نتایج جستجو برای: excessive erythrocytosis

تعداد نتایج: 54835  

Journal: :Japanese Journal of Pharmacology 1981

Journal: :Hematology. American Society of Hematology. Education Program 2000
Thomas C. Pearson Maria Messinezy Nigel Westwood Anthony R. Green Anthony J. Bench Brian J.P. Huntly Elizabeth P. Nacheva Tiziano Barbui Guido Finazzi

This review focuses on polycythemia vera (PV)-its diagnosis, cellular and genetic pathology, and management. In Section I, Dr. Pearson, with Drs. Messinezy and Westwood, reviews the diagnostic challenge of the investigation of patients with a raised hematocrit. The suggested approach divides patients on their red cell mass (RCM) results into those with absolute (raised RCM) and apparent (normal...

Journal: :The New England journal of medicine 2007
Linda M Scott Wei Tong Ross L Levine Mike A Scott Philip A Beer Michael R Stratton P Andrew Futreal Wendy N Erber Mary Frances McMullin Claire N Harrison Alan J Warren D Gary Gilliland Harvey F Lodish Anthony R Green

BACKGROUND The V617F mutation, which causes the substitution of phenylalanine for valine at position 617 of the Janus kinase (JAK) 2 gene (JAK2), is often present in patients with polycythemia vera, essential thrombocythemia, and idiopathic myelofibrosis. However, the molecular basis of these myeloproliferative disorders in patients without the V617F mutation is unclear. METHODS We searched f...

Journal: :Circulation 1993
J K Perloff A J Marelli P D Miner

BACKGROUND Adults with cyanotic congenital heart disease and elevated hematocrit levels are often phlebotomized because of an assumed risk of cerebral arterial thrombotic stroke. Whether a relation exists between hematocrit level, symptomatic erythrocytosis (hyperviscosity), and stroke remains to be established in this patient population. METHODS AND RESULTS Accordingly, 112 cyanotic patients...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1991
J W Boylan J B Van Liew P U Feig

Erythrocytosis and microcytosis have been described in strains of genetically hypertensive rats and in essentially hypertensive humans. Published discussion of these phenomena has centered around their relationship to observed alterations in ionic transport and the pathogenesis of hypertension. In presenting data for another strain of spontaneously hypertensive rats in which these findings are ...

Journal: :Case Reports in Women's Health 2019

Journal: :Current Hematologic Malignancy Reports 2016

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