نتایج جستجو برای: ras oncogene

تعداد نتایج: 70453  

2015
Marco Medici Norra Kwong Trevor E. Angell Ellen Marqusee Matthew I. Kim Mary C. Frates Carol B. Benson Edmund S. Cibas Justine A. Barletta Jeffrey F. Krane Daniel T. Ruan Nancy L. Cho Atul A. Gawande Francis D. Moore Erik K. Alexander

BACKGROUND Oncogenic mutations are common in thyroid cancers. While the frequently detected RAS-oncogene mutations have been studied for diagnostic use in cytologically indeterminate thyroid nodules, no investigation has studied such mutations in an unselected population of thyroid nodules. No long-term study of RAS-positive thyroid nodules has been performed. METHODS We performed a prospecti...

2016
K. Toutouzas E. Messaris M. M. Konstadoulakis E. Chatzigianni M. Karayannis P. S. Davaris G. Androulakis

Journal: :Annual review of biochemistry 1987
M Barbacid

PRIMARY STRUCTURE ................................................................ ,.......... 7HI Mammalian ras Genes ............................................. ,............................. 781 ras Genes of Other Eukaryotes.. .................................................... ........... 782 ras Oncogenes......................................................................................

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2012
Till Maurer Lindsay S Garrenton Angela Oh Keith Pitts Daniel J Anderson Nicholas J Skelton Benjamin P Fauber Borlan Pan Shiva Malek David Stokoe Mary J C Ludlam Krista K Bowman Jiansheng Wu Anthony M Giannetti Melissa A Starovasnik Ira Mellman Peter K Jackson Joachim Rudolph Weiru Wang Guowei Fang

The Ras gene is frequently mutated in cancer, and mutant Ras drives tumorigenesis. Although Ras is a central oncogene, small molecules that bind to Ras in a well-defined manner and exert inhibitory effects have not been uncovered to date. Through an NMR-based fragment screen, we identified a group of small molecules that all bind to a common site on Ras. High-resolution cocrystal structures del...

Journal: :Blood 1986
M Andreeff D E Slater J Bressler M E Furth

Human hematopoietic malignancies provide an excellent model for the study of the activity of cellular oncogenes in a context of known defects in cell proliferation and differentiation. A flow cytometric immunofluorescence assay was developed to quantitate the expression of the cellular ras oncogenes in relation to the cell cycle in individual leukemic cells. Specific binding of a monoclonal ant...

Journal: :Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research 1996
H Greulich H Hanafusa

The v-crk oncogene, identified as the transforming gene of the CT10 avian sarcoma virus, encodes an adaptor protein capable of transforming chicken embryo fibroblasts (CEFs). Because the Src homology 3 domain of v-Crk is capable of binding the Ras family guanine nucleotide exchange factors Sos and C3G, the contribution of cellular Ras to signaling by v-Crk was evaluated. NIH-3T3 cell lines stab...

2010
Min Zeng Hirotoshi Kikuchi Maria S. Pino Daniel C. Chung

The KRAS proto-oncogene plays a key role in the development of many human tumors and is commonly activated by somatic mutation or signaling through specific growth factor receptors. However, the interaction between the micro-environment and K-ras activity has not been defined. Hypoxia invariably develops as tumors outgrow their supply of oxygen. A series of well-orchestrated cellular adaptation...

Journal: :Cancer research 1987
J G Collard J F Schijven E Roos

Noninvasive, nonmetastatic BW5147 T-lymphoma cells were transfected with the activated human c-Ha-ras oncogene and were examined subsequently for the acquisition of invasive properties in vitro and of metastatic potential in vivo. It was found that several transfectants harboring the ras gene had become invasive in vitro, as assessed in hepatocyte cultures, and metastatic after tail vein inject...

Journal: :Cancer research 1997
F Aklilu M Park D Goltzman S A Rabbani

Parathyroid hormone related peptide (PTHRP) is the major causal agent in the syndrome of malignancy-associated hypercalcemia (MAH). Several studies have shown that PTHRP production is increased in response to growth factors and oncogenes, such as Tpr-Met, that are associated with the tyrosine kinase signaling pathway. Using site-directed mutagenesis of Tpr-Met and chemical inhibitors of phospho...

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