نتایج جستجو برای: pou1f1

تعداد نتایج: 147  

2017
Chandra Shekhar Pareek Paweł Błaszczyk Piotr Dziuba Urszula Czarnik Leyland Fraser Przemysław Sobiech Mariusz Pierzchała Yaping Feng Haja N. Kadarmideen Dibyendu Kumar

BACKGROUND RNA-seq is a useful next-generation sequencing (NGS) technology that has been widely used to understand mammalian transcriptome architecture and function. In this study, a breed-specific RNA-seq experiment was utilized to detect putative single nucleotide polymorphisms (SNPs) in liver tissue of young bulls of the Polish Red, Polish Holstein-Friesian (HF) and Hereford breeds, and to u...

2011
Kenji Ohba Shigekazu Sasaki Akio Matsushita Hiroyuki Iwaki Hideyuki Matsunaga Shingo Suzuki Keiko Ishizuka Hiroko Misawa Yutaka Oki Hirotoshi Nakamura

Thyrotropin-releasing hormone (TRH) activates not only the secretion of thyrotropin (TSH) but also the transcription of TSHβ and α-glycoprotein (αGSU) subunit genes. TSHβ expression is maintained by two transcription factors, Pit1 and GATA2, and is negatively regulated by thyroid hormone (T3). Our prior studies suggest that the main activator of the TSHβ gene is GATA2, not Pit1 or unliganded T3...

پایان نامه :وزارت علوم، تحقیقات و فناوری - دانشگاه اراک - دانشکده علوم پایه 1388

چکیده مطالعه ی سیتوژنتیکی در سرطان پستان القاء شده بوسیله ی dmba در موش های صحرائی نژاد sd مقدمه: در جوامع اروپائی 7/22 در صد مرگ های سرطانی ناشی از سرطان پستان بوده است و بعد از بیماری قلبی رتبه ی دوم را داشته است. در آمریکا نیز 15 در صد مرگ ناشی از سرطان ها ابتلا به سرطان پستان میباشد. سرطان پستان در کشور ما بر طبق گزارش کشوری ثبت موارد سرطانی رتبه ی اول در بین زنان را به خود اختصاص داده ...

Journal: :Journal of virology 1999
G J Chaudry K B Farrell Y T Ting C Schmitz S Y Lie C J Petropoulos M V Eiden

The Chinese hamster cell lines E36 and CHOK1 dramatically differ in susceptibility to amphotropic murine leukemia virus (A-MuLV) and gibbon ape leukemia virus (GALV); E36 cells are highly susceptible to both viruses, CHOK1 cells are not. We have previously shown that GALV can infect E36 cells by using both its own receptor, HaPit1, and the A-MuLV receptor, HaPit2. Given that the two cell lines ...

Journal: :Acta biochimica Polonica 2002
Piotr Grabarczyk Piotr J Wysocki Katarzyna Gryska Andrzej Mackiewicz

Recombinant retroviral vectors are still the most common gene delivery vehicles for gene therapy purposes, especially for construction of genetically modified tumor vaccines (GMTV). However, these vehicles are characterized by relatively low titre and in the case of many tumor cell lines, low transduction efficiency. We constructed bicistronic retroviral vector pseudotypes of amphotropic murine...

Journal: :Journal of virology 2000
C S Tailor A Nouri D Kabat

Because mutations in envelope glycoproteins of retroviruses or in their cell surface receptors can eliminate function by multiple mechanisms, it has been difficult to unambiguously identify sites for their interactions by site-directed mutagenesis. Recently, we developed a gain-of-function approach to overcome this problem. Our strategy relies on the fact that feline leukemia virus subgroup B (...

Journal: :Journal of virology 2000
C S Tailor A Nouri D Kabat

Chinese hamster ovary (CHO) cells are resistant to infections by gibbon ape leukemia virus (GALV) and amphotropic murine leukemia virus (A-MLV) unless they are pretreated with tunicamycin, an inhibitor of N-linked glycosylation. These viruses use the related sodium-phosphate symporters Pit1 and Pit2, respectively, as receptors in nonhamster cells, and evidence has suggested that the correspondi...

Journal: :Journal of virology 2001
M M Anderson A S Lauring S Robertson C Dirks J Overbaugh

Different subgroups of feline leukemia virus (FeLV) use different host cell receptors for entry. Subgroup A FeLV (FeLV-A) is the virus that is transmitted from cat to cat, suggesting that cells expressing the FeLV-A receptor are important targets at the earliest stages of infection. FeLV-B evolves from FeLV-A in the infected cat through acquisition of cellular sequences that are related to the ...

Journal: :Endocrinology 2014
Esther Diaz-Rodriguez Angela R Garcia-Rendueles Alejandro Ibáñez-Costa Ester Gutierrez-Pascual Montserrat Garcia-Lavandeira Alfonso Leal Miguel A Japon Alfonso Soto Eva Venegas Francisco J Tinahones Juan A Garcia-Arnes Pedro Benito Maria Angeles Galvez Luis Jimenez-Reina Ignacio Bernabeu Carlos Dieguez Raul M Luque Justo P Castaño Clara V Alvarez

Acromegaly is caused by somatotroph cell adenomas (somatotropinomas [ACROs]), which secrete GH. Human and rodent somatotroph cells express the RET receptor. In rodents, when normal somatotrophs are deprived of the RET ligand, GDNF (Glial Cell Derived Neurotrophic Factor), RET is processed intracellularly to induce overexpression of Pit1 [Transcription factor (gene : POUF1) essential for transcr...

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