نتایج جستجو برای: connexin 26

تعداد نتایج: 167621  

Journal: :iranian journal of public health 0
m hashemzadeh chaleshtori m montazer zohour l hoghooghi rad h pour-jafari dd farhud m dolati

despite the enormous heterogeneity of genetic hearing loss, mutations in the gjb2 (connexin 26) gene located on “dfnb1” locus (13q12) account for up to 50% of cases of autosomal recessive non-syndromic hearing loss (arnshl) in some populations. this study describes the analysis of 100 autosomal recessive and sporadic nonsyndromic hearing loss individuals from 79 families each having at least on...

Journal: :Toxicological sciences : an official journal of the Society of Toxicology 2008
Philip Marx-Stoelting Johanna Mahr Thomas Knorpp Sandra Schreiber Markus F Templin Thomas Ott Albrecht Buchmann Michael Schwarz

Connexin (Cx) 26 and 32 are the major gap junction proteins in liver. We recently demonstrated that Cx32 is essential for phenobarbital (PB)-mediated tumor promotion in mouse liver. To investigate whether Cx26 plays a similar role, an initiation-promotion experiment was conducted using mice with a liver-specific knockout of Cx26. Control and Cx26-deficient mice were injected a single dose of N-...

Journal: :Journal of molecular and cellular cardiology 2014
Yann Prudat Jan P Kucera

Altered gap junctional coupling potentiates slow conduction and arrhythmias. To better understand how heterogeneous connexin expression affects conduction at the cellular scale, we investigated conduction in tissue consisting of two cardiomyocyte populations expressing different connexin levels. Conduction was mapped using microelectrode arrays in cultured strands of foetal murine ventricular m...

2017
Liang Xu Andrea Carrer Francesco Zonta Zhihu Qu Peixiang Ma Sheng Li Federico Ceriani Damiano Buratto Giulia Crispino Veronica Zorzi Gaia Ziraldo Francesca Bruno Chiara Nardin Chiara Peres Flavia Mazzarda Anna M. Salvatore Marcello Raspa Ferdinando Scavizzi Youjun Chu Sichun Xie Xuemei Yang Jun Liao Xiao Liu Wei Wang Shanshan Wang Guang Yang Richard A. Lerner Fabio Mammano

Background: Mutations leading to changes in properties, regulation, or expression of connexin-made channels have been implicated in 28 distinct human hereditary diseases. Eight of these result from variants of connexin 26 (Cx26), a protein critically involved in cell-cell signaling in the inner ear and skin. Lack of non-toxic drugs with defined mechanisms of action poses a serious obstacle to t...

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