نتایج جستجو برای: cellular senescence

تعداد نتایج: 453587  

2014
Chunhua Wan Jiao Liu Xiaoke Nie Jianya Zhao Songlin Zhou Zhiqing Duan Cuiying Tang Lingwei Liang Guangfei Xu

The widespread environmental pollutant 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) is a potent toxicant that causes significant neurotoxicity. However, the biological events that participate in this process remain largely elusive. In the present study, we demonstrated that TCDD exposure triggered apparent premature senescence in rat pheochromocytoma (PC12) and human neuroblastoma SH-SY5Y cells. ...

2010
Ju Hee Lee Jung Hoan Yoo Sang Ho Oh Kyu-Yeop Lee Kwang Hoon Lee

PURPOSE Endothelial cells maintain the homeostasis of blood, which consists of plasma and cellular components, and regulate the interaction between blood and the surrounding tissues. They also have essential roles in vascular permeability, the circulation, coagulation, inflammation, wound healing, and tissue growth. The senescence of endothelial cells is closely related to the aging of the adja...

Journal: :Molecular biology of the cell 2007
Kwang Seok Kim Young Bae Seu Suk-Hwan Baek Mi Jin Kim Keuk Jun Kim Jung Hye Kim Jae-Ryong Kim

The insulin-like growth factor (IGF) signaling pathway plays a crucial role in the regulation of cell growth, differentiation, apoptosis, and aging. IGF-binding proteins (IGFBPs) are important members of the IGF axis. IGFBP-5 is up-regulated during cellular senescence in human dermal fibroblasts and endothelial cells, but the function of IGFBP-5 in cellular senescence is unknown. Here we show t...

Journal: :American journal of physiology. Lung cellular and molecular physiology 2013
Hiromichi Hara Jun Araya Saburo Ito Kenji Kobayashi Naoki Takasaka Yutaka Yoshii Hiroshi Wakui Jun Kojima Kenichiro Shimizu Takanori Numata Makoto Kawaishi Noriki Kamiya Makoto Odaka Toshiaki Morikawa Yumi Kaneko Katsutoshi Nakayama Kazuyoshi Kuwano

Mitochondria are dynamic organelles that continuously change their shape through fission and fusion. Disruption of mitochondrial dynamics is involved in disease pathology through excessive reactive oxygen species (ROS) production. Accelerated cellular senescence resulting from cigarette smoke exposure with excessive ROS production has been implicated in the pathogenesis of chronic obstructive p...

Journal: :American journal of respiratory cell and molecular biology 2004
Takao Tsuji Kazutetsu Aoshiba Atsushi Nagai

Cellular senescence is a state of irreversible growth arrest induced either by telomere shortening (replicative senescence) or by telomere-independent signals (stress-induced senescence). The alveolar epithelium is often injured by a variety of inhaled toxins, including cigarette smoke (CS). In the present study, we investigated whether exposure to CS induces senescence of alveolar epithelial c...

Journal: :Journal of Clinical Investigation 2018

Journal: :Molecular and Cellular Endocrinology 2017

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