نتایج جستجو برای: hemophilic arthropathy

تعداد نتایج: 7023  

Journal: :The Journal of Experimental Medicine 1957
John B. Graham Emily M. Barrow

It has been possible to duplicate in the hemophilic dog four of the major experiments which have suggested in humans an "anticephalin" hypothesis for the pathogenesis of hemophilia. The experiments in the dog have been considerably extended, as compared with the human experiments, by a variety of techniques. I. Asbestos was placed in contact with hemophilic dog plasma, and the clotting time bec...

Journal: :The Malaysian journal of pathology 2012
Pauline Balraj Munirah Ahmad Alan Soo Beng Khoo Yasmin Ayob

Haemophilia B is caused by coagulation defects in the factor IX gene located in Xq27.1 on the X chromosome. Identification of mutations contributing to defective factor IX may be advantageous for precise carrier and prenatal diagnosis. We studied 16 patients from 11 families, consisting of 8 patients of the Malay ethnic group, of which 6 were siblings. Factor IX mutations have not been previous...

Journal: :Thrombosis and haemostasis 2013
H J Metzner S W Pipe T Weimer S Schulte

The prophylactic treatment of haemophilia B and the management of haemophilia A or B with inhibitors demand frequent administrations of coagulation factors due to the suboptimal half-lives of the products commercially available and currently in use, e.g. recombinant factor IX (rFIX) and recombinant factor VIIa (rFVIIa), respectively. The extension of the half-lives of rFIX and rFVIIa could allo...

Journal: :PharmacoEconomics 2002
Alexander H Miners Caroline A Sabin Keith H Tolley Christine A Lee

OBJECTIVE To assess the cost effectiveness of primary prophylaxis with clotting factor instead of treatment following a bleed (on-demand) for individuals with severe haemophilia. DESIGN Different data sources on the clinical effects and costs of treatments were combined using a Markov model. SETTING English treatment centres. PERSPECTIVE UK societal. PARTICIPANTS Hypothetical cohorts of...

2015
Paul E. Monahan Junjiang Sun Tong Gui Genlin Hu William B. Hannah David G. Wichlan Zhijian Wu Joshua C. Grieger Chengwen Li Thipparat Suwanmanee Darrel W. Stafford Carmen J. Booth Jade J. Samulski Tal Kafri Scott W.J. McPhee Jude Samulski

Vector capsid dose-dependent inflammation of transduced liver has limited the ability of adeno-associated virus (AAV) factor IX (FIX) gene therapy vectors to reliably convert severe to mild hemophilia B in human clinical trials. These trials also identified the need to understand AAV neutralizing antibodies and empty AAV capsids regarding their impact on clinical success. To address these safet...

Journal: :Thrombosis and haemostasis 2005
Yesim Dargaud Suzette Béguin Anne Lienhart Raed Al Dieri Christine Trzeciak Jean Claude Bordet H Coenraad Hemker Claude Negrier

In haemophilia patients, a relationship is usually observed between the clinical expression of the disease and plasmatic factor VIII/factor IX (FVIII/FIX) activity. However, it is known from clinical experience, that some haemophilia patients, despite similar FVIII/FIX plasma levels, could exhibit different bleeding phenotype. After determining preanalytical test conditions, we evaluated the th...

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