نتایج جستجو برای: homozygosity mapping

تعداد نتایج: 200921  

Journal: :Blood 2012
Hengameh Abdollahpour Giridharan Appaswamy Daniel Kotlarz Jana Diestelhorst Rita Beier Alejandro A Schäffer E Michael Gertz Axel Schambach Hans H Kreipe Dietmar Pfeifer Karin R Engelhardt Nima Rezaei Bodo Grimbacher Sabine Lohrmann Roya Sherkat Christoph Klein

We describe a novel clinical phenotype associating T- and B-cell lymphopenia, intermittent neutropenia, and atrial septal defects in 3 members of a consanguineous kindred. Their clinical histories included recurrent bacterial infections, viral infections, mucocutaneous candidiasis, cutaneous warts, and skin abscesses. Homozygosity mapping and candidate gene sequencing revealed a homozygous prem...

Journal: :Molecular Vision 2009
L Abu Safieh AO Khan FS Alkuraya

PURPOSE To describe the first cataract-causing recessive mutation in the crystalline, alpha-b gene CRYAB. METHODS Homozygosity mapping complemented by linkage analysis was performed in a family with autosomal recessive juvenile cataract. RESULTS A homozygous missense mutation in CRYAB was identified. The mutation replaces a highly conserved amino acid residue in a dual function domain of th...

Journal: :Journal of medical genetics 1998
C D Fenske S Jeffery J L Weber R S Houlston J V Leonard P J Lee

The microsomal glucose-6-phosphatase (G6Pase) complex regulates the final step in glucose production from glycogenolysis and gluconeogenesis. Glycogen storage disease type 1c (GSD-1c) results from deficient activity of the phosphate/ pyrophosphate transporter of this complex and is associated with neutropenia as well as hepatomegaly and hypoglycaemia. Using three affected subjects from a single...

2015
S Nanthapisal C Murphy E Omoyinmi A Standing Y Hong SM Gomes N Klein D Eleftheriou PA Brogan

Patients and methods We used next generation (NGS) and Sanger sequencing to study select paediatric cases of PAN. Inclusion criteria were: 1. Onset of PAN <age-10-years; 2. Suspected familial PAN; 3. Sporadic PAN particularly with neurological involvement; and 4. Clinical features resembling the recent description of deficiency of ADA2 (DADA2). Whole exome sequencing was performed using a comme...

Journal: :Journal of medical genetics 2000
L Faivre M Le Merrer A Megarbane B Gilbert G Mortier V Cusin A Munnich P Maroteaux V Cormier-Daire

Acromesomelic dysplasia Maroteaux type (AMDM) is an autosomal recessive disorder belonging to the group of acromesomelic dysplasias. AMDM is characterised by severe dwarfism with shortening of the middle and distal segments of the limbs. An AMDM gene has recently been mapped to human chromosome 9p13-q12 by homozygosity mapping in four consanguineous families. Here, we show linkage of the diseas...

Journal: :American journal of human genetics 2008
Janna Nousbeck Ronen Spiegel Akemi Ishida-Yamamoto Margarita Indelman Ayelet Shani-Adir Noam Adir Ehud Lipkin Sivan Bercovici Dan Geiger Maurice A van Steensel Peter M Steijlen Reuven Bergman Albrecht Bindereif Mordechai Choder Stavit Shalev Eli Sprecher

Single-gene disorders offer unique opportunities to shed light upon fundamental physiological processes in humans. We investigated an autosomal-recessive phenotype characterized by alopecia, progressive neurological defects, and endocrinopathy (ANE syndrome). By using homozygosity mapping and candidate-gene analysis, we identified a loss-of-function mutation in RBM28, encoding a nucleolar prote...

2018
David S Lynch Viorica Chelban Jana Vandrovcova Alan Pittman Nicholas W Wood Henry Houlden

We describe a consanguineous family in which two brothers were affected by childhood onset spastic ataxia with optic atrophy and loss of motor and language skills. Through a combination of homozygosity mapping and whole-genome sequencing, we identified a homozygous copy number variant in GLS as the cause. The duplication leads to complete knockout of GLS expression. GLS encodes the brain- and k...

Journal: :Human molecular genetics 1997
H Chaïb J Kaplan S Gerber C Vincent H Ayadi R Slim A Munnich J Weissenbach C Petit

Usher syndrome (USH) is a clinically and genetically heterogeneous disorder characterized by congenital hearing loss combined with retinitis pigmentosa. This dual sensorineural deficiency is transmitted in an autosomal recessive mode. Usher syndrome type I (USH1) is the most severe form. Four loci responsible for USH1 (USH1A, 1B, 1C and 1D) have previously been mapped, among which only the USH1...

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