jcv

تعداد نتایج: 492  

Journal: :The Journal of clinical investigation 2014
Sheila A Haley Walter J Atwood

JC virus (JCV) causes progressive multifocal leukoencephalopathy (PML), a demyelinating disease in humans. The disease, once considered fatal, is now managed with immune reconstitution therapy; however, surviving patients remain severely debilitated. Until now, there has been no animal model to study JCV in the brain, and research into treatment has relied on cell culture systems. In this issue...

Journal: :Frontiers in bioscience 2013
Guglielmo Lucchese

The present report describes the peptide commonality between JC virus (JCV) and the human proteome at the heptamer level. In total, 53 viral heptapeptides occur in functionally important human proteins with potential consequences for host functions and JCV pathogenesis. A paradigmatic example of a crucial peptide match is the SGKTTLA sequence, shared by JCV LT antigen and human nicotinamide/nic...

2013
Sarah Beltrami Emanuela Branchetti Ilker K. Sariyer Jessica Otte Michael Weaver Jennifer Gordon

Neurofibromatosis type 2 protein (NF2) has been shown to act as tumor suppressor primarily through its functions as a cytoskeletal scaffold. However, NF2 can also be found in the nucleus, where its role is less clear. Previously, our group has identified JC virus (JCV) tumor antigen (T-antigen) as a nuclear binding partner for NF2 in tumors derived from JCV T-antigen transgenic mice. The associ...

Journal: :Annals of neurology 2010
Leonid Gorelik Michaela Lerner Sarah Bixler Mary Crossman Brian Schlain Kenneth Simon Amy Pace Anne Cheung Ling Ling Chen Melissa Berman Fairuz Zein Ewa Wilson Ted Yednock Alfred Sandrock Susan E Goelz Meena Subramanyam

OBJECTIVE A study was undertaken to establish an enzyme-linked immunosorbent assay (ELISA) to detect JC virus (JCV)-specific antibodies in multiple sclerosis (MS) patients, and to evaluate its potential utility for identifying patients at higher or lower risk (ie, risk stratification) of developing progressive multifocal leukoencephalopathy (PML). METHODS A 2-step assay for detecting and conf...

Journal: :Neurology 2012
A K Trampe C Hemmelmann A Stroet A Haghikia K Hellwig H Wiendl S Goelz A Ziegler R Gold A Chan

OBJECTIVE To investigate the rate of seropositivity of anti-JC virus (JCV) antibodies in a German multiple sclerosis (MS) cohort treated with natalizumab in the postmarketing setting and to assess anti-JCV serostatus in samples obtained before diagnosis of progressive multifocal leukoencephalopathy (PML). METHODS This was a blinded, retrospective cross-sectional and longitudinal analysis for ...

Journal: :Multiple sclerosis 2013
Tomas Olsson Anat Achiron Lars Alfredsson Thomas Berger David Brassat Andrew Chan Giancarlo Comi Mefkure Eraksoy Harald Hegen Jan Hillert Poul Erik Hyldgaard Jensen Lucia Moiola Kjell-Morten Myhr Annette Oturai Sven Schippling Aksel Siva Per Soelberg Sorensen Anne-Kathrin Trampe Thomas Weber James Potts Tatiana Plavina Dominic Paes Meena Subramanyam Heinz Wiendl Hussein Dib Deniz Uren Bernhard Hemmer Dorothea Buck

JC virus (JCV) is an opportunistic virus known to cause progressive multifocal leukoencephalopathy. Anti-JC virus (Anti-JCV) antibody prevalence in a large, geographically diverse, multi-national multiple sclerosis (MS) cohort was compared in a cross-sectional study. Overall, anti-JCV antibody prevalence was 57.6%. Anti-JCV antibody prevalence in MS patients ranged from approximately 47% to 68%...

2015
Deniz Durali Marie-Ghislaine de Goër de Herve Jacques Gasnault Yassine Taoufik

Progressive multifocal leukoencephalopathy (PML) is a deadly demyelinating disease due to JC virus (JCV) replication in the brain. PML classically occurs in patients with severe immunodepression, and cases have recently been linked to therapeutic monoclonal antibodies such as natalizumab and also rituximab, which depletes B cells. B cells appear to play a complex role in the pathogenesis of PML...

Journal: :The Journal of general virology 2009
Parmjeet Randhawa Raphael Viscidi Joseph J Carter Denise A Galloway Tim D Culp Cathy Huang Bala Ramaswami Neil D Christensen

The human antibody response to polyomavirus capsid proteins is not well characterized. Recombinant BK virus (BKV), JC virus (JCV) and simian virus 40 (SV40) virus-like particles (VLP) were produced in a baculovirus system, and mouse monoclonal antibodies (mAbs) to these proteins were generated using standard methods. Nine of 12 BKV mAbs showed neutralizing activity. The non-neutralizing antibod...

Journal: :Cancer research 2003
Michele Carbone Carol Burck Monica Rdzanek Jennifer Rudzinski Rochelle Cutrone Maurizio Bocchetta

SV40 has been associated with mesothelioma development. The possible role of the closely related human polyomaviruses JC virus (JCV) and BK virus (BKV) in mesothelioma remained unclear. We found that JCV did not infect human mesothelial cells. BKV and SV40 infected mesothelial cells, expressed viral oncoproteins, and caused similar alterations of key cell regulatory genes. BKV replicated faster...

Journal: :Journal of neurovirology 1998
G L Gallia J Gordon K Khalili

In the last several years, studies have reported the detection of sequences similar to the polyomavirus, Simian Virus 40 (SV40) in human tumors including choroid plexus papillomas and ependymomas. Taken together with well-established evidence that SV40, as well as the human polyomaviruses JC virus (JCV) and BK virus (BKV), are oncogenic in several animal models, new interest has resurfaced rega...

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