نتایج جستجو برای: osteoblast response

تعداد نتایج: 984412  

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2009
Hiroyuki Inose Hiroki Ochi Ayako Kimura Koji Fujita Ren Xu Shingo Sato Makiko Iwasaki Satoko Sunamura Yasuhiro Takeuchi Seiji Fukumoto Kuniaki Saito Takashi Nakamura Haruhiko Siomi Hiroshi Ito Yoshiyasu Arai Ken-ichi Shinomiya Shu Takeda

Growing evidence shows that microRNAs (miRNAs) regulate various developmental and homeostatic events in vertebrates and invertebrates. Osteoblast differentiation is a key step in proper skeletal development and acquisition of bone mass; however, the physiological role of non-coding small RNAs, especially miRNAs, in osteoblast differentiation remains elusive. Here, through comprehensive analysis...

2013
Takashi Matsumoto Atsushi Yamada Ryo Aizawa Dai Suzuki Masayuki Tsukasaki Wataru Suzuki Mutsuko Nakayama Koutaro Maki Matsuo Yamamoto Kazuyoshi Baba Ryutaro Kamijo

Bone morphogenetic proteins (BMPs) regulate many aspects of skeletal development, including osteoblast and chondrocyte differentiation, cartilage and bone formation, and cranial and limb development. Among them, BMP-2, one of the most potent osteogenic signaling molecules, stimulates osteoblast differentiation, while it inhibits myogenic differentiation in C2C12 cells. To evaluate genes involve...

Journal: :Journal of musculoskeletal & neuronal interactions 2006
T Bellido

Both chronic excess of PTH, as in hyperparathyroidism, and intermittent elevation of PTH (by daily injections) increase the number of osteoblasts. However, whereas the former condition can lead to bone catabolism, intermittent administration of PTH causes bone anabolism. The striking difference between bone loss and bone gain in the two conditions might result from a negative vs. positive balan...

Journal: :TRANSACTIONS OF THE JAPAN SOCIETY OF MECHANICAL ENGINEERS Series A 2001

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1989
S A Kerner R A Scott J W Pike

Osteoblast-specific expression of the bone protein osteocalcin is controlled at the transcriptional level by the steroid hormone 1 alpha,25-dihydroxyvitamin D3. As this protein may represent a marker for bone activity in human disease, we examined the regulation of its expression at the molecular level by evaluating human osteocalcin gene promoter function. We describe regions within the promot...

Journal: :Cell 2000
Yutaka Yoshida Sakae Tanaka Hisashi Umemori Osamu Minowa Michihiko Usui Naoko Ikematsu Eri Hosoda Takeshi Imamura Junko Kuno Teruhito Yamashita Kohei Miyazono Masaki Noda Tetsuo Noda Tadashi Yamamoto

Bone morphogenetic protein (BMP) controls osteoblast proliferation and differentiation through Smad proteins. Here we show that Tob, a member of the emerging family of antiproliferative proteins, is a negative regulator of BMP/Smad signaling in osteoblasts. Mice carrying a targeted deletion of the tob gene have a greater bone mass resulting from increased numbers of osteoblasts. Orthotopic bone...

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