نتایج جستجو برای: interleukin 33

تعداد نتایج: 221536  

2017
Jinhui Ma Wanshou Guo Zirong Li Bailiang Wang Shirui Li Peng Wang

The recently discovered IL-33 as an IL-1 cytokine family member has been proved to be specifically released from osteonecrotic bones. We aimed to investigate the potential role of IL-33 in the development of osteonecrosis of femoral head (ONFH). Forty patients diagnosed with ONFH and forty age-, sex-, and body mass index- (BMI-) matched healthy subjects were included in this prospective study b...

Journal: :Cell 2016
Justin I. Odegaard Min-Woo Lee Yoshitaka Sogawa Ambre M. Bertholet Richard M. Locksley David E. Weinberg Yuriy Kirichok Rahul C. Deo Ajay Chawla

For placental mammals, the transition from the in utero maternal environment to postnatal life requires the activation of thermogenesis to maintain their core temperature. This is primarily accomplished by induction of uncoupling protein 1 (UCP1) in brown and beige adipocytes, the principal sites for uncoupled respiration. Despite its importance, how placental mammals license their thermogenic ...

Journal: :Circulation. Heart failure 2012
Eric D Abston Jobert G Barin Daniela Cihakova Adriana Bucek Michael J Coronado Jessica E Brandt Djahida Bedja Joseph B Kim Dimitrios Georgakopoulos Kathleen L Gabrielson Wayne Mitzner DeLisa Fairweather

BACKGROUND IL-33 through its receptor ST2 protects the heart from myocardial infarct and hypertrophy in animal models but, paradoxically, increases autoimmune disease. In this study, we examined the effect of IL-33 or ST2 administration on autoimmune heart disease. METHODS AND RESULTS We used pressure-volume relationships and isoproterenol challenge to assess the effect of recombinant (r) IL-...

Journal: :The Egyptian journal of immunology 2015
Rasha M Fawzy Emtethal A Said Soha M Mohamed Nehad A Fouad Essam M Akl

Behcet's disease (BD) is a chronic systemic inflammatory disorder characterized by a ‎‎course of remissions and exacerbations of unpredictable frequency and duration‎. Pro-‎inflammatory cytokines seem to be responsible for the enhanced inflammatory ‎‎response in BD. AIM OF THE WORK This study aimed to investigate serum levels of IL-33 in patients with Behcet's disease (BD) and their relations...

Journal: :Immunity 2014
Steven J Van Dyken Alexander Mohapatra Jesse C Nussbaum Ari B Molofsky Emily E Thornton Steven F Ziegler Andrew N J McKenzie Matthew F Krummel Hong-Erh Liang Richard M Locksley

Chitin, a polysaccharide constituent of many allergens and parasites, initiates innate type 2 lung inflammation through incompletely defined pathways. We show that inhaled chitin induced expression of three epithelial cytokines, interleukin-25 (IL-25), IL-33, and thymic stromal lymphopoietin (TSLP), which nonredundantly activated resident innate lymphoid type 2 cells (ILC2s) to express IL-5 and...

Recently, inflammation has been found to be a significant factor in the development of Schizophrenia (SCZ). The aim of the present research was to investigate whether interleukin-33 (IL-33, OMIM: 608678) gene polymorphism (rs11792633, C/T) is associated with the development of SCZ or not.DNA was isolated from the serum of 70 patients with SCZ and 70 healthy controls. The PCR based method was us...

2012
Hiroki Murai Huibin Qi Barun Choudhury Jim Wild Nilesh Dharajiya Swapnil Vaidya Anjana Kalita Attila Bacsi David Corry Alexander Kurosky Allan Brasier Istvan Boldogh Sanjiv Sur

BACKGROUND A series of epidemiologic studies have identified the fungus Alternaria as a major risk factor for asthma. The airway epithelium plays a critical role in the pathogenesis of allergic asthma. These reports suggest that activated airway epithelial cells can produce cytokines such as IL-25, TSLP and IL-33 that induce Th2 phenotype. However the epithelium-derived products that mediate th...

2016
Chao-Yie Yang James Delproposto Krishnapriya Chinnaswamy William Clay Brown Shuying Wang Jeanne A. Stuckey Xinquan Wang Claudio M Soares

Suppression of Tumorigenicity 2 (ST2), a member of the interleukin-1 receptor (IL-1R) family, activates type 2 immune responses to pathogens and tissue damage via binding to IL-33. Dysregulated responses contribute to asthma, graft-versus-host and autoinflammatory diseases and disorders. To study ST2 structure for inhibitor development, we performed the principal component (PC) analysis on the ...

Journal: :Cell 2016
Diane Mathis

Chawla and colleagues find that interleukin-33 is a critical controller of brown and beige adipocyte thermogenesis. Its primary impact is on the splicing of transcripts encoding uncoupling protein-1, resulting in uncoupled respiration and effective heat generation. Interleukin-33 acts perinatally to ensure adaptive thermogenesis lifelong.

2015
E Suzanne Cohen Ian C Scott Jayesh B Majithiya Laura Rapley Benjamin P Kemp Elizabeth England D Gareth Rees Catherine L Overed-Sayer Joanne Woods Nicholas J Bond Christel Séguy Veyssier Kevin J Embrey Dorothy A Sims Michael R Snaith Katherine A Vousden Martin D Strain Denice T Y Chan Sara Carmen Catherine E Huntington Liz Flavell Jianqing Xu Bojana Popovic Christopher E Brightling Tristan J Vaughan Robin Butler David C Lowe Daniel R Higazi Dominic J Corkill Richard D May Matthew A Sleeman Tomas Mustelin

In response to infections and irritants, the respiratory epithelium releases the alarmin interleukin (IL)-33 to elicit a rapid immune response. However, little is known about the regulation of IL-33 following its release. Here we report that the biological activity of IL-33 at its receptor ST2 is rapidly terminated in the extracellular environment by the formation of two disulphide bridges, res...

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