نتایج جستجو برای: congenital insensitivity to pain

تعداد نتایج: 10730988  

2014
Leema Reddy Peddareddygari Kinsi Oberoi Raji P Grewal

Congenital insensitivity to pain (CIP) is a rare autosomal recessive genetic disease caused by mutations in the SCN9A gene. We report a patient with the clinical features consistent with CIP in whom we detected a novel homozygous G2755T mutation in exon 15 of this gene. Routine electrophysiological studies are typically normal in patients with CIP. In our patient, these studies were abnormal an...

Journal: :JPMA. The Journal of the Pakistan Medical Association 1999
U Shah M Arshad T Mozaffar

There has been tremendous progress in the detection and diagnosis of hereditary neuropathies and while the genes responsible for these rare neuropathies have improved our understanding of the development of the sensory system, they remain clinical enigmas with rare occurrences and diverse presentations. In 1963, Swanson first described two brothers with congenital insensitivity to pain, anhidro...

Journal: :Revista brasileira de anestesiologia 2009
Carlos Rogério Degrandi Oliveira Valter César Paris Renato Augusto Pereira Felipe Souza Thyrso de Lara

BACKGROUND AND OBJECTIVES Congenital insensitivity to pain and Anhidrosis (CIPA) or hereditary sensory and autonomic neuropathy type IV (HSAN IV) is a rare autosomal recessive neuropathy of the group of hereditary sensory and autonomic neuropathies (HSAN) characterized by insensitivity to pain, anhidrosis, and mental retardation. Since it is a rare condition, reports on the anesthetic conduct i...

Journal: :The Journal of clinical investigation 2017
Jianying Huang Carlos G Vanoye Alison Cutts Y Paul Goldberg Sulayman D Dib-Hajj Charles J Cohen Stephen G Waxman Alfred L George

Voltage-gated sodium channel (NaV) mutations cause genetic pain disorders that range from severe paroxysmal pain to a congenital inability to sense pain. Previous studies on NaV1.7 and NaV1.8 established clear relationships between perturbations in channel function and divergent clinical phenotypes. By contrast, studies of NaV1.9 mutations have not revealed a clear relationship of channel dysfu...

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